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HSCI Exam 1 study guide

Total questions: 25

Worksheet time: 13mins

Name
Class
Date
1.

How is glucose stored and why is it stored that way?

a)

Stored as glycogen, saves space, doesn't have as much influence on osmotic flux compared to glucose

b)

stored as glucagon. so it is ready to break down glycogen

c)

stored as glucose in our cell membranes. this is so cells have a quick source of energy.

d)

Stored as alanine. This is because we are resting, and do not need to use it.

2.

Where are the NADs in glycolysis coming from?

a)

The electron transport chain

b)

The Krebs cycle

c)

From the conversion of pyruvate to lactate

d)

From the Endoplasmic reticulum

3.

What are the precursors to acetyl Coa?

a)

pyruvate and glycogen

b)

glycogen and lactate

c)

fatty acids and Co2

d)

pyruvate and fatty acids

4.

Sam holds his breath for an impossibly long length of time. What kind of effect would this have on his enzymes?

a)

Would increase the efficiency of his enzymes by up regulating their production

b)

Would inhibit/denature them due to a lowering in pH

c)

Would inhibit/denature them due to a raise in pH

d)

Would inhibit/denature them due to lowering the alkalinity

5.

You lay in your bed for hours upon hours consuming large amounts of fries and sugary soda for your week off. What process is mainly taking place to the glucose being consumed?

a)

glycogenesis

b)

gluconeogenesis

c)

glycogenolysis

d)

lactic acid fermentation

6.

Where does glycolysis take place? Does it need oxygen?

a)

cytosol, no

b)

inter-membrane matrix, no

c)

Matrix of the mitochondria, yes

d)

outside of the cell, no

7.

Our Krebs cycle has stopped working. Why would this mainly have such a huge effect on the ETC?

a)

CO2 buildup would occur and would halt our ETC

b)

No O2 being brought into the ETC

c)

No FADHS or NADHs to funnel into the ETC

d)

No effect; our ETC functions independently of the Krebs cycle

8.

Where is ATP produced throughout the glycolysis-Krebs-oxidative phosphorylation pathway?

a)

2 net from glycolysis, 4 from Krebs, 28 from ETC

b)

0 net from glycolysis, 2 from Krebs, 28 from ETC

c)

4 net from glycolysis, 1 from Krebs, up to 34 from ETC

d)

2 net from glycolysis, 2 from Krebs, up to 34 from ETC

9.

If the weight of pyruvate is 88, what is the molecular weight of lactate?

a)

86 daltons

b)

90 daltons

c)

90 frisch-hertz

d)

90 joules

10.

What is a rate limiting enzyme and which enzyme in glycolysis can be considered one?

a)

hexokinase- a rate limiting enzyme is the slowest reaction of the bunch.

b)

hexokinase- a rate limiting enzyme is the fastest reaction of the bunch

c)

Glucokinase-a rate limiting enzyme is the slowest enzyme of the bunch

d)

phosphofructokinase-a rate limiting enzyme is the slowest reaction of the bunch

11.

How does coffee relate to the effect of cAMP?

a)

coffee inhibits or blocks PDE

b)

coffee degrades cAMP

c)

Coffee inhibits cAMP

d)

Coffee makes PDE more efficient

12.

What does the percent saturation of a binding site depend on?

a)

The size of the receptors binding site and the color of the ligand

b)

# of free ligands and their affinity

c)

ions charge and concentration

d)

The number of free ligands and Ca+ available

13.

What are the two things an effector protein will do?

a)

prompt a second messenger/convert GTP to GDP

b)

Inhibit PDE and degrade GTP to GDP

c)

Stop the G protein from working and send out glutamate

d)

Prompt a second messenger/convert GDP to GTP

14.

an olympic runner has transitioned to a couch potato and has not exercised in MONTHS. Do you think there will be an up-regulation or down-regulation of their receptors?

a)

up-regulation

b)

down-regulation

15.

A lipid soluble messenger and a water soluble messenger reach their designated receptors at the same time. Which messenger will have a slower, more sustained response? Why?

a)

the Water-soluble messenger

b)

The Lipid-soluble messenger

16.

T/F: The larger the concentration gradient, the larger the equilibrium potential because a larger, electrically-driven movement of ions will be required to balance the movement due to the concentration difference.

a)

True

b)

False

17.

What is the position of optimal influence for an axon terminal on the soma/cell body. WHY?

a)

Midway to the axon hillock. This allows for a more spacious position where it is not competing against other axon terminals

b)

Farthest from the axon hillock. This allows for more spatial summation to occur.

c)

Closest to the axon hillock. This is because there’s less decremental spread.

d)

Close to other axon terminals that send out ligands that are IPSPs

18.

How does the number of neurotransmitters/ligands on a post synaptic cell relate to the amplitude of voltage change?

a)

Depends on if the NTs/ligands stimulate an IPSP or an EPSP.

b)

Depends if the axon is myelinated or not.

c)

Depends on if the ligands summate at the same receptor to produce an even greater response.

d)

Depends if the soma of the dendrite is larger than normal

19.

Why will all action potentials have the same amplitude regardless of stimulus strength?

a)

This is because the frequency is proportionate to the amplitude and the frequency is always the same.

b)

Once threshold is reached, the amplitude summates to the highest voltage through spatial summation

c)

Once threshold is reached, membrane events are no longer dependent on the strength of the stimulus

20.

What is the difference between a normal sized nerve and a wide nerve?

a)

a wider nerve will have a faster conduction velocity compared to the normal sized nerve.

b)

There is no difference because velocity is the same for both.

c)

The thinner nerve has as faster frequency than a wider nerve because their relative refractory periods are shorter than wider ones.

d)

The wider nerve will be much slower than the thinner nerve.

21.

Why are refractory periods important?

a)

They are the period in which the action potential depolarizes greatly

b)

They determine when an axon will be myelinated.

c)

They ensure discrete action potentials and one way propagation

d)

They ensure one way propagation towards the axon hillock

22.

An EPSP was received now the membrane has a voltage of -55mv on the dendrite. as the graded potential transverses the membrane towards the axon hillock, the voltage constantly INCREASES. Why?

a)

Decremental spread

b)

Temporal summation

c)

Spatial summation

d)

Landre Hure summation

23.

What is the difference between a graded potential, synaptic potential and pacemaker potential?

a)

There is no difference as they are all different ways to say the same thing.

b)

A graded potential tells us the voltage on the soma/dendrite, a synaptic potential is on the axon and a pacemaker potential is on the pericardium.

c)

A graded potential and synaptic potential are the same thing, but a pacemaker potential refers to an action potential.

d)

They are all the same way to refer to an action potential

24.

What dictates the strength of an action potential?

a)

frequency of a graded potential

b)

Frequency of an action potential

c)

The amplitude of the action potential

d)

The velocity of the action potential

25.

An axon terminal of a nerve is immediately next to the dendrite of its neighboring nerve. What do you call this nerve?

a)

Axodendritic nerve

b)

Axoaxonic nerve

c)

dendroaxonic nerve

d)

exodendritic nerve