heart cant supply oxygen to the lungs

heart cant supply oxygenated blood to meet the bodys metabolic needs

heart is tired from pumping so much blood

heart is sad because someone. broke it :(

It involves just systolic dysfunction

it involves systolic dysfunction and diastolic dysfunction or both

only sys and diastolic dysfunction

it is when the heart cannot eject and relax

Its seen in respiratory disease

its seen in leg disease

its seen in cardiovascular disease

eating too much sugar

cardiomyopathies and myocarditis

myocardititii

cardivacsular disease

yes it can be see when the heart has defects in the valve, defects from birth

yes, it can also be seen with not enough blood flow in the heart (cardiac ischemia,) hypertension, valvular or congenital defects

yes

maybe

its when our ventricles dilate and this decreases contractility

its when our ventricles do not dilate and this decreases contractility

its when out ventricles get tired and can not long contract

its when our ventricles are rapidly contractng

cardiomegaly, reduced cardiac output and reduce ejection fraction

cardiosmaller, large cardiac outlet, large ejection fraction

a sad heart

dysfunction of the heart

It is characterized by an increased myocardial compliance but the contracting is initially normal

decreased mycocardial compliance and the contraction is not normal

it is characterized by a decreased myocardial compliance but the contracting is initially normal

abnormal contraction

Asymmetrical involvement of IVS

it leads to hypertrophy in the left ventricle

Increased diastolic pressure but normal cardiac output

a crying heart :(

may lead to sudden arrhythimia

may lead to myocardial ischemia

sudden death

hypertension in the lungs

hypertrophic and restrictive cardiomyopathies

hypertension and restrictive cardiomyopathies

cardiomyopthies only

hytertension only

its when the heart is reduced

its when an abnormal protein shows up in your heart

its when the myocardial or endocardial tissue of the heart is replaced with cancer, infiltrative or fibrotic disease

its when the heart tissue is replaced with a spongy layer tissue

it must just go with the flow

it must adapt to new mechanisms

it has to find a way to survive

to maintain pumping functions to keep is alive and to maintain cardiac output and adequate perfusion of vital organs

it has to try to do something to keep the body alive

to try to do something so it doesnt fail!!!

short term mechanisms

long term mechanisms

peripheral mechanisms

middle mechanisms

these are activated within a few hours

activated within a few days

activated within a few months

activated within a few minutes

these are activated all of a sudden

slow mechanisms

these are activated rapidly but they continue over a long period of time

dont work

mechanisms occurring over years

mechanisms developed within weeks to months to save the heart from failure

tries to help heart adaptive in a few minutes

fast fixing the heart

increasing the muscle length enhances the tension developed during contraction

its a short term adaptive mechanisms

long term mechanism

decrease muscle length does not have tension during contraction

with sleeping

with normal activites

with exercise

with pumping blood

increases volume to activate the kidneys

adregenergic system- increases contractility or the velocity with which the heart muscle contracts and also increases heart rate

it activates the neurohumoral sytem

increases heartrate

it maintains arterial pressure and tissue perfusion

increases the cardiac preload

the immune system

increases the cardiac afterload

its when cardiac output is working really really fast

its when your cardiac output does not work anymore

It is when the arterial pressure is maintained when you have a reduced cardiac output that is primitive but necessary and effective

blood is flowing to the non important parts of the body

It is when the blood flow must be redistributed to more important parts of the body, blood goes from kidneys, skin, to brain and heart

blood must be going everywhere in the body

blood does not flow anymore

Its when the blood shifts from the peripheral pool to the central circulation-- so blood is needed in the kidney so we take the blood away from other parts of our body

it enhances ventricular filling and helps maintain cardiac output

its when cardiac output doesnt work anymore

your body needs blood in all parts of our body

Our heart is having a volume overload and this leads to ventricular dilation, increasing the demand of the heart

its when the heart can handle the pressure being presented to it

its when the heart can adapt to change

stress does not affect the heart

T=PxR/M

Its when the heart becomes distended and the radius increases and the wall tension does too to generate the same pressure

its when the heart can handle the pressure

heart is getting bigger and it is in the progression of heart failure

When the heart has more pressure and higher wall tension, leadings to pathological ventricular remodeling

we want to reduce wall tension, so wall thickness increases and this increases the muscle cells in our heart, our muscle mass leading to heart failure

heart is remodeling because it can handle the change in mass

heart needs some love

EJ Increases

EDVP decreases

EJ Decreases

EDVP Decreases

EJ decreases

EDVP increases

EJ increases

EDVP Increases

decreases, the left ventricle is dysfunctional

the resting cardiac output may be normal, but when we try exercising we may suddenly fall or collapse

We can exercise even harder

We are able to live a normal life and exercise constantly

Diabetes

Blood Redistribution and Edema

High blood sugar

our blood is flowing to every single part of our body now

the heart is going back to normal

Since our blood is going to our heart, other parts of out body are failing

our skeletal muscles are weaking, anaerobic metabolism, lactic acidosis, weakness and fatigue

liver dysfunction, reduced liver perfusion

diminished renal perfusion, na and nitrogen retention

lung failure

left side failure leads to right side failure :(

on the left side of our heart, we have pulmonary congestion, on the right side we have fluid accumulation

intermitten claudication-- chronic ischemia of the legs with hypoxic pains while walking

ankle edema, congestive hepatomegaly, ascites, pleural effusion

myocarditis

ischemia heart disease

valve heart disease

cardiomyopathy

ischemia heart disease

left side hf, lung disease, pulmonary hpertension, pulmonary emboli

disease

liver

it becomes depressed

it increases

it decreases

its happy

tachnypea-- increased rate of respiration

rales-- indicating fliud in alveolar spaces

dyspnea- not feeling enough air, feels like ur choking

orthopnea-- difficulty breathing when lying down

paroysmal noctural dyspnea-- sudden inability to breathe, awakeing patient during night

congestion of organs such as liver

clubbing of fingers and cyanosis

swelling, jugular venous distension, and increased jugular vein pressure

edema of lower extremities

its fluid moving into interstitial and alveolar spaces, it has to do with profuse sweating and cyanosis

its when you are drowning in your own fliud

but its not always related to heart failure, but it is life threatening and needs to be treated immediately

its when you are okay and nothing happens to your body

shows the heart enlarged and pulmonary congestion

used for seeing smoke in your lungs

used to see the liver

used to see how your heart is beating

checking out the heart dancing

it measures the pressure in both the right and left side of the heart with wedging

its when you want to check ur heart beat

its a ultrasound of the heart

is shows the differenes between systolic and diastolic failure

it is when u see the heart beat

looks at cardiac vs non cardiac causes of dyspnea

a peptide releassed from the ventricles in the heart in response to changes in ventricular filling pressure

causes natriuresis and vasodilation

its when ur heart is sleeping

seeing the renal functions, aka looking at kidney electrolytes

electrolytes are minerals in blood and other body fliuds that carry an electric charge

looking at the sugars in the kidney

pay attention to pulmonary edema, ankles

control levels of stress

ask about their diseases, medical history, make sure they dont sit flat in the chair if they cannot, they might not get air.

watch for medication interactiions