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NSAIDs Dr. Khan part 2

Total questions: 29

Worksheet time: 29mins

Name
Class
Date
1.

The solution: inhibit COX-2 selectively

a)

high selectivity agents produced- increased MI and stroke risk, many pulled from market

b)

low selectivity agents produced- increased MI and stroke risk, many pulled from market

c)

high selectivity agents produced-low MI and stroke risk, many pulled from market

2.

First NSAID marketed as a selective COX-2 inhibitor (COX-2I) is:

a)

Celecoxib

b)

Aspirin

c)

Ibuprofen

3.

Celecoxib up to 35% increased risk of CV events

a)

True

b)

False

4.

COX-1:

TXA2

Promotes platelet aggregation and Hemostasis/Thrombosis

a)

Platelets

b)

Endothelial cells

5.

COX-2:

PGI2: inhibit platelet aggregation

a)

endothelial cells

b)

platelets

6.

•Uses: analgesic, antipyretic, CV conditions

•Oral, buffered, EC, chewable, rectal administration

a)

Aspirin

b)

Ibuprofen

c)

Diclofenac

d)

Meloxicam

7.

Is utilized for cardioprotection

a)

Aspirin

b)

Diclofenac

c)

Ibuprofen

d)

Meloxciam

8.

Unique amongst NSAIDs​:

Having Irreversible inhibition and ​Inhibition of prostaglandin and thromboxane synthesis​

a)

Aspirin

b)

Celecoxib

c)

APAP

d)

Ibuprofen

9.

Covalently inhibit COX enzymes by ACETYLATION

a)

Aspirin

b)

Ibuprofen

c)

Diclofenac

d)

Nabumetone

10.

Aspirin have Biosynthesis of both PGs and TX will be inhibited

a)

True

b)

False

11.

Aspirin induced asthma: NSAIDS/ASA cause more severe attacks.

Mechanism -> due to increased leukotrienes

a)

True

b)

False

12.

Avoid aspirin in children and teenagers with viral infections, because increase ­risk of Reye’s Syndrome (N/V, somnolence, hepatic encephalopathy, lethargy, confusion)

a)

True

b)

False

13.

Acetaminophen is alternative to patients who can’t use NSAIDs or in situations when anti-inflammatory effect is undesirable

a)

True

b)

False

14.

•Boxed warnings: risk of errors and hepatotoxicity

•Note: variable recommended MDD; always ≤4 grams/day

•FDA: not more than 325 mg of APAP per dose in combination products

a)

APAP

b)

ASA

c)

Ibuprofen

d)

Naproxen

15.

APAP Kinetics:

a)

–A: extensive absorption in intestine

–M: hepatic oxidation (minimal at therapeutic dose), conjugation*

–E: t1/2 = 2-3 hours, prolonged at toxic doses

b)

–A: extensive absorption in intestine

–M: liver oxidation (minimal at therapeutic dose), conjugation*

–E: t1/2 = 2-3 hours, prolonged at toxic doses

c)

–A: low absorption in intestine

–M: hepatic oxidation (minimal at therapeutic dose), conjugation*

–E: t1/2 = 2-3 hours, prolonged at toxic doses

d)

–A: extensive absorption in intestine

–M: hepatic oxidation (minimal at therapeutic dose), conjugation*

–E: t1/2 = 2-3 hours, not prolonged at toxic doses

16.

What are the ADRs cause APAP?

a)

rash

b)

insomnia

c)

constipation

d)

nausea

e)

vomiting

17.

Acetaminophen have a metabolism mostly ______________and sulfate conjugation (80-90%)

a)

glucuronidation

b)

glucorinic

c)

sulfation

d)

oxidation

18.

Oxidative metabolism in APAP: 10% via 2E1 & 1A2 à toxic metabolite via 2E1 1A2 (NAPQIà ->toxic metabolite

a)

Inactivated by glutathione

b)

activated by glutathione

c)

Inactivated by glucorinic acid

19.

APAP: Ethanol induces CYP2E1-> increased risk of hepatotoxicity with >2 drinks/day

a)

True

b)

False

20.

Acetaminophen can cause:

a)

Hepatotoxicity

b)

Livertoxicity

21.

Neuromuscular blockers-used during surgical procedures and in intensive care units

a)

True

b)

False

22.

Skeletal Muscle Relaxants-reduce spasticity in a variety of neurological conditions

a)

True

b)

False

23.

Skeletal muscle relaxants

a)

Soma

b)

Flexeril

c)

Zanaflex

d)

Motrin

24.

Carisoprodol (SOMA) Poor CYP2C19 metabolizers->Increase drug level

a)

True

b)

False

25.

_______________ is structurally related to tricyclic antidepressants and possess antimuscarinic effects.

Sedative, dry mouth

a)

Cyclobenzaprine

b)

Carisoprodol

c)

Tizanidine

d)

Ibuprofen

26.

MOA: Exerts effect at GABAB receptors (G protein coupled receptor)

a)

Baclofen

b)

Cyclobenzaprine

c)

Carisoprodol

d)

Diazepam

27.

Baclofen: hyperpolarization causes presynaptic inhibition by reducing Ca influx ® reduces release of excitatory neurotransmitters in the brain and spinal cord

a)

True

b)

False

28.

________________ is a congener of clonidine that has been studied for its spasmolytic actions. Has significant central alpha-2 agonist effect. Inhibits nociceptive transmission

a)

Zanaflex

b)

Lioresal

c)

Soma

d)

Valium

29.

___________ Blocks neuromuscular transmission by entering the nerve terminals, and inhibiting the release of acetylcholine.

a)

Botulinum toxin Type A (BOTOX)

b)

Cyclobenzaprine (FLEXERIL)

c)

Tizanidine (ZANAFLEX)

d)

Carisoprodol (SOMA)