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WorksheetsNeoplasia
Total questions: 20
Worksheet time: 10mins
Apoptosis is best defined as:
Programmed cell death mediated by intrinsic enzymes
Passive cell death due to ischemia
Cell death due to inflammatory injury
Cell death associated with necrosis
Neoplasia is defined as:
Abnormal mass persisting after removal of stimulus
Reversible increase in cell number
Growth coordinated with normal tissue
Inflammatory proliferation
Cancer cells most commonly evade apoptosis through which pathway?
Lysosomal pathway
Extrinsic pathway only
Intrinsic mitochondrial pathway
Autophagy pathway
The intrinsic pathway of apoptosis is triggered by all EXCEPT:
DNA damage
Hypoxia
Misfolded proteins
Fas–FasL interaction
Which of the following is a pro-apoptotic protein?
BCL-2
BCL-XL
MCL-1
BAX
Anti-apoptotic members of the BCL-2 family include:
BAX and BAK
BAD and BID
BCL-2 and BCL-XL
BIM and PUMA
Which proteins act as sensors in the intrinsic apoptotic pathway?
BAX, BAK
BAD, BID, BIM, PUMA
Caspase-9
Apaf-1
Release of which molecule from mitochondria activates the caspase cascade?
ATP
Cytochrome C
SMAC
BCL-2
Apaf-1 combines with cytochrome C to form:
Death receptor complex
Apoptosome
Caspase-8 complex
IAP complex
Loss of function mutation of which gene is most important in evasion of apoptosis?
RB
p53
MYC
RAS
Overexpression of MDM-2 leads to:
Increased p53 synthesis
Decreased degradation of p53
Increased degradation of p53
Activation of apoptosis
Follicular lymphoma is associated with which translocation?
t(9;22)
t(15;17)
t(14;18)
t(8;14)
In follicular lymphoma, tumor growth occurs mainly due to:
Increased mitosis
Increased angiogenesis
Evasion of apoptosis
Increased necrosis
Cellular senescence is characterized by:
Apoptosis
Permanent cell cycle arrest
Necrosis
Autophagy
Senescence is mediated by increased levels of:
RAS and MYC
p53 and p16 (INK4A)
BCL-2 and MCL-1
Caspase-3
p53 and p16 maintain RB in which state to cause senescence?
Hyperphosphorylated
Mutated
Hypophosphorylated
Inactive
Cells that escape senescence but are not immortal undergo:
Differentiation
Apoptosis immediately
Mitotic crisis
Autophagy
Telomeres are best described as:
RNA segments in nucleus
Protein structures in cytoplasm
DNA segments at chromosome ends
Mitochondrial DNA
Dysfunctional p53 during telomere shortening leads to:
Apoptosis
Senescence
Non-homologous end joining
Autophagy
Cancer stem cells are characterized by:
Limited replicative capacity
Absence of telomerase
Capacity for self-renewal
Increased senescence
