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Muscle and Nervous Tissue Quiz

Total questions: 20

Worksheet time: 10mins

Name
Class
Date
1.

A patient is experiencing muscle weakness and fatigue. Using your knowledge of skeletal muscle structure, reason which component of the muscle fiber is most likely malfunctioning if ATP production is impaired, and explain your choice.

a)

Sarcolemma

b)

Mitochondria

c)

Myofibrils

d)

T-tubules

2.

Given the unique intercalated discs found in cardiac muscle, plan an experiment to demonstrate how these structures contribute to the synchronized contraction of the heart. Which method would best provide evidence for their function?

a)

Isolate cardiac muscle cells and observe contraction rates individually

b)

Use fluorescent markers to track electrical impulses across intercalated discs

c)

Measure ATP levels in cardiac muscle tissue

d)

Compare contraction strength between cardiac and skeletal muscle

3.

A researcher is studying smooth muscle contraction in the digestive tract. Based on the characteristics of smooth muscle, which reasoning best explains why smooth muscle can sustain contractions for longer periods than skeletal muscle?

a)

Smooth muscle has more mitochondria

b)

Smooth muscle uses less ATP per contraction

c)

Smooth muscle fibers are multinucleated

d)

Smooth muscle contracts only voluntarily

4.

You are tasked with designing a drug that targets synaptic transmission in neurons. Using evidence from the structure and function of synapses, which component should the drug most strategically target to reduce neuronal communication?

a)

Axon hillock

b)

Synaptic vesicles

c)

Myelin sheath

d)

Nissl bodies

5.

A muscle biopsy reveals a high density of myofibrils and a regular arrangement of sarcomeres. Reason which type of muscle tissue this sample most likely represents and justify your answer.

a)

Cardiac muscle

b)

Skeletal muscle

c)

Smooth muscle

d)

Nervous tissue

6.

A patient’s heart is not pumping efficiently. Using your understanding of cardiac muscle function, reason which structural feature is most likely compromised and explain its impact.

a)

Lack of striations

b)

Damaged intercalated discs

c)

Reduced number of nuclei per cell

d)

Absence of myofibrils

7.

Given the involuntary nature of smooth muscle, plan a strategy to test the effect of autonomic nervous system stimulation on smooth muscle contraction in the bladder.

a)

Apply voluntary electrical stimulation to the muscle

b)

Use autonomic neurotransmitters and measure contraction response

c)

Block all neural input and observe contraction

d)

Compare contraction with skeletal muscle under voluntary control

8.

A neuroscientist wants to increase the speed of action potential transmission in neurons. Using evidence from neuron structure, which modification would most effectively achieve this goal?

a)

Increase the number of dendrites

b)

Add more myelin to the axon

c)

Enlarge the cell body

d)

Increase synaptic cleft width

9.

A muscle cell is unable to contract despite receiving a nerve impulse. Using your knowledge of skeletal muscle structure, reason which part of the excitation-contraction coupling process is most likely disrupted.

a)

Release of calcium from the sarcoplasmic reticulum

b)

Breakdown of acetylcholine in the synaptic cleft

c)

Reuptake of sodium ions

d)

Formation of myelin sheath

10.

A patient with arrhythmia is found to have abnormal gap junctions in cardiac muscle. Using evidence, explain how this structural abnormality could lead to irregular heartbeats.

a)

Gap junctions prevent muscle contraction

b)

Gap junctions disrupt electrical signal synchronization

c)

Gap junctions increase ATP production

d)

Gap junctions cause muscle hypertrophy

11.

A scientist is comparing the regeneration capacity of skeletal and smooth muscle. Using reasoning, which muscle type is more likely to regenerate after injury and why?

a)

Skeletal muscle, due to satellite cells

b)

Smooth muscle, due to mitotic activity

c)

Cardiac muscle, due to intercalated discs

d)

Skeletal muscle, due to multinucleation

12.

A neuron is unable to transmit signals efficiently. Using evidence from synapse structure, which malfunction would most likely cause this problem?

a)

Excessive neurotransmitter release

b)

Blocked receptor sites on the postsynaptic membrane

c)

Increased axon diameter

d)

Enhanced dendritic branching

13.

A muscle cell is observed to contract rhythmically and involuntarily, with branching fibers and a single nucleus per cell. Using reasoning, identify the muscle type and justify your answer.

a)

Skeletal muscle

b)

Cardiac muscle

c)

Smooth muscle

d)

Nervous tissue

14.

A patient is unable to move a limb voluntarily. Using your knowledge of skeletal muscle structure and function, reason which neural component is most likely damaged.

a)

Sensory neuron

b)

Motor neuron

c)

Interneuron

d)

Schwann cell

15.

A researcher is investigating the effect of neurotransmitter reuptake inhibitors on synaptic transmission. Using evidence, predict the outcome on neuronal communication.

a)

Decreased synaptic transmission

b)

Increased duration of neurotransmitter action

c)

Reduced action potential frequency

d)

Enhanced myelin formation

16.

A smooth muscle cell is exposed to a calcium channel blocker. Using reasoning, predict the effect on muscle contraction and explain why.

a)

Increased contraction due to more calcium

b)

Decreased contraction due to less calcium influx

c)

No change in contraction

d)

Increased contraction due to ATP synthesis

17.

A patient with multiple sclerosis has demyelinated neurons. Using evidence from neuron structure and function, reason how this affects signal transmission.

a)

Signals travel faster

b)

Signals are blocked at the synapse

c)

Signals travel slower and may be lost

d)

Signals are unaffected

18.

A muscle cell is found to lack striations and contracts slowly. Using reasoning, identify the muscle type and explain your choice.

a)

Skeletal muscle

b)

Cardiac muscle

c)

Smooth muscle

d)

Nervous tissue

19.

A scientist is studying the effect of acetylcholine on skeletal muscle contraction. Using evidence, explain how acetylcholine initiates contraction at the neuromuscular junction.

a)

Acetylcholine binds to receptors on the sarcolemma, triggering depolarization

b)

Acetylcholine breaks down ATP

c)

Acetylcholine inhibits calcium release

d)

Acetylcholine forms myelin

20.

A patient’s cardiac muscle cells are unable to contract in unison. Using reasoning, which structural feature is most likely defective and why?

a)

Sarcomeres

b)

Intercalated discs

c)

Myofibrils

d)

Nissl bodies