Worksheets[LRH] CC Primer Part 2
Total questions: 75
Worksheet time: 43mins
main storage of lipids
triglycerides
cholesterol
phospholipid
precursor of steroid horomone
triglycerides
cholesterol
phospholipid
most abundant lipid in the body
triglycerides
cholesterol
phospholipid
main transporter of exogenous triglycerides
chylomicron
very low density lipoprotein
low density lipoprotein
high density lipoprotein
largest but the least dense
chylomicron
very low density lipoprotein
low density lipoprotein
high density lipoprotein
main transporter of endogenous triglycerides
chylomicron
very low density lipoprotein
low density lipoprotein
high density lipoprotein
main transporter of cholesterol to the peripheral tissue
chylomicron
very low density lipoprotein
low density lipoprotein
high density lipoprotein
bad cholesterol
chylomicron
very low density lipoprotein
low density lipoprotein
high density lipoprotein
reverse cholesterol pathway
chylomicron
very low density lipoprotein
low density lipoprotein
high density lipoprotein
smallest but the most dense
chylomicron
very low density lipoprotein
low density lipoprotein
high density lipoprotein
carries endogenous TAG and cholesterol
intermediate density lipoprotein
lipoprotein A
LIPOPROTEIN X
B-VLDL
Sinking prebeta lipoprotein
intermediate density lipoprotein
lipoprotein A
LIPOPROTEIN X
B-VLDL
it interferes with the clot lysis because it competes with plasmin
intermediate density lipoprotein
lipoprotein A
LIPOPROTEIN X
B-VLDL
obstructive jaundice
intermediate density lipoprotein
lipoprotein A
LIPOPROTEIN X
B-VLDL
floating beta lipoprotein; found in type 3 hyperlipoproteinemia
intermediate density lipoprotein
lipoprotein A
LIPOPROTEIN X
B-VLDL
APO A
HDL
Chylomicrons
LDL, VLDL
Chylomicrons and VLDL
Choose the incorrect match
APO B48: Chylomicrons
APO B100: LDL AND VLDL
APO C1 AND APO C2: HDL
APO E: IDL, CHYLOMICRONS
Characterized by markedly reduced or almost efficient HDL
Tangier
Bassen Kronzweig
andersons disease
sitosterolemia
defective apo-B synthesis
Tangier
Bassen Kronzweig
andersons disease
sitosterolemia
defective apo-B48 synthesis
Tangier
Bassen Kronzweig
andersons disease
sitosterolemia
Familial LPL Deficiency
type I
Type IIA
Type IIB
Type
choose the correct match
Type IIA: Familial hypercholesterolemia
Type IIB: Familial Combined hyperlipidemia
Type III: Familial dysbetalipoproteinemia
Type IV: Familial hypertriglyceridemia
Type V: Familial hyperlipoproteinemia
Familial hyperlipoproteinemia
Increase CM, Increase TAG
Increase TAG and CHOL, Increase CM and VLDL
Increase CHOL, Increase LDL
Increase TAG, Increase VLDL
Familial hypercholesterolemia
Increase CM, Increase TAG
Increase TAG and CHOL, Increase CM and VLDL
Increase CHOL, and LDL
Increase TAG, Increase VLDL
Familial LPL Deficiency
Increase CM, Increase TAG
Increase TAG and CHOL, Increase CM and VLDL
Increase CHOL, and LDL
Increase TAG, Increase VLDL
Familial Combined hyperlipidemia
Increase CM, Increase TAG
Increase TAG and CHOL, Increase CM and LDL
Increase CHOL, and LDL
Increase TAG, Increase VLDL
Lipid profile includes
HDL
VLDL
LDL
CHOLESTEROL
TAG
Reference method for lipoproteins
(a)
the most anodal
chylomicrons
ldl
vldl
hdl
high cholesterol
> 200 mg/dl
> 200 mmol/l
>240 mg/dl
>240 mmol/l
high TAG
> 500 mg/dl
> 500 mmol/l
>240 mg/dl
>240 mmol/l
high LDL
> 500 mg/dl
> 500 mmol/l
>190 mg/dl
>190 mmol/l
HIGH RISK FOR CHD
40 MG/DL
28 MG/DL
140 MG/DL
120 MG/DL
alpha 1 globulin
a1 antitrypsin, a1 fetoprotein, GC globulin, a1 antichymotrypsin
haptoglobin, alpha 2 macroglobulin, ceruloplasmiin
transferrin, hemopexin, b2-microglobulin
immunoglobulins
alpha 2 globulin
a1 antitrypsin, a1 fetoprotein, GC globulin, a1 antichymotrypsin
haptoglobin, alpha 2 macroglobulin, ceruloplasmiin
transferrin, hemopexin, b2-microglobulin
immunoglobulins
beta globulin
a1 antitrypsin, a1 fetoprotein, GC globulin, a1 antichymotrypsin
haptoglobin, alpha 2 macroglobulin, ceruloplasmiin
transferrin, hemopexin, b2-microglobulin
immunoglobulins
choose the false statement
albumin: detect malnutrition and transports thyroxine
albumin: most abundant plasma protein
alpha 1 antitrypsin: major inhibitor of protease activity
alpha 1 fetoprotein: greatest affinity for progesterone
choose the false statement
albumin: detect malnutrition and transports thyroxine
albumin: most abundant plasma protein
alpha 1 antitrypsin: vitamin d carrier
alpha 1 fetoprotein: tumor marker for liver cancer
choose the false statement
haptoglobin: binds free hemoglobin
alpha 2 microglobulin: increased in nephrosis
ceruloplasmin: transports iron
hemopexin: bind to heme and helps diagnose early hemolysis
choose the false statement
haptoglobin: binds free hemoglobin
b2 microglobulin: increased in nephrosis
transferrin: transports iron
decrease ceruloplasmin: wilson's disease and menkes kinky hair syndrome
cirrhosis
beta gamma bridging
spiked increase of gamma
low albumin, high alpha 2 and increase beta
low or flat curve in gamma region
polyclonal gammopathy
beta gamma bridging
spiked increase of gamma
low albumin, high alpha 2 and increase beta
low or flat curve in gamma region
nephrotic syndrome
beta gamma bridging
spiked increase of gamma
low albumin, high alpha 2 and increase beta
low or flat curve in gamma region
inflammation
beta gamma bridging
spiked increase of gamma
low albumin, high alpha 2 and increase beta
alpha 1, alpha 2 and beta increase
earliest cardiac marker
(a)
first enzyme to increase during heart attack
(a)
gold standard for the diagnosis of acute coronary syndrom
(a)
glycoprotein used to help predict short term risk for premature delivery
fetal fibronectin
plasma fibronectin
alpha fetoprotein
early assessment of changes to GFR
(a)
help differentiate alzheimers from other forms of dementia.
(a)
reference method for protein measurement
biuret method
kjeldahl
bromcresol green albumin
not polar
Unconjugated bilirubin
conjugated bilirubin
attached to a protein
Unconjugated bilirubin
conjugated bilirubin
delta bilirubin
RBC Destruction yields what product
heme
water
iron
protein
select the correct order
Heme - B2 - Liver - B1 - Intestines - Urobilinogen - Feces and Urine
Globin - B1 - Liver - B2 - Intestines - Urobilinogen - Feces and Urine
Heme - B1 - Liver - B2 - Intestines - Urobilinogen - Feces and Urine
Heme - B1 - Kidney - B2 - Intestines - Urobilinogen - Feces and Urine
serum bilirubin: increased
urine bilirubin: negative
pre hepatic
hepatic
post hepatic
it is a bilirubin transport disorder
gilbert's syndrome
crigler-najjar syndrome
DUBIN JOHNSON SYNDROME
LUCEY-DRISCOLL SYNDROME
ROTOR SYNDROME
disorder where B1 cannot be conjugated to B2 in the liver cell
gilbert's syndrome
crigler-najjar syndrome
DUBIN JOHNSON SYNDROME
LUCEY-DRISCOLL SYNDROME
ROTOR SYNDROME
it is a bilirubin transport disorder
gilbert's syndrome
crigler-najjar syndrome
DUBIN JOHNSON SYNDROME
LUCEY-DRISCOLL SYNDROME
ROTOR SYNDROME
B2 cannot enter the intestine due to no UDPGT
gilbert's syndrome
crigler-najjar syndrome
dubin johnson syndrome
lucey driscoll syndrome
ROTOR SYNDROME
both b2 and b1 is elevated
gilbert's syndrome
crigler-najjar syndrome
dubin johnson syndrome
lucey driscoll syndrome
ROTOR SYNDROME
there is a circulating inhibitor of bilirubin conjugation in the blood
gilbert's syndrome
crigler-najjar syndrome
dubin johnson syndrome
lucey driscoll syndrome
rotor syndrome
more than 80% damage in the liver
hepatitis
cirrhosis
select the false statement
evelyn malloy uses alkaline pH
The dissociating agent of Jendrassik-Grof is Caffeine sodium benzoate which is also preferred
the diazo product of evelyn mallo is red or reddish purple and read at 560 nm
the diazo product of Jendrassik-Grof is blue which is read at 600nm
select the false statement
inulin clearance is most commonly used
creatinine clearance is directly related to muscle mass
earliest GFR test is the urea clearance test
cystatin C denotes damage of the proximal convoluted tubule
major end product of protein catabolism
(a)
major end product of muscle metabolism
(a)
marked elevation of plasma urea and other NPNs accompanied by acidemia and hyperkalemia and associated with renal failure with presence of burr cells
uremia
pre renal azotemia
renal azotemia
post renal azotemia
result of reduced renal blood flow includes shock, hemorrgae, heart failure and dehydratyion
uremia
pre renal azotemia
renal azotemia
post renal azotemia
very high plasma urea concentration accompanied by renal failure
uremia
pre renal azotemia
renal azotemia
post renal azotemia
obstruction anywhere in the urinary tract like renal calculi and tumors of the bladder
uremia
pre renal azotemia
renal azotemia
post renal azotemia
major end product of purine metabolism
(a)
select false statement
highest protein in nephrotic syndrome
red cells and red cell casts in urine for glomerulonephritis
cholesterol crystals and wacy casts are found in the urine of patients suffering from renal failure
abnormal alteration in the excretion of amino acids is seen in tubular defects
product of amino acids deamination
(a)
most commonly seen in children with viral infection and takes too much aspirin causing too high ammonia levels
hepatitis
cirrhosis
hepatic and renal failure
reye syndrome
