WorksheetsDone
Total questions: 38
Worksheet time: 35mins
Cause of increased BP is unknown, not related to another medical condition is..
(a)
HTN caused by comorbid conditions
(a)
Which system involves the kidney and its blood volume control?
(a)
An example of this system includes elevated aldosterone levels
(a)
Decreased elasticity and receptor sensitivity to NE are function of what system?
(a)
What kind of drugs can increase BP and are typically avoided with patients who have HTN?
(a)
High BP outside of medical environment
(a)
High BP only within a medical environment
(a)
What is the criteria for diagnosis of HTN
(a)
SBP <120 AND DBP <80
(a)
SBP 120-129 AND DBP <80
(a)
SBP 130-139 OR DBP 80-89
(a)
SBP > or = 140 OR DBP > or = 90
(a)
SBP <90 OR DBP <60
(a)
BP target goal
(a)
Blocks NA+/Cl- symporter on DCT of kidney to decrease blood volume
(a)
ADR in Thiazide Diuretics
Dehydration
Increase Ca2+ in blood
Hyperuricemia
Decrease K+ absorption
Decrease uric acid concentration
Inhibit conversion of Ang I to Ang II, inhibits bradykinin breakdown
(a)
ADR of ACEIs
Angioedema
Decreased GFR
Cough
Rash
Hyperkalemia
Block Ang II action at AT1 receptors
(a)
ADRs of ARBs
Headache
Hypokalemia
Dizziness
Hyperkalemia
Decreased GFR
Block L-type Ca2+ channels on vasculature
(a)
ADRs of DHP CCBs
Headache
Peripheral edema
Reflex tachycardia
N/V
Increased GFR
Block L-type Ca2+ channels on SA/VA nodes of heart
(a)
ADRs of Non-DHP CCBs
Decreased HR
Peripheral edema
Headache
Increases GFR
Block B adrenergic receptors; decrease HR, Renin, and Sympathetic outflow
(a)
ADRs of Beta-Blockers
Fatigue
Bradycardia
Sedation
SOB
Sleep disturbances
Inhibit NA/K/2Cl- to lower blood volume
(a)
ADRs of Loop diuretics
Electrolyte embalances
Increased K+
Increased NA+
Increased Ca2+
Inhibit aldosterone-mediated stimulation of ENaC and Na/ATpase synthesis; Increase Na+ decrease K+
(a)
ADRs of Aldosterone Antagonists
N/V
Hyperkalemia
Headache
Gynecomastia
Alpha-2 agonist; Decrease NE release and sympathetic activity
(a)
ADR of Centrally-acting Antihypertensives
Sedation
Bradycardia
Constipation
Dizziness
N/V
Block Alpha-1 receptors on vasculature
(a)
ADRs of Alpha-1 Antagonists
Fainting
Dizziness
N/V
Runny nose
Sexual dysfunction
Open K+ channels = hyperpolarization of vascular smooth muscle = vasodialtion
(a)
ADRs of Direct vasodilators
Reflex tachycardia
Hypertrichosis
Decreased GFR
Bradycardia
ALL Antihypertensive medication have this ADR in common
(a)
