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Worksheets

Lectures 4 to 6

Total questions: 14

Worksheet time: 28mins

Name
Class
Date
1.

1. Activator Protein-1 (AP-1) family members do not include:

a)

JUN

b)

FOS

c)

ATF

d)

MAF

e)

STAT

2.

1. What parameters affect AP-1 activity?

a)

dimer binding partner and the response element to which the AP-1 dimer binds

b)

post-translational modifications to AP-1 members

c)

transcription of AP-1 members by inflammatory cytokines and growth factors

d)

interaction with glucocorticoid receptor (GR) and retinoic-acid receptor (RAR)

e)

all of the above

3.

The bZIP domain (basic leucine zipper domain) is needed

a)

to facilitate formation of dimeric transcription factors.

b)

to methylate DNA

c)

to form beta sheets.

d)

to phosphorylate Src.

4.

Both extrinsic and intrinsic apoptosis pathways aim to activate the executioner caspase, also called

a)

caspase 8

b)

caspase 3

c)

caspase 9

d)

procaspase

e)

none of the above

5.

Phosphatidylserine (PS) is normally

a)

expressed/faces on extracellular layer of bilayer of sick/damaged/dying cells

b)

expressed/faces intracellular layer of bilayer of healthy cells

c)

attracts Annexin V dye

d)

only found in bacterial plasma membranes

6.

What of the following morphological features of are not found in a cell undergoing Ferroptosis?

a)

blebbing of the plasma membrane

b)

small mitochondria with condensed mitochondrial membrane

c)

normal nuclear size and lack of chromatin condensation

d)

enlarged mitochondria

e)

cell no longer flat but rounding-up

7.

Which of the following statements is not true?

a)

MDM2 binds to p53 to block p53 transcriptional activity

b)

MDM2 binds p53 to increase p53 transcriptional activity

c)

p53 function is activated by oxidative stress

d)

p53 target genes function in metabolic homeostasis, DNA repair, growth arrest

e)

Loss of normal p53 function is associated with Li-Fraumeni disease in which humans develop multiple types of cancer.

8.

The function of p53 can be suppressed by the following

a)

mutations

b)

MDM2

c)

nuclear exclusion

d)

PTEN

e)

all of the above

9.

What is the function of PI3K?

a)

Phosphorylate PIP2 to generate PIP3

b)

Dephosphorylate PIP3 to generate PIP2

c)

Degrade p110

d)

none of the above

10.

Insulin binding to the insulin receptor results in phosphorylation of the cytoplasmic domain. How does activated insulin receptor attract PI3K?

a)

The phosphorylated tyrosine on the receptor cytoplasmic region binds the SH2 domains of PI3K

b)

The IRS1/2 binds to the receptor’s phosphorylated region and the phosphate groups on IRS1/2 binds the SH2 domain of PI3K

c)

Both

d)

None of the above

11.

Akt only requires phosphorylated Threonine-308 by mTORC2 to be fully activated.

a)

True

b)

False

12.

Which of the following is not a member of the JAK family?

a)

JAK1

b)

TYK2

c)

JAK3

d)

IL-10

13.

SOCS and CIS proteins are

a)

Products of JAK/STAT signaling

b)

Part of the JAK/STAT Negative Feedback Loop

c)

Kinases that phosphorylate STAT in the absence of JAK

d)

Kinases that help JAK phosphorylate more STAT

14.

How is STAT signaling affected by oxidative stress caused by cigarette smoke condensate extract, UV exposure, or exposure to tumor-derived extracellular vesicles?

a)

Decreases STAT phosphorylation compared to healthy cells

b)

reduces cells surface expression of the cytokine receptor subunit IFNAR1

c)

Increases STAT phosphorylation compared to healthy cells

d)

Has no effect on STAT activation