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WorksheetsNecrosis and Its Types Quiz
Total questions: 20
Worksheet time: 10mins
Necrosis is characterized by:
No inflammation
Apoptosis features
Loss of membrane integrity
Cell shrinkage
Coagulative necrosis is typically seen in:
Brain infarct
TB
Ischemic injury to kidney
Lipid-rich tissues
Liquefactive necrosis is classical in:
Liver infarction
Brain infarction
Spleen infarction
Heart infarction
Caseous necrosis is seen in:
Ischemia
Fungal infection
TB
Pancreatitis
Fat necrosis in pancreatitis is due to:
Protein denaturation
Lipase action
ROS
Calcium depletion
Fibrinoid necrosis occurs in:
Brain infarct
TB
Vasculitis
Pneumonia
Dry gangrene is:
Infection plus ischemia
Pure ischemia
Only infection
Immunologic
Wet gangrene includes:
Only ischemia
Bacterial infection only
Ischemia plus infection
Viral infection
Gas gangrene is caused by:
Staph aureus
E. coli
Clostridium perfringens
TB
Caseous appearance is:
Soft and liquid
Firm and dry
Cheese-like
Gelatinous
First biochemical change in reversible injury:
Nuclear pyknosis
ATP depletion
Membrane rupture
Karyolysis
Most important consequence of ATP depletion:
Cell shrinkage
Na/K pump failure
DNA damage
Mitochondrial fission
Mitochondrial permeability transition leads to:
ATP increase
More protein synthesis
ATP failure
Less ROS
Increased cytosolic Ca2+ causes:
Inhibits enzymes
Activates destructive enzymes
Prevents ROS
Stabilizes membranes
Free radicals primarily damage:
Ribosomes
Mitochondria only
All cellular components
Lysosomes only
Lipid peroxidation is caused by:
Calcium
ATP depletion
ROS
Proteases
Membrane damage results in:
Reduced permeability
Cell swelling and enzyme leakage
Increased ATP
Cell shrinkage
Early reversible injury feature:
Nuclear fragmentation
Swelling of ER
Membrane rupture
Karyorrhexis
Lysosomal rupture causes:
Cell protection
Autodigestion
ATP generation
Cell repair
Cytochrome c leakage leads to:
Necrosis only
Apoptosis
Mitosis
Inflammation
