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Pathology Supple Set C

Total questions: 20

Worksheet time: 7mins

Name
Class
Date
1.

1-4 weeks after group A beta hemolytic nephritogenic streptococci infection of the pharynx or impetigo is etiology of.

a)

Acute Proliferative GN

b)

Chronic Proliferative GN

c)

Acute Poststreptococcal GN

d)

Chronic Poststreptococcal GN

2.

Pathogenesis of APGN.

a)

Glomerular deposition of immune complex from circulating or planted antigen

b)

Proliferating & damage of glomerular cells

c)

Infiltration of leucocytes, neutrophils

d)

Hyper-complementemia

3.

Focal proliferative GN.

a)

Down syndrome

b)

Goodpasture's syndrome

c)

SLE

d)

Diabetes Mellitus

4.

Microscopic changes in the glomeruli of APGN.

a)

Glomerulus show diffuse hypercellularity

b)

Glomerulus show diffuse hypocellularity

c)

Caused by intracapillary luecocytes (neutrophils), and proliferation of intrinsic glomerular cells

d)

Caused by extracapillary luecocytes (neutrophils), and proliferation of extrinsic glomerular cells

5.

Definition of hydronephrosis.

a)

Dilatation of renal pelvis and calyces associated with progressive hypertrophy of kidney due to obstruction to inflow of urine.

b)

Dilatation of renal pelvis and calyces associated with progressive atrophy of kidney lead to obstruction to outflow of urine.

c)

Dilatation of renal pelvis and calyces associated with progressive atrophy of kidney due to obstruction to inflow of urine.

d)

Dilatation of renal pelvis and calyces associated with progressive atrophy of kidney due to obstruction to outflow of urine.

6.

Pathogenesis of hydronephrosis.

a)

1. partial obstruction decrease GFR

2. filtrate diffuse back affected pelvi-calyces

3. overwork and dilated causing renal atrophy

4. obstruction cause interstitial inf. reaction lead to fibrosis

b)

1. Fully obstruction decrease GFR

2. filtrate diffuse back affected pelvi-calyces

3. overwork and dilated causing renal atrophy

4. obstruction cause interstitial inf. reaction lead to fibrosis

c)

1. partial obstruction decrease GFR

2. filtrate diffuse back affected pelvi-calyces

3. overwork and dilated causing renal hypertrophy

4. obstruction cause interstitial inf. reaction lead to fibrosis

d)

1. partial obstruction increase GFR

2. filtrate diffuse back affected pelvi-calyces

3. overwork and dilated causing renal atrophy

4. obstruction cause interstitial inf. reaction lead to fibrosis

7.

Label.

a)

A : Double layered epithelium (tall columnar & basal cells)

B : Fibromuscular stroma

C : Papillary infolding

D : Corpora amylacea

b)

A : Double layered epithelium (simple columnar & basal cells)

B : Corpora amylacea

C : Papillary infolding

D : Fibromuscular stroma

c)

A : Double layered epithelium (simple columnar & basal cells)

B : Fibromuscular stroma

C : Papillary infolding

D : Corpora amylacea

d)

A : Fibromuscular stroma

B : Double layered epithelium (simple columnar & basal cells)

C : Papillary infolding

D : Corpora amylacea

8.

Method of microbial spreading.

a)

Air-borne

b)

Direct

c)

Hematogenous

d)

Local extension

9.

Choose the correct.

a)

Bacterial : Turbid

Viral : Clear

Fungal : Clear

b)

Bacterial : Clear

Viral : Clear

Fungal : Turbid

c)

Bacterial : > 40

Viral : Normal-Low

Fungal : Normal

d)

Bacterial : < 40

Viral : Normal

Fungal : Normal-Low

10.

Cloudy appearance showing.

a)

Platelets

b)

Neutrophils

c)

Bacterial

d)

Glucose

11.

Increase opening pressure cause swelling of meninges and lead to.

(choose most correct)

a)

haemorrhage

b)

pus formation

c)

Headache

d)

Scars

12.

Bacteria consume glucose, so glucose level will be?

a)

increase

b)

decrease

13.

Immune response cause WBC's count?

a)

increase

b)

decrease

14.

Elevated protein level due to?

a)

Formation of BBB

b)

Breakdown of BBB

15.

Contiguos spread.

a)

Otitis media

b)

Sunisitis

c)

Endocarditis

d)

Dental infection

16.

Hematogenous spread.

a)

Pericarditis

b)

Pulmonary embolism

c)

Endocarditis

d)

Lung abscess

17.

Immunocompromised State.

a)

HIV/AIDS

b)

Chemotherapy

c)

Pre-transplant patients

d)

Diabetes Mellitus

18.

Trauma or Neurosurgery.

a)

Ventriculoperitoneal shunt

b)

Penetrating head injuries

c)

Craniotomy

d)

Lobatomy

19.

Complications of bacteria infection.

a)

Hydronephrosis

b)

Hydrocephalus

c)

Cerebral abscess

d)

Sensorineural hearing loss

20.

How bacteria infection developed into an abscess.

a)

1. Microb enter brain via (contigous/hematogenous spread)

2. Aided by patient increase immunity

3. Early stage (cerebritis)

4. Late stage (abscess formation)

b)

1. Microb enter brain via (contigous/hematogenous spread)

2. Aided by patient reduce immunity

3. Early stage (abscess formation)

4. Late stage (cerebritis)

c)

1. Microb enter brain via (contigous/hematogenous spread)

2. Aided by patient increase immunity

3. Early stage (abscess formation)

4. Late stage (cerebritis)

d)

1. Microb enter brain via (contigous/hematogenous spread)

2. Aided by patient reduce immunity

3. Early stage (cerebritis)

4. Late stage (abscess formation)