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AV 21 final

Total questions: 30

Worksheet time: 16mins

Name
Class
Date
1.

Which receptor activation in airway smooth muscle most directly causes bronchoconstriction and increased mucus secretion?

a)

Beta-2 adrenergic receptors in airways

b)

Alpha-1 adrenergic receptors in airways

c)

Nicotinic receptors on airway glands

d)

M3 muscarinic receptors in airways

2.

Which drug class is intended for immediate relief during an acute asthma episode?

a)

Leukotriene pathway antagonists

b)

Short-acting β2-agonist bronchodilators

c)

Glucocorticoids for long-term reduction

d)

Anti-IgE monoclonal antibody therapy

3.

A patient has frequent nocturnal awakenings due to asthma. Which therapy category should be prioritized to reduce attack frequency?

a)

Control therapy with anti-inflammatory agents

b)

Rescue xanthine preparations before bedtime

c)

Antimuscarinic inhalers used only during attacks

d)

Quick relief bronchodilators for episodic attacks

4.

Which class of bronchodilators primarily activates β2 adrenoceptors to produce rapid relief of bronchoconstriction?

a)

β2 adrenoceptor agonists

b)

Antimuscarinic agents

c)

Xanthine derivatives

d)

Leukotriene modifiers

5.

A patient inhales a β2 agonist. Which sequence best explains how bronchodilation occurs?

a)

β2 activation → ↓ cAMP → smooth muscle contraction

b)

Mast cell degranulation → ↑ cAMP → airway dilation

c)

β2 activation → adenyl cyclase stimulation → ↑ cAMP

d)

Muscarinic blockade → ↓ cAMP → airway relaxation

6.

Which statement best describes epinephrine as a bronchodilator?

a)

Potent bronchodilator with prolonged action

b)

Weak bronchodilator with slow onset

c)

Selective β2 bronchodilator with minimal side effects

d)

Potent bronchodilator with rapid onset

7.

Which clinical scenario makes non-selective β-agonists unsuitable in asthma management?

a)

Normal glucose tolerance status

b)

Mild intermittent asthma symptoms

c)

Stable sinus rhythm without tachycardia

d)

Hypertension or heart failure risk

8.

Which route is primarily used to administer selective beta-2 agonists for asthma management?

a)

Subcutaneous depot injection

b)

Inhalation via inhaler or nebulizer

c)

Intravenous continuous infusion

d)

Oral tablets daily dosing

9.

A patient presents with an acute asthma attack needing rapid relief. Which drug class and examples are most appropriate?

a)

Anticholinergics: ipratropium, tiotropium

b)

Short-acting beta-2 agonists: salbutamol, terbutaline

c)

Inhaled corticosteroids: budesonide, fluticasone

d)

Long-acting beta-2 agonists: salmeterol, formoterol

10.

Explain why long-acting beta-2 agonists are combined with inhaled corticosteroids for chronic control rather than used alone for acute relief.

a)

They have slow onset and long duration, not for acute relief

b)

They cause bronchospasm without steroids

c)

They reduce steroid absorption in the lungs

d)

They only work overnight but not daytime

11.

Which statement best differentiates short-acting from long-acting selective beta-2 agonists?

a)

Long-acting have onset 1–2 min and 1–2 hr duration

b)

Short-acting have onset 15–30 min and 4–6 hr duration

c)

Long-acting are first-line for acute exacerbations

d)

Short-acting are given only orally, never inhaled

12.

Class IV antiarrhythmics primarily act at the AV and SA nodes to slow conduction and prolong the effective refractory period. Which pair represents these Class IV calcium channel blockers?

a)

Amiodarone and ibutilide

b)

Adenosine and lidocaine

c)

Verapamil and diltiazem

d)

Sotalol and quinidine

13.

What is a primary function of an implantable cardiac defibrillator (ICD)?

a)

Maintain sinus rhythm after cardioversion

b)

Prevent bradycardia during myocardial infarction

c)

Deliver continuous pacing to treat heart block

d)

Automatically detect and treat ventricular fibrillation

14.

In atrial fibrillation, which beta blocker is commonly used in the treatment map?

a)


Esmolol ultrashort agent

b)


Propranolol nonselective agent

c)


Metoprolol selective agent

d)

Acebutolol partial agonist

15.

Which alternative agent is shown for AV nodal reentry besides beta blockers and verapamil?

a)

Lidocaine anesthetic option

b)

Digoxin cardiac glycoside option

c)


Amiodarone iodine-rich option

d)


Epinephrine adrenergic option

16.

A 60-year-old man with myocardial infarction shows ventricular tachycardia on ECG. Immediate treatment should be

a)

Amiodarone

b)

Lidocaine

c)

Quinidine

d)

Verapamile

17.

Which therapeutic strategy directly addresses myocardial oxygen demand in angina management?

a)

Stimulating platelet aggregation pathways

b)

Increasing hematocrit and blood viscosity

c)

Reducing heart rate and contractility

d)

Enhancing coronary collateral growth

18.

All of the following drug classes can reduce myocardial oxygen demand in angina Except:

a)

CCB

b)

Antiplatelet drugs

c)

Nitrates

d)

Beta blockers

19.

Which nitrate is primarily formulated for longer duration prophylaxis rather than acute relief?

a)

Glyceryl trinitrate

b)

Isosorbide mononitrate

c)

Short-acting isosorbide dinitrate

d)

Sublingual nitroglycerin

20.

Which two overarching strategies guide drug treatment of angina pectoris?

a)

Increase oxygen delivery to myocardium

b)

Increase myocardial glycogen stores

c)

Reduce myocardial oxygen demand

d)

Lower plasma glucose concentration

21.

Co-administration of sildenafil with nitroglycerin is contraindicated primarily because it increases the risk of:

a)

Dangerous hypotension and syncope

b)

Rebound hypertension and angina

c)

Methemoglobinemia and cyanosis

d)

Severe hypokalemia and arrhythmia

22.

Which effects decrease myocardial oxygen demand with beta blockers? Select all that apply.

a)

Lowered cardiac output

b)

Enhanced β2-mediated bronchodilation

c)

Decreased contractility

d)

Reduced heart rate

e)

Increased systolic blood pressure

23.

Excessive sodium and water retention leading to increased blood volume refers to which mechanism?

a)

Volume Overload

b)

Increased Peripheral Resistance

c)

Impaired Vasodilation

24.

Which effects are reduced as a result of ACE inhibition?

a)

Heart rate and myocardial contractility

b)

Renin release and natriuresis

c)

Vasoconstriction and aldosterone secretion

d)

Platelet aggregation and thrombin generation

25.

Which statement best reflects this mechanism of ARNIs?

a)

They solely inhibit neprilysin, causing sodium retention and vasoconstriction

b)

They act as beta-blockers to reduce heart rate without affecting RAAS or natriuretic peptides.

c)

They combine RAAS inhibition with enhanced natriuretic peptide effects, producing vasodilation and reduced fibrosis.

d)


They increase aldosterone activity to promote fluid retention.

26.

Which hypertension phenotypes are shown to decrease with ARNI therapy? Except:

a)

Uncontrolled (resistant) hypertension

b)


Systolic hypertension

c)


Orthostatic hypotension

d)

Nocturnal hypertension

27.

What are some characteristics of direct thrombin inhibitors

a)

decreased conversion of fibrinogen to fibrin

b)

increased endothelial cell/platelet activation

c)

decreased endothelial cell/platelet activation

d)

decreased activation of factor XIII

e)

increased conversion of fibrinogen to fibrin

28.

What are some ways we can reverse bleeding ADRs due to Warfarin? select all possible ways

a)

vit K

b)

Aspirin

c)

Plasma transfusion

d)

Rifampin

29.

All of the following drugs are anticoagulants, Except:

a)

Heparins

b)

Clopidogrel

c)

Warfarin

d)

Direct thrombin inhibitors

e)

Direct factor Xa inhibitors

30.

All of the following are treatment strategies to prevent or reverse thrombus formation, Except:

a)

Anticoagulants

b)


Inhibit platelet activity

c)

Vitamin K supplements

d)

Dissolve clot