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Session II: Epigenetic Regulation and Metabolism Interplay

Total questions: 10

Worksheet time: 6mins

Name
Class
Date
1.

Isocitrate dehydrogenase 1 (IDH1) is rarely found mutated in distinct tumor types.

a)

FALSE

b)

TRUE

2.

IDH1/2 mutation is one of the most frequent alterations involved in the development of extrahepatic cholangiocacinoma

a)

TRUE

b)

FALSE

3.

In genetically engineered mouse models, mIDH1 can drive the growth of cholangiocarcinoma via the paracrine regulation of anti-tumor immunity and cell-autonomous epigenetic reprogramming

a)

FALSE

b)

TRUE

4.

What type of response does AG120 treatment induce ?

a)

interferon alpha

b)

interferon beta

c)

interferon gamma

5.

A combined therapy of AG120 and anti-CTLA-4 inhibits tumor growth and protects mice from tumor recurrence.

a)

TRUE

b)

FALSE

6.

Hypoxia Inducible Factor (HIF) transcription factors are activated in hypoxia via activation of prolyl hydroxylases (PHDs)

a)

TRUE

b)

FALSE

7.

In in vitro human cancer cell studies, acute and prolonged hypoxia impact promoter accessibility at subsets of hypoxia driven genes and chromatin structure

a)

FALSE

b)

TRUE

8.

Which processes can change chromatin state ?

a)

Post-translational modification of histone tails

b)

Histone dimer eviction and histone variant incorporation

c)

Lipids oxidation

d)

ATP-dependent chromatin remodelling

9.

Hypoxia induces a redistribution of H3K4me3, specifically at promoter sites.

a)

TRUE

b)

FALSE

10.

Which JmjC enzymes are responsible for the redistribution of H3K4me3 ?

a)

KDM5 isoforms

b)

PHD isoforms

c)

KDM6 isoforms