WorksheetsQuiz on OP Poisoning and Related Compounds
Total questions: 50
Worksheet time: 17mins
Which enzyme is inhibited by organophosphate compounds?
Monoamine oxidase
Acetylcholinesterase
Cytochrome oxidase
Sucrase
The antidote of choice in OP poisoning is:
Pralidoxime
Atropine
Ethanol
Magnesium sulfate
The classical clinical mnemonic for OP poisoning includes:
SLUDGE
ABCDE
SAMPLE
RACE
SLUDGE stands for:
Salivation, Lacrimation, Urination, Diarrhea, Gastrointestinal upset, Emesis
Sweating, Lethargy, Unconsciousness, Diarrhea, Giddiness, Emesis
Shock, Liver toxicity, Unconsciousness, Dehydration, Gastrointestinal upset, Encephalitis
Salivation, Liver toxicity, Urination, Dystonia, Generalized edema
Occupational exposure to OP compounds is most common in:
Industrial workers
Farmers
Healthcare workers
Teachers
Which drug reactivates acetylcholinesterase in OP poisoning?
Atropine
Pralidoxime
Naloxone
Phenobarbital
DUMBBELSS in OP poisoning stands for all except:
Diarrhea
Miosis
Bradycardia
Tachycardia
Mechanism of OP toxicity primarily involves:
Irreversible inhibition of acetylcholinesterase
DNA synthesis suppression
Enzyme induction
Competitively antagonizing GABA
In OP poisoning, atropine is administered until:
Pupils dilate
Heart rate is >80/min and chest is clear
Sweating stops
Patient is sedated
Which of the following is NOT used in OP poisoning therapy?
Atropine
Pralidoxime
Activated charcoal
Acepromazine
Carbamate compounds inhibit which enzyme?
Monoamine oxidase
Acetylcholinesterase
Cytochrome oxidase
Glutathione peroxidase
Carbamate toxicity lasts for:
Several weeks
Months
Less than 24 hours
Indefinitely
Symptoms of carbamate poisoning resemble those of:
Alkaloid poisoning
Organophosphate poisoning
Metal toxicity
Vitamin deficiency
The primary treatment for carbamate poisoning is:
Broad-spectrum antibiotics
Decontamination and supportive care
High-dose steroids
Insulin therapy
Which antidote is occasionally used for carbamate toxicity?
Atropine
Diazepam
Naloxone
Phenobarbital
Pralidoxime is:
Always required in carbamate poisoning
Recommended only for severe cases
Never used in carbamate poisoning
Used for all pesticide poisoning
The most common cause of death in carbamate poisoning is:
Liver failure
Respiratory failure
Kidney failure
Heart attack
Which among the following is a carbamate pesticide?
Parathion
Captan
DDT
Malathion
Nicotinic effects of carbamate poisoning include:
Tachycardia
Muscle fasciculations
Bronchospasm only
None
Carbamates cause:
Covalent binding to acetylcholinesterase
Reversible binding to acetylcholinesterase
No enzyme inhibition
Permanent toxicity
Organochlorines are classified as:
Biodegradable
Persistent organic pollutants
Insect repellents
Parasite inhibitors
Which is an example of an organochlorine pesticide?
DDT
Malathion
Carbaryl
Aldicarb
Organochlorine exposure is associated with:
Neurotoxicity
Hypercholesterolemia
Diabetes mellitus
Hypothyroidism
The collective term DDX refers to:
DDT, DDE, DDD
DDT, DDA, DDP
DDA, DDB, DDC
DDA, DDE, DDO
Organochlorines act mainly by:
Inhibiting sodium channels
Activating potassium channels
Impairing GABA synthesis
Blocking calcium channels
Which organ system is primarily affected by chlorinated cyclodienes?
Hepatic
Renal
Pulmonary
Gastrointestinal
Most organochlorines are:
Quickly biodegradable
Non-persistent
Rapidly excreted
Environmentally persistent
Health risks of organochlorines include all except:
Endocrine disruption
Carcinogenicity
Neurotoxicity
Hyperglycemia
Organochlorines are mainly used as:
Herbicides
Antibiotics
Pesticides
Fungicides
Alternatives to organochlorine pesticides include:
Organo-phosphates
Alkaloids
Heavy metals
None
Aluminium phosphide poisoning primarily occurs due to:
Inhalation
Ingestion
Contact with skin
Injection
Fatal dose of aluminium phosphide is:
10 gm
5 gm
3 gm
1 gm
Aluminium phosphide poisoning is associated with characteristic:
Apple odor
Garlic odor
Fish odor
Banana odor
Phosphine gas is liberated from aluminium phosphide when:
Heated
Exposed to dry air
Exposed to moisture
Kept in sunlight
Mechanism of toxicity of aluminium phosphide includes:
Inhibition of acetylcholinesterase
Inhibition of cytochrome oxidase in mitochondria
Stimulation of beta-adrenergic receptors
Altered DNA methylation
Clinical features of aluminium phosphide poisoning include:
Myocarditis, hypotension, and shock
Chronic liver disease
Seizures only
Hypokalemia only
The recommended breath test for phosphine gas detection involves:
Silver nitrate turning black
Sulfuric acid forming white fumes
Potassium permanganate forming violet color
Copper sulfate forming green color
Garlic odor in aluminium phosphide poisoning is also seen in:
Arsenic poisoning
Carbamate poisoning
Cyanide poisoning
Lead poisoning
Complications of aluminium phosphide poisoning commonly involve:
Acute liver failure
Acute respiratory failure
Cardiovascular collapse
Renal failure
Aluminium phosphide poisoning can cause all except:
Subendocardial infarction
Esophageal stricture
Accumulation of acetylcholine at NM junction
Metabolic acidosis
OP compounds used as pesticides include:
Parathion
Aldrin
DDT
Captan
Carbamate pesticides include:
Aldicarb
Parathion
DDT
Lindane
Organochlorine induces what type of toxicity?
Renal
Neurotoxicity
Haematological
Cardiac
Aluminium phosphide poisoning causes metabolic acidosis mainly due to:
Lactic acid accumulation
Ketosis
Uric acid accumulation
Hypoglycemia
Most OP and carbamate poisonings affect the:
Muscarinic and nicotinic receptors
Only nicotinic receptors
Only muscarinic receptors
Only adrenergic receptors
The main focus in acute carbamate poisoning treatment is:
Maintenance of airway
Renal support
Antibiotic therapy
Diuretic therapy
Organochlorine pesticides are best analyzed using:
Electron Capture Detector
UV spectrometry
Chromatography
Radioimmunoassay
DDT is referred to chemically as:
Dichlorodiphenyltrichloroethane
Dimethyltrichloroethane
Diaminotriphenylethane
Dehydrodiphenyltrichloroethane
Phosphine gas is confirmed in breath by which test?
Silver nitrate paper
Benzidine reaction
Melzer's reagent
Copper sulfate paper
The fatal period of aluminium phosphide poisoning is typically:
72 hours
24 hours
48 hours
12 hours
