WorksheetsRESPI PHYSIO PART 2
Total questions: 47
Worksheet time: 24mins
determines the quantity of oxygen mass reaching the alveoli per minute (g/min)
Ventilation
Perfussion
Pulmonary shunt
Dead space
expresses the flow of blood in the lungs (l/min).
Ventilation
Perfussion
Pulmonary shunt
Dead space
An area with no ventilation (and thus a V/Q of zero)
Ventilation
Perfussion
Pulmonary shunt
Dead space
An area with no perfusion (and thus a V/Q of infinity)
Ventilation
Perfussion
Pulmonary shunt
Dead space
increased pulmonary ventilation.
Hyperventilation
Hyperpnoea
Tachypnoea
Dyspnoea
Apnoea
increase in the rate and depth of breathing (rapid deep breathing) irrespective of subjective sensation of the patient.
Hyperventilation
Hyperpnoea
Tachypnoea
Dyspnoea
Apnoea
rapid shallow breathing
Hyperventilation
Hyperpnoea
Tachypnoea
Dyspnoea
Apnoea
distressed or difficult breathing.
Hyperventilation
Hyperpnoea
Tachypnoea
Dyspnoea
Apnoea
temporary cessation of breathing
Hyperventilation
Hyperpnoea
Tachypnoea
Dyspnoea
Apnoea
alternative phases of apnoea and hyperventilation.
Periodic breathing
Hypoxia
Cyanosis
Hypercapnia
Asphyxia
deficiency of oxygen at tissue level.
Periodic breathing
Hypoxia
Cyanosis
Hypercapnia
Asphyxia
bluish discoloration of skin and mucous membranes.
Periodic breathing
Hypoxia
Cyanosis
Hypercapnia
Asphyxia
decreased level of CO2 in body fluids.
Periodic breathing
Hypocapnia
Cyanosis
Hypercapnia
Asphyxia
increased level of CO2 in body fluids.
Periodic breathing
Hypocapnia
Cyanosis
Hypercapnia
Asphyxia
suffocation, cessation of breathing. Acute hypercapnoea and hypoxia develop together.
Asphyxia
Hyper & hypocapnia
Cyanosis
Hypoxia
Periodic breathing
Volume of air inspired or expired with each normal breath
Tidal volume
inspiratory reserve volume
expiratory reserve volume
residual volume
Extra volume of air that can be inspired over and above normal tidal volume when the person inspires with full force.
Tidal volume
inspiratory reserve volume
expiratory reserve volume
residual volume
Maximum extra volume of air that can be expired by forceful expiration after the end of a normal tidal expiration.
Tidal volume
inspiratory reserve volume
expiratory reserve volume
residual volume
Volume of air remaining in the lungs after the most forceful expiration.
Tidal volume
inspiratory reserve volume
expiratory reserve volume
residual volume
Vital capacity + Residual
Maximum volume to which the lungs can be expanded with the greatest possible inspiratory effort.
Total lung capacity (TLC)
Inspiratory capacity (IC)
Functional residual capacity (FRC)
Forced vital capacity (FVC)
Tidal volume+ Inspiratory reserve
The amount of air that can be inspired, beginning at the end of normal expiratory level.
Total lung capacity (TLC)
Inspiratory capacity (IC)
Functional residual capacity (FRC)
Forced vital capacity (FVC)
Expiratory reserve volume + Residual
The amount of air that remains in the lungs at the end of normal expiration.
Total lung capacity (TLC)
Inspiratory capacity (IC)
Functional residual capacity (FRC)
Forced vital capacity (FVC)
Inspiratory reserve volume+ Tidal volume +Expiratory reserve volume
The maximum amount of air that can be expired during forceful expiration from the lungs after a maximum inspiration.
