SEPTIC SHOCK Quiz 1

What are the key pathophysiological mechanisms involved in septic shock?

• The key pathophysiological mechanisms involved in septic shock include systemic inflammation, endothelial dysfunction, and microcirculatory abnormalities.

• Decreased cardiac output, increased systemic vascular resistance, and decreased pulmonary vascular resistance

• High blood pressure, low heart rate, and normal body temperature

• Respiratory alkalosis, metabolic acidosis, and hyperkalemia

How does the immune response contribute to the pathophysiology of septic shock?

• Excessive release of inflammatory mediators

• Decreased release of inflammatory mediators

• No effect on the release of inflammatory mediators

• Release of anti-inflammatory mediators

Explain the role of cytokines in the pathophysiology of septic shock.

• Cytokines inhibit the inflammatory response and reduce vascular permeability.

• Cytokines have no role in the pathophysiology of septic shock.

• Cytokines trigger an inflammatory response and cause widespread vasodilation and increased vascular permeability.

• Cytokines only affect a small area of the body and do not cause widespread effects.

Describe the process of vasodilation and hypotension in septic shock.

• Vasodilation and hypotension in septic shock are caused by the release of inflammatory mediators, leading to the relaxation of blood vessels and a decrease in systemic vascular resistance.

• Vasodilation and hypotension in septic shock are caused by dehydration, leading to the constriction of blood vessels and an increase in systemic vascular resistance.

• Vasodilation and hypotension in septic shock are caused by excessive blood clotting, leading to increased systemic vascular resistance.

• Vasodilation and hypotension in septic shock are caused by a decrease in inflammatory mediators, leading to the constriction of blood vessels and an increase in systemic vascular resistance.

What is the significance of endothelial dysfunction in the pathophysiology of septic shock?

• Endothelial dysfunction leads to decreased vascular permeability

• Endothelial dysfunction has no impact on septic shock

• Endothelial dysfunction improves vasoregulation

• Endothelial dysfunction contributes to increased vascular permeability, microvascular thrombosis, and impaired vasoregulation, leading to organ dysfunction and hypotension.

What are the early clinical manifestations of septic shock?

• Cough, sore throat, runny nose, and headache

• Nausea, vomiting, diarrhea, and abdominal pain

• Dizziness, blurred vision, confusion, and slurred speech

• Fever, chills, rapid heart rate, rapid breathing, and low blood pressure

How does septic shock affect organ function and perfusion?

• Increases organ function and perfusion due to inflammation and vasoconstriction

• Causes organ function and perfusion to remain the same

• Has no effect on organ function and perfusion

• Decreases organ function and perfusion due to inflammation and vasodilation

Explain the relationship between septic shock and altered mental status.

• Decrease in blood flow and oxygen delivery to the brain

• No relationship between septic shock and altered mental status

• Increase in blood flow and oxygen delivery to the brain

• Septic shock causes dehydration and electrolyte imbalance

What are the common cardiovascular manifestations of septic shock?

• Normal blood pressure, irregular heartbeat, and increased cardiac output

• Hypotension, bradycardia, and increased cardiac output

• Hypertension, bradycardia, and increased cardiac output

• Hypotension, tachycardia, and decreased cardiac output

Describe the respiratory manifestations of septic shock.

• Rapid breathing, shortness of breath, and low oxygen levels in the blood

• Slow breathing

• High oxygen levels in the blood

• No respiratory symptoