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BioCh

Total questions: 104

Worksheet time: 53mins

Name
Class
Date
1.

What is the normal range of total serum bilirubin ?

a)

Above 7

b)

1.5-1.7

c)

0.2-1

d)

2.0-4.0

2.

gluconeogenesis operates after fasting for :

a)

20H

b)

6H

c)

15H

d)

10H

3.

Control of urea cycle involves the key enzyme?

a)

Arginase

b)

Carbamoyl phosphate synthetase I

c)

Ornithine transcarbamoylase

d)

Argininosuccinase

4.

Favism is caused by which of the following?

a)

Pyruvate kinase deficiency

b)

Galactokinase deficiency

c)

G6PD deficiency

d)

Pyrimidine 5' nucleotidase deficiency

5.

The absorption of carbohydrates active transport require:

a)

Contransport of sodium

b)

Glucose transport 5

c)

The presence of ribose

d)

None of the above

6.

The intracellular site of glycolytic enz is:

a)

The mitochondria

b)

The endoplasmic reticulum

c)

The cytosol

d)

The nucleus

7.

The following enz is irreversible enzs of glycolysis:

a)

Pyrvate kinase

b)

Phosphohexose iso

c)

Aldolase

d)

Enolase

8.

In RBC's num if ATPs reuilting from glycolysis is:

a)

2 ATPs

b)

6 ATPs

c)

8 ATPs

d)

10 ATPs

9.

Energy is produced at substrate level in the following reactions:

a)

Glucokinase and pyrvate kinase

b)

Pyrvate kinase and phosphofructokinase

c)

Pyrvate kinase and phosphoglycerate kinase

d)

Glucokinase and phosphofructokinase

10.

The following enz it glycolysis requires NAS as coenzyme

a)

Mlate dehydrogenase

b)

Glyceraldehyde-3-P-dehydrogenase

c)

Succinate dehydrogenase

d)

Iocitrate dehydrogenase

11.

Oxidation decarboxylation of pyrvate require:

a)

NAD

b)

FAD

c)

Lipoic acid

d)

All of the above

12.

Carboxylation of pyruvate require the following Exc:

a)

NAD

b)

Mn²

c)

Biotin

d)

ATP

13.

Hexose monophosphate pathway is important for

a)

Production od energy

b)

Production of NAD and CO2

c)

Production of NADPH and Ribose-1-P

d)

None of the above

14.

Hexose monophosphate pathway is important for RBC's cuz:

a)

It is needed for heme synthesis

b)

It protect the cell form hemolysis

c)

It induced the respiratory burst

d)

All of the above

15.

UDP glucuronate formed by uronic acid parhway is important for

a)

Conjugation with bilirubin

b)

Detoxification reactions

c)

Formation of GAG's

d)

All of the above

16.

Essential pentosuria is a metabolic disorder caused by deficiency of:

a)

G6P

b)

G6PD

c)

L -Xylulose reductase

d)

None of the above

17.

The key enz of glycogenesis is:

a)

Phosphorylase

b)

Phosphate

c)

Glycogen synthase

d)

Glucokinase

18.

The key enz of glycogenesis ia active when:

a)

Phosphorylated

b)

Dephoshorylated

c)

Methylated

d)

Glycated

19.

The key enz of glycogenesis is:

a)

Activated by insulin

b)

Inhibited by insulin

c)

Activated by gluagons

d)

None of the above

20.

The ket enz of glycogenolysis is

a)

Phosphorylase

b)

Phosphatase

c)

Glycogen synthase

d)

Glucokinase

21.

The key enz of glycogenolysis is active when:

a)

Phosphorylated

b)

Dephoshorylated

c)

Methylated

d)

Carboxylated

22.

The key enz of glycogenolysis is active when:

a)

Phosphorylated

b)

Dephoshorylated

c)

Methylated

d)

Carboxylated

23.

The following are Gluconeogenesis substrate exc

a)

Glycerol

b)

Pyrvate

c)

Acetyl CoA

d)

Propionyl CoA

24.

Gluconeogenesis is not active in skeletal muscle due to the absence of:

a)

Glucokinase

b)

G6P

c)

F1,6biphophatase

d)

None

25.

Galactosemia is a metabolic disease caused by deficiency of:

a)

Galactokinase

b)

Galactose 1-P uridyl transfrase

c)

UDP Galactose 4 epimerase

d)

Anyone of the previous enzs

26.

Galactosemia is associated with catract due to accumulation of:

a)

Sorbitol

b)

Galactiol

c)

Glucose

d)

All of the above

27.

Hereditary fructose intolerance leads to hypoglycemic attacks due to:

a)

Activation of phosphorylate

b)

Inherited of phosphorylase

c)

Inherited of glycogen synthase

d)

All of the above

28.

Renal threshold is the

a)

Maximum capacity of the Renault tubes to reabsorbed glucose

b)

Maximum capacity of the renal tubes to execrete glucose

c)

Maximum capacity of the renal tubes to undergo Gluconeogenesis

d)

None

29.

a - oxidation

a)

Occurs following B - oxidation

b)

Occurs to beat methylated FA

c)

None

30.

Refsum disease:

a)

Metabolic disease caused by impaired a - oxidation

b)

It lead to accumulation of phytanic acid

c)

It lead to neurological disorder

d)

All of the above

31.

De Novo synthesis of FA require

a)

Multienzyme complex

b)

NADPH+H+

c)

Acetyl CoA

d)

All of the above

32.

De Novo synthesis of FA require

a)

Multienzyme complex

b)

NADPH+H+

c)

Acetyl CoA

d)

All of the above

33.

Novo synthesis of FA occurs in:

a)

The mitochondria

b)

The microsome

c)

The cytosol

d)

The preoxisome

34.

Microsomal system of FA synthesis is:

a)

Concerned with FA elongation

b)

Active mainly the brain

c)

Uses malonyl CoA

d)

All of the above

35.

Lipogenesis is activated by:

a)

Insulin

b)

Glucagon

c)

Glucocorticoids

d)

None

36.

The active form of hormone sensitive lipase enz is:

a)

Phosphorylated

b)

Dephoshorylated

c)

Not eegulated by phosphorylation

d)

None

37.

Dietary lipids enter the lacteats in the form of:

a)

Micelle

b)

Chylomicron

c)

VLDL

d)

None

38.

Tryaacylglycerol contacts of chlomicron is hydrolysed:

a)

Hornone sensitive lipase

b)

Lipoprotein lipase

c)

Intestinal lipase

d)

None

39.

The most common patheay of FA oxidation is:

a)

a - oxidation

b)

B - oxidation

c)

w - oxidation

d)

∆ - oxidation

40.

The intracellular site of B - oxidation is:

a)

Mitochondria

b)

Cytosol

c)

Endoplasmic reticulum

d)

Preoxisome

41.

B - oxidation of odd FA may be followed by Gluconeogenesis cuz:

a)

It give olny acetyl CoA

b)

It gives propionyl CoA

c)

It gives glycerol

d)

All of the above

42.

The role of carnitine in B - oxidation is:

a)

Transport FA across mitochondrial membrane

b)

Activation of the FA

c)

Collection of energy

d)

All of the above

43.

B - oxidation requires the following coenzymes:

a)

TTP and lipoic acid

b)

NAD and FAD

c)

NADP and FMN

d)

All of the above

44.

The following phospholipid is a second massenger for hormone action:

a)

Phosphatidyl chline

b)

Phosphatidyl ethanolamine

c)

Phosphatidyl inositol

d)

Phosphatidyl serin

45.

The precursor of sphingosine is:

a)

Acetyl CoA and serin

b)

Palmitoyl CoA and glucine

c)

Palmitoyl CoA and serin

d)

Choline and phophatidic acid

46.

Acetyl CoA is the precursor of the following compounds:

a)

Cholesterol

b)

FA

c)

Ketone bodies

d)

All of the above

47.

Among cholesterol derivatives are:

a)

Bilirubin

b)

Vitamin D3

c)

Arachidonic acid

d)

All of the above

48.

The following hormones helps rhe conversion of chdlsterol into bile salt:

a)

Insulin

b)

Thyroxin

c)

Glucagon

d)

Growth hormone

49.

The lipoprotein fraction that carries chocolate in plasma safely is:

a)

LDL HDL

b)

HDL

c)

VLDL

d)

Chylomicron

50.

Ketone bodies include:

a)

Acetyl CoA

b)

Acetoacetate

c)

Oxaloacetate

d)

None

51.

Ketogenesis occurs in:

a)

Liver mitochondria

b)

Liver cytosol

c)

Mitochondria of extrahepatic tissues

d)

Cytosol of extrahepatic tissue

52.

The following metabolyte of kreb's cycle is important for ketolysis:

a)

Isocitrate

b)

Malate

c)

Succinate CoA

d)

Fumarate

53.

Ketosis means:

a)

Synthesis of ketone bodies

b)

Oxidation of ketone bodies

c)

Accumulation of ketone bodies in the blood

d)

All of the above

54.

The following are antiketogenic substance exc

a)

insulin

b)

Carbohydrates

c)

Protein

d)

FA

55.

The following lipoproteins are derived from VLDL

a)

HDL

b)

LDL

c)

IDL

d)

Chylomicron

56.

Fatty live means accumulation of the following lipids in liver

a)

Phospholipid

b)

Cholesterol

c)

Triacylglycerol

d)

Ketone bodies

57.

Methyl donors are among the lipotropic factors cuz:

a)

They enter in the synthesis of FA

b)

They help the regeneration of SAM

c)

They carry lipids away from the liver

d)

They help the biliart excretion of lipids

58.

Methyl donor include the following exc:

a)

Glycine betaine

b)

PAPS

c)

SAM

d)

Methyl H4F

59.

Lipotrpic factors help:

a)

The synthesis of phospholipids and proteins

b)

Helps the excretion of lipids in bile

c)

Prevent fatty acid synthesis

d)

None

60.

The following peptide is essential for the absorption of A.a

a)

Intestinal peptide

b)

Gastric peptide

c)

Glutathione

d)

All of the above

61.

The following conditions are associated with -ve nitrogen balance

a)

Convalescent from diseases

b)

Growth of children

c)

Pregnancy

d)

Diabetes mellitus

62.

The most important method for A.a deamination is:

a)

Oxidation deamination

b)

Transamination

c)

Transdeamination

d)

Reductive deamination

63.

The following are specific methods of deamination exc:

a)

Glycine oxidation

b)

Histidase

c)

Glutaminase

d)

Transamination

64.

The most important A.a is deamination Process is:

a)

Glutamic acid

b)

Tyrosine

c)

Histidine

d)

Proline

65.

The end product of ammonia metabolic in the liver:

a)

Urea

b)

Uric acid

c)

Creatine

d)

Glutamine

66.

The following A.a have glucogenic fates exc:

a)

Tyrosine

b)

Glutamic acid

c)

Alanine

d)

Leucine

67.

The following A.a are needed fir creatin synthesis

a)

Arginine, glycine, methonion

b)

Arginine, ornithine, glycing

c)

Glycine, arginine, cysteine

d)

None

68.

Creatine synthesis occur in the following organs exc:

a)

The liver

b)

THe kidney

c)

The muscles

d)

The brain

69.

Creatine is important for

a)

Storage of energy in muscles

b)

Muscle contraction

c)

Muscle relaxation

d)

All of the above

70.

The following are derivatines if glycine exc

a)

Bile salt

b)

Glutathione

c)

Heren

d)

Thyroid hormones

71.

The following A.a enters the structure of glutathione

a)

Glutamine Methionine Glycine

b)

Glycine Cysteine Glutamic acid

c)

Glycine alanine serine

d)

None

72.

Primary hyperxaluira is a metabolic disease characterized by:

a)

Impaired metabolim of glyoxalic acid

b)

Inhibited of glycine cleavage system

c)

Excessive formation of oxaloacetate acid

d)

None

73.

The A.a serine is important for the formation of:

a)

Ethanolamine

b)

Coline

c)

Sphingosine

d)

All of the above

74.

Serine is non essential cuz it could be synthesisized from

a)

Glycine

b)

Threonine

c)

Alanine

d)

All of the above

75.

The following are important derivatives of cysteine

a)

Bile salt

b)

SAM

c)

PAPS

d)

All of the above

76.

Cysteine is non essential being synthesized from

a)

Serine methionine

b)

Homocysteine serine

c)

Homocysteine methionine

d)

None

77.

The main important of methionine is that is

a)

Acts as sulfate donor

b)

Act as methyl donor

c)

Act as phosphate donor

d)

All of the above

78.

The active form of methionine is

a)

SAM

b)

SAH

c)

PAPS

d)

None

79.

Homocystinuria is a metabolic disorder caused by deficiency of:

a)

Cysthionine synthase

b)

Cysthionase

c)

Cystine

d)

All of the above

80.

Glutanmine help rhe elemination of ammonia from:

a)

The liver

b)

The brain

c)

The RBC's

d)

The kidneys

81.

Arginine enters the following compounds exc:

a)

Urea

b)

Creatine

c)

Nitric oxide

d)

Melanin

82.

Phenyl ketonuria is a metabolic disorder caused by deficiency of:

a)

Tyrosinase

b)

Homogentisic acid oxidase

c)

Phenylalanine hydroxylase

d)

Tyrosine transminase

83.

Among the important derivatives of tyroxine is:

a)

Serotonin

b)

Thyroid hormones

c)

Melatonin

d)

All of the above

84.

Among the important derivatives fo tryptophan is:

a)

Serotonin

b)

Thyroid hormones

c)

Melatonin

d)

All of the above

85.

The following A.a enter in formation of neurotransmitters exc:

a)

Alanine

b)

Phenylalanine

c)

Tyrsine

d)

Tryptophan

86.

Hartnup disease is metabolic disorder of;

a)

Tyrosine

b)

Tryptophan

c)

Histidine

d)

None

87.

Histidine enter in the following compound:

a)

Ergothionine

b)

Creatine

c)

Creatinine

d)

None

88.

Proline and hydroxyproline are important components of:

a)

Keratin

b)

Collagen

c)

Heme

d)

Urines

89.

A.a enters purine synthesis exc

a)

Glycine

b)

Alanine

c)

Aspartic

d)

Glutamine

90.

A.a gives glutamic acid exc

a)

Histidine

b)

Arginine

c)

Proline

d)

Asparagine

91.

PRPP sourse needed for purine and pyrimidinee synthesis:

a)

Dietary ribose

b)

HMP

c)

a and B

d)

None

92.

Nitrogen No. 9 purine ring is derived from

a)

Asparagine

b)

Glutamine

c)

Glycine

d)

Methionine

93.

The first purine nucleotide synthesized:

a)

AMP

b)

IMP

c)

GMP

d)

XMP

94.

Amino group attached to C6 of adenine derived from

a)

Asparagine

b)

Glutamine

c)

Glycine

d)

Aspartic

95.

CMP inhibits following enzs

a)

PRPP - synthase

b)

PRPP - glutamyl amido transfrase

c)

IMP dehydrogenase

d)

All of the above

96.

AMP inhibits the following enzs

a)

PRPP - synthase

b)

PRPP - glutamyl amido transfrase

c)

adenlosuccinate synthase

d)

All of the above

97.

The end product of purine catabolism is:

a)

Urea

b)

Uric acid

c)

Ammonia and CO2

d)

Amino acids

98.

Metabolic Gout is caused by:

a)

G6P deficiency

b)

Increased activity of PRPP synthase

c)

Deficiency of HGPRTase

d)

All of the above

99.

Hypuricemia is caused by:

a)

G6P deficiency

b)

Increased activity of PRPP synthase

c)

Adenosine deaminnase deficiency

d)

All of the above

100.

Share in pyrmidine biosynthesis

a)

Aspartic Glutamine CO2

b)

Asparagine Glutamine CO2

c)

Aspartic Glutamic CO2

d)

All of the above

101.

Carbamoyl phosphate synthase II, diffrrs than that of urea cycle by

a)

It is cyoslic while that of urea synthesis is mitochondrial

b)

The source kf nitrogen is glutamine while that of urea is ammonia

c)

It doesn't require N-acetyl glutamine as acivator

d)

All of the above

102.

The end product of uracil catabolism are

a)

Uric acid

b)

B alanine, ammonia and CO2

c)

Urea

d)

None

103.

The end product of thymine catabolism are

a)

Uric acid

b)

B aminoisobutyric , ammonia and CO2

c)

Urea

d)

None

104.

Allopurinol is a synthesic base analog used in treatment of gout cuz

a)

Prevent purin synthase

b)

Inhibits adenosine deaminase

c)

Competes with xanthine and hypoxanthine on xanthine oxidase

d)

All of the above

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