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Hypertension and Kidney Disease

Total questions: 20

Worksheet time: 10mins

Name
Class
Date
1.
In the collecting duct, what is reabsorbed? 
a)
Ions like Na+ and Cl-
b)
Nutrients like glucose and amino acids 
c)
Substances like drug metabolites and waste 
d)
Water 
2.

What is "normal" blood pressure

a)

120/110 mmHg

b)

120/80 mmHg

c)

100/80 mmHg

d)

140/80 mmHg

3.

A diastolic pressure of 80 - 89

a)

normal blood pressure

b)

prehypertension

c)

hypertension stage 1

d)

hypertension stage 2

4.

A primary hypertension/ or essential hypertension is caused by:

a)

Medications such as pain relievers

b)

Diseases such as obesity and kidney disease

c)

Diabetes

d)

no known cause

5.

A patient is being treated with furosemide, a loop diuretic, for her blood pressure. Furosemide works by inhibiting the transport system of the thick ascending limb of the loop of Henle. Which of the following is most inhibited by this drug?

a)

Na+–K+–2Cl– symporters

b)

Na+-Glucose symporter

c)

Na+-H+ symporter

d)

Na+-H+ antiporter

6.

Systemic hypertension leads to kidney injury by affecting renal------------- leading to chronic kidney disease

a)

vasculature

b)

glomerulus

c)

interstitium

d)

tubule

e)

all of the above

7.

The progression timeline of CKD as a result of systemic hypertension is as follows:

a)

Arteriolar sclerosis- Glomerulosclerosis- Tubular atrophy- Interstitial fibrosis

b)

Onset phase- Oliguric (anuric) phase- Diuretic phase- Recovery phase

c)

None of the above

d)

Both answers are correct

8.

This image demonstrates the histology of hypertensive kidney disease. Choose the correct answer/ answers

a)

Lesion A is glomerular collapse

b)

Lesion B is arteriolar sclerosis

c)

Lesion A is arteriolar sclerosis

d)

Lesion B is tubular atrophy

9.

Hypertension is present in ---------- of stage 4-5 CKD patients.

a)

23.3%

b)

35.8%

c)

84.1%

d)

59.9%

10.

Blood pressure in CKD patients may be affected by the following factor (s).

a)

Autoregulation

b)

K

c)

Parasympathetic system

d)

Humoral systems

11.

The pathogenesis of hypertension varies with the type of disease; glomerular versus -------- and acute versus -------.

(a)  

12.

The following is true as regard the pathogenesis of hypertension in acute glomerular disease. (1 or more answer)

a)

It results from dehydration, stimulation of RAAS and diminished release of ANP.

b)

It results from fluid overload

c)

It results from Na retention as a result of its enhanced reabsorption at the collecting duct.

d)

Relative resistance of the collecting tubule to ANP.

e)

Decreased activity of the Na-K-ATPase pump at the collecting tubule.

13.

The following statement (s) is/are true.

a)

Hypertension is common is vasculitis and scleroderma renal crisis.

b)

In acute vascular disease, hypertension results from suppression of the RAAS.

c)

In acute vascular disease, hypertension results from ischemia- induced activation of the RAAS.

d)

The difference in the mechanism of hypertension between glomerular and vascular disease may be of therapeutic importance.

14.

Hypertension in CKD patients may result from:

a)

A reduction in endothelium derived vasodilators e.g nitric oxide.

b)

The administration of erythropoietin.

c)

An increase in intracellular calcium induced by PTH excess.

d)

Calcification of the arterial tree.

e)

All of the above.

15.

Hypertension following renal transplantation may be due to ..(1 or more answer).

a)

Hyperlipidemia

b)

Donor hypertension

c)

Hepatitis C virus

d)

Renal artery stenosis

e)

Graft dysfunction

16.

The following is true as regard the treatment of hypertension in renal disease (1 or more answers).

a)

There is no role for non-pharmacological therapy in renal disease patients with hypertension.

b)

Angiotensin receptor blockers are drugs of choice in treating CKD patients with DM, hypertension and proteinuria.

c)

Combining ACEI and ARBS are considered best antihypertensive therapy for stage 4 CKD patients.

d)

Ambulatory blood pressure monitoring is of prognostic value in CKD patients.

17.

Which of the following statements about angiotensin II is false?

a)

It causes vasoconstriction of the efferent arteriole

b)

It increases the amount of albumin filtered by the glomeruli

c)

It is increased in renal acidosis

d)

It increases aldosterone production

e)

It is reduced in people with DM

18.

Furosemide acts on which part of the nephron?

a)

Proximal tubule

b)

Descending limn of the loop of Henle

c)

Ascending limb of the loop of Henle

d)

Distal tubule

e)

Collecting duct

19.

When the concentration of ADH increases,

a)

more urine is produced

b)

more salt is secreted by the nephron

c)

less urine is produced

d)

less water is reabsorbed by the nephron and collecting duct

e)

the specific gravity of the urine decreases

20.

How is Na+ reabsorbed?

a)

by receptor-mediated endocytosis

b)

by osmosis

c)

by active transport using ATP

d)

by diffusion

e)

by facilitated diffusion