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Biological Explanation of Anorexia

Total questions: 23

Worksheet time: 12mins

Name
Class
Date
1.

Which biological explanation should you write about in your exam?

a)

evolutionary

b)

genetic

c)

hormonal

d)

neuroanatomical

2.

Strober et al conducted a study of first degree relative of people with and without eating disorders. What sort of study was this?

a)

twin study

b)

family/familial study

c)

kinship study

d)

adoption study

3.

According to Strober et al. (2000), eating disorders were ...... in people with a relative with an eating disorder than those without.

a)

11 times

b)

9 times

c)

5 times

d)

15 times

4.

Heritability of anorexia is estimated to be around....

a)

60%

b)

65%

c)

70%

d)

75%

5.

Which gene controls the development of dopamine transporter molecules?

a)

DAT1

b)

COMT

c)

5-HTR2A

d)

EPHX2

6.

DAT1 codes for a protein that creates...

a)

dopamine transporter molecules

b)

dopamine receptors

c)

an enzyme that breaks down dopamine

d)

serotonin receptors

7.

A mutation of the DAT1 gene leads to abnormal dopamine transporter molecules meaning that levels of dopamine in the synapse are...

a)

higher than average

b)

lower than average

8.

High levels of dopamine in the synapse can lead to down regulation of the ....

a)

transporter molecules on the presynaptic cell

b)

receptors on the presynaptic cell

c)

transporter molecules on the postsynaptic cell

d)

receptors on the postsynaptic cell

9.

Down regulation as a consequence of a DAT1 mutation could mean people have...

a)

lower than average dopaminergic activity

b)

higher than average dopaminergic activity

10.

Which of the following is NOT true. Mutation of the DAT1 gene could mean that people develop anorexia due to...

a)

lack pleasure at the thought of food

b)

eating is not a rewarding experience for them

c)

they don't feel hungry

d)

low levels of dopamine due to down regulation of post-synaptic receptors

11.

Dopamaine transporter molecules are on the...

a)

presynaptic cell

b)

postsynaptic cell

12.

DAT1 mutation can lead to abnormal transmission of neural impulses in the....

a)

mesocorticolimbic pathway

b)

hippocampus

c)

cingulate cortex

d)

hypothalamus

13.

The 5HTR2A gene codes for a protein linked to serotonin

a)

transporter molecules

b)

receptors (all of them)

c)

a specific sub-type of receptor called 2A

d)

MAO the enzyme that breaks down serotonin

14.

5HT 2A receptors are found on the

a)

post synaptic cell

b)

pre synaptic cell

15.

Serotonin is ...

a)

an inhibitory neurotransmitter

b)

an excitatory neurotransmitter

16.

Inhibitory neurotransmitters make the post synaptic cell...

a)

less likely to fire an action potential

b)

more likely to fire an action potential

17.

Mutation of the 5HTR2A gene leads to abnormality of the 2a receptors meaning...

a)

neural impulses continue to be fired in this neural network

b)

neural impulses are inhibited in this neural network

18.

Lack of inhibition due to mutation of the 5HTR2A gene could mean that people...

a)

still feel full when they should be getting hungry

b)

feel hungry even when they should be still feeling full

19.

Which of the following brain regions is associated with decreased 5HTR2a activity and is linked to anorexia?

a)

cingulate cortex

b)

nucleus accumbens

c)

mesocorticolimbic pathways

d)

striatum

20.

Treasure and Holland conducted a ....

a)

twin study

b)

family study

c)

adoption study

d)

genome wide association study

21.

In Treasure and Holland the concordance rate for MZ twins was....

a)

65%

b)

32%

c)

40%

d)

12%

22.

In Treasure and Holland the concordance rate for DZ twins was....

a)

65%

b)

32%

c)

40%

d)

12%

23.

Which of the following is a not a strength of the genetic explanation of anorexia...

a)

twin studies evidence e.g. Treasure and Holland

b)

application to the possible development of preventative drug treatments

c)

The equal environments assumption

d)

Kaye's review paper on 5HT2a and anorexia.