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Mycology III

Total questions: 27

Worksheet time: 29mins

Name
Class
Date
1.

Victor noticed unusual brown-black discolourations on the palm of his right hand and presented to his local medical centre two weeks later. Victor has no other symptoms. The GP suspects he has a fungal infection and collected a skin scraping to send to diagnostic lab. Victor most likely has which of the following types of fungal infections?

a)

invasive mycosis

b)

superficial mycosis

c)

subcutaneous mycosis

d)

cutaneous mycosis

e)

none of the above

2.

Which of the following fungal species is most likely responsible for superficial mycosis?

a)

Aspergillus fumigatus

b)

Rhizopus spp.

c)

Candida albicans

d)

Crytococcus neoformans

e)

Dermatophytes

3.

Commensal bacteria play an important role in preventing the overgrowth of Candidia albicans. If the commensal bacteria are disturbed or absent, greater numbers of C. albicans can colonise the mucosal epithelium. This is due to:

a)

decreased mucus production by host cells

b)

increased secretion of short-chain fatty acids by epithelial cells

c)

reduction in shear forces

d)

increased expression of candidalysin by C. albicans

e)

reduced competition for adhesion sites on epithelial cells

4.

Which of the following strategies is NOT used by Candida albicans to evade innate immunity?

a)

shielding of PAMPS

b)

inhibition of neutrophil recruitment and neutrophil extracellular traps (NETs)

c)

escape from the phagosom

d)

biofilm formation

e)

resistance to antimicrobial peptides

5.

Which of the following is NOT a challenge encountered by Candida albicans when it enters a new host?

a)

fluid fluxes

b)

biofilm formation

c)

cytokines

d)

reactive oxygen species

e)

pH changes

6.

Which of the following describes the role/s that Als3 protein plays in Candida albicans invasion?

a)

mediates yeast cells' attachment to host cell

b)

promotes the formation of an invasion pocket

c)

allows fungal hypha to bind to E-cadherin

7.

Advantages to the yeast from biofilm formation include:

a)

protection from immune cell action

b)

physical protection by formation of an extracellular matrix

c)

act as reservoirs of infection through spore seeding

d)

all of the answers are correct

e)

none of the answers are correct

8.

Candida albicans biofilms produce a specialised type of cell that enables persistence. What is this type of cell called?

a)

macrophage cell

b)

persister cell

c)

epithelial cell

d)

yeast cell

e)

hyphal cell

9.

The Candida albicans switch from yeast to hypha is triggered by which of these stimuli?

a)

alkaline pH

b)

osmotic stress

c)

human serum

d)

low oxygen

e)

all of the answers are correct

10.

Which is the least severe mycoses (disease caused by fungi)

a)

superficial

b)

cutaneous

c)

subcutaneous

d)

invasive

11.

Which is the most severe mycoses (disease caused by fungi)

a)

superficial

b)

cutaneous

c)

subcutaneous

d)

invasive

12.

What are the requirements for fungal disease?

a)

exposure

b)

survival & growth of fungus

c)

damage to the host

d)

antibiotics in fungus

13.

What are fungal virulence factors?

a)

fungal attributes that directly or indirectly enhance invasiveness by promoting infection

b)

fungal attributes that only directly enhance invasiveness

c)

fungal attributes that directly or indirectly inhibit invasiveness by promoting infection

d)

fungal attributes that only indirectly enhance invasiveness

14.

What are the virulence factors that promote fungal colonisation?

a)

adhesins

b)

invasins

c)

broad metabolic flexibility

d)

stress resistance

e)

immune evasion

15.

What are virulence factors that damage the host?

a)

morphological transition

b)

secreted hydrolytic enzymes & toxins

c)

stress resistance

d)

immune evasion

e)

adhesins

16.

Select all that is correct about Candida albicans

a)

opportunistic

b)

colonies skin, mouth, genital mucosa, intestinal mucosa

c)

is thermally dimorphic

d)

grow as both yeast and hyphal form in human host

e)

main portal of entries are GI tract, intravascular catheters, genital tract

17.

What are the in vivo challenges for C. albicans

a)

bacterial microbiota

b)

shear forces

c)

chemical and physical stresses

d)

physical barriers

e)

innate immune mechanisms

18.

Commensal bacteria prevent the overgrowth of C. albicans by

a)

competition for adhesion sites

b)

secretion of short chain fatty acids

c)

secretion of quorum sensing molecules

d)

decreased pH from bacterial metabolites

e)

increased attack by macrophages

19.

T or F. C. albicans adhesion to host cells (yeast) is essential to colonisation and survival in host

(a)  

20.

T or F. C. albicans interactions usually occur between yeasts and epithelial cells & are mediated by passive forces

(a)  

21.

T or F. C. aldbicans adhesion to yeast is more adherent that hyphae

(a)  

22.

What are the two main mechanisms of cellular invasion?

a)

induced endocytosis

b)

active penetration

c)

host cell adhesion

d)

covalent attachment

23.

T or F. Induced endocytosis is initiated by binding of C. albicans invasions Ssa1 and/or Als3 to their host receptors

(a)  

24.

Select all that are correct about induced endocytosis

a)

occurs via recruitment of clathrin and cytoskeleton reorganisation

b)

forms an invasion pocket

c)

hypha invades pocket

d)

needs a carrier protein to bind

25.

Select all that are correct about active penetration

a)

secreted into invasion pocket created by invading hypha

b)

damages epithelial membranes

c)

triggers danger response signalling pathways

d)

activates epithelial immunity

e)

uses saps, lipases, phospholipases, candidalysin

26.

What are the two main ways that Candida albicans evade the host immune response?

a)

escape from phagocytic cells

b)

biofilm formation

c)

denaturation

d)

tricking the cell into thinking its a friend then BOOM you have a deadly disease in ur body

27.

Why are C. albicans biofilms so hard to eradicate?

a)

extracellular matrix

b)

persister cell

c)

upregulation of drug efflux pumps

d)

increased cell density and stress response

e)

decreased metabolic rate