Total lung capacity (TLC)
Inspiratory capacity (IC)
Functional residual capacity (FRC)
Forced vital capacity (FVC)
This limit the duration of inspiration and increases respiratory rate
apneustic centre
DRG
Nucleus of tractus solitarius
pneumotaxic centre
VRG
this is mainly activated by increased systemic arterial CO2 (pCO2) to produce respiratory response
carotid bodies by increase CO2
carotid bodies increase [H+]
chemosensitive area of the medulla by increase CO2
chemosensitive area of the medulla by increase [H+]
receptors in the by decrease CO2
Kussmaul breathing can be seen in which condition?
ketoacidosis
Hypocapnia
Heart failure
Alkalosis
Hypoxia
Lung function test was done on a patient with bronchial asthma. This is the most likely result to be expected
FEV1/FEV = 40%
FEV1/FEV = 80%
FEV1/FEV = 60%
FEV1/FEV = 100%
Caused by stay at high altitude for a long time
Pulmonary Oedema
Tachypnoea
Seizures
Heartfailure
Predisposing factor for bronchial asthma
Cold weather
Cyanosis abolished
Warm weather
Breathing pure oxygen
This effect stimulates central chemoreceptor by crossing blood brain barrier
Hypercapnia
Hyperpnoea
Tachypnoea
Dyspnoea
Apnoea
A man who is diving under the sea needs to hold his breath. Breath holding is controlled by this.
Voluntary ventilation
Involuntary ventilation
Quiet respiration
internal respiration
This is associated with respiratory center depression
Narcotic overdose
oxygen poisoning
Decompression sickness
Acclimatization
This releases dopamine that initiate afferent neurons impulses through glassopharyngeal and vagus nerve.
Glomus type 1
Supportive type 2
Clara cell
Basal cell
Serum IgE usually raised in this patient affected with this disease
Bronchial asthma
Bronchiectasis
Emphysema
Chronic bronchitis
When pneumotaxic centre is damage in experimental animal . It causes slow and deep breathing
apneustic center
medulla oblongata
Pre-Botzinger complex
Dorsal respiratory group of neurons
Ventral respiratory group of neurons
This is the volume of air present in the conducting zone of bronchial tree.
Anatomical dead space
Physiological dead space
Alveolar dead space
Tidal volume
Residual volume
The arterial PO2 is normal in this condition.
Ventilation perfusion imbalance
Circulatory failure
Depression of respiratory centre
Airway obstruction
Gas exchange failure
This mainly contributes to the paraesthesia associated with hyperventilation
Low carbon dioxide
Increased blood pH level
Cerebral vasoconstriction
Decreased ionized blood calcium
Blood vessel constriction
This is the manifestation of chronic oxygen poisoning on exposure to high barometric pressure.
Pulmonary congestion
Reduction of neuronal excitability
Disturbances of vision
Irritability
Muscle twitching
This is the functional residual capacity of an asthmatic patient whose vital capacity, inspiratory capacity and residual volume measured as 3.5 L, 3 L and 2 L respectively.
6.5 L
5 L
2.5 L
5.5 L
4.5 L
This is approximately the O2 carrying capacity of blood when the haemoglobin in the blood is 10 grams per deciliter.
10 milliliters per deciliter
15 milliliters per deciliter
20 milliliters per deciliter
18 milliliters per deciliter
13 milliliters per deciliter
This is the effect of hypercapnia.
Paraesthesias
Peripheral vasodilatation
Respiratory alkalosis
Dizziness
Light-headedness
This type of flow occurs at higher velocities through an airway like trachea.
Transitional
Laminar
Streamline
Turbulent
Resistance
The stimulation of this results in slow and deep inspiration due to the lesion of the pneumotaxic respiratory center in experimental animals.
Pre-Botzinger complex
Dorsal respiratory group of neurons
Apneustic centre
Cerebral cortex
Ventral respiratory group of neurons
This is true regarding physiological dead space
Determined by the geometry of the branching airway
Not important in the perinatal period
Smaller than the anatomic dead space
Increased in lung disease
Measured using the arterial PO2
During voluntary respiration, this sends signals directly to the respiratory spinal motor neurons bypassing the respiratory centres.
Pre-Botzinger complex
Dorsal respiratory group of neurons
Apneustic centre
Cerebral cortex
Ventral respiratory group of neurons
Hypoxia and hypercapnia occur together in this condition.
Chronic obstructive pulmonary disease
Mountain sickness
Poisoning of cellular oxidative enzymes
Carbon monoxide poisoning
Anaemia
