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OCD Quiz

Total questions: 19

Worksheet time: 19mins

Name
Class
Date
1.

Which component is the obsessional part of OCD

a)

Cognitive component

b)

Behavioural component

c)

Emotional Component

2.

Which component is the compulsive part of OCD

a)

Cognitive component

b)

Emotional Component

c)

Behavioural Component

3.

Which method did Groothest use to provide support for OCD being linked to biology

a)

Questionnaire

b)

Unstructured interviews

c)

Case study

d)

Meta-analysis

4.

Which type of twin did Groothest find had a higher concordance rate for OCD?

a)

MZ (identical twins)

b)

DZ (non identical twins)

c)

PZ

5.

When the concordance rate is not 100% what does this suggest is also involved in the condition?

a)

Genetics

b)

Environmental triggers

c)

Animals

6.

Children who have family members with OCD usually show the following

a)

They too have the exact same OCD which suggest it is learned

b)

They have OCD but it differs to the OCD their family member has which suggests it is biological

c)

They show no sign of having OCD

7.

What 2 genes have been linked with the development of OCD?

a)

BERT Gene

b)

GERT Gene

c)

SERT Gene

d)

COMT Gene

8.

What % of OCD sufferers respond well to anti-depressants (SSRIs)?

a)

10%

b)

20%

c)

50%

d)

90%

9.

What gene leads to high levels of dopamine which could also be a cause of OCD?

a)

BOMT Gene

b)

HOMT Gene

c)

COMT Gene

d)

SOMT Gene

10.

In which 2 ways could you criticise the biological explanation for OCD

a)

Too deterministic

b)

Too holistic

c)

Too reductionist

d)

Too idiographic

11.

Which part of the brain is thought to be leaky and allows trivial information to become obsessive thoughts

a)

Thalamus

b)

Frontal lobe

c)

Basal Ganglia

d)

Temporal Lobe

12.

A second biological treatment for OCD

a)

Psychotherapy

b)

Cognitive Behavioural Treatment

c)

REBT

d)

Deep Brain Stimulation

13.

What % of population suffers from OCD?

a)

15%

b)

10%

c)

5%

d)

2%

14.

The SERT gene leads to what happening?

a)

No serotonin being released into synaptic gap at all

b)

Too much serotonin being released into synaptic gap so the receptors are overwhelmed

c)

Serotonin being taken back into the pre-synaptic neuron too quickly so there is not enough serotonin in synapse for long enough

15.

How do anti-depressants (SSRIs) help with OCD?

a)

They stop serotonin from being released into the synapse

b)

They block the re-uptake channels on the pre-synaptic terminal button to allow more serotonin in the synapse for longer

c)

The block the receptor sites on the post synaptic dendrites

d)

They attach to the axon to make the electrical impulse travel faster

16.

2 pieces of evidence to support the role of the basal ganglia in the development of OCD

a)

PET scans show more activity in the thalamus which suggests the basal ganglia is faulty.

b)

COMT gene is faulty

c)

Deep brain stimulation helps to treat OCD

d)

Having stressful events in life

17.

What 2 results did Lewis find to support the biological explanation for OCD

a)

Studied MZ and DZ twins

b)

Found 37% of OCD sufferers had a parent with OCD

c)

Found 21% of OCD sufferers had a sibling with OCD

d)

Found that people with OCD had a pet dog with OCD

18.

Evidence for brain dysfunction being involved in OCD?

a)

Person with OCD has a high chance of having a parent with OCD

b)

PET scans and found more activity in the thalamus - shows basal ganglia is leaky

c)

Higher concordance rate for OCD for MZ twins

19.

Why is it difficult to establish if brain dysfunction is the cause of OCD?

a)

Unethical to do well controlled lab exp and make parts of brain faulty to see if causes OCD

b)

Researcher can't get a big enough sample size