Regulating Stroke Volume, Skeletal Muscle Pump and Frank-Starling Mechanism

Regulating Stroke Volume, Skeletal Muscle Pump and Frank-Starling Mechanism

Assessment

Interactive Video

Biology, Health Sciences

University

Hard

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10 questions

Show all answers

1.

MULTIPLE CHOICE QUESTION

30 sec • 1 pt

What is the difference between end diastolic volume (EDV) and end systolic volume (ESV)?

EDV is the volume of blood in the ventricle after contraction, ESV is before contraction.

EDV is the volume of blood in the ventricle before contraction, ESV is after contraction.

EDV is the volume of blood in the atria, ESV is in the ventricle.

EDV and ESV are the same.

2.

MULTIPLE CHOICE QUESTION

30 sec • 1 pt

How can the stroke volume be increased?

By increasing the end diastolic volume.

By increasing the end systolic volume.

By decreasing the end diastolic volume.

By decreasing both EDV and ESV.

3.

MULTIPLE CHOICE QUESTION

30 sec • 1 pt

What effect does increasing the time before ventricular contraction have on stroke volume?

It only affects heart rate.

It has no effect on stroke volume.

It increases the stroke volume.

It decreases the stroke volume.

4.

MULTIPLE CHOICE QUESTION

30 sec • 1 pt

What is the primary function of the skeletal muscle pump?

To regulate blood pressure.

To pump blood out of the heart.

To increase venous return to the heart.

To decrease heart rate.

5.

MULTIPLE CHOICE QUESTION

30 sec • 1 pt

How do veins ensure one-way blood flow?

Through muscle contractions.

By being closer to the heart.

By using valves that prevent backflow.

By having thicker walls than arteries.

6.

MULTIPLE CHOICE QUESTION

30 sec • 1 pt

What is the role of sarcomeres in muscle contraction?

They are the functional units of contraction.

They regulate heart rate.

They store calcium ions.

They transport oxygen in the blood.

7.

MULTIPLE CHOICE QUESTION

30 sec • 1 pt

What happens when the heart muscle stretches due to increased blood volume?

The contraction strength decreases.

The heart rate decreases.

The heart stops contracting.

The contraction strength increases.

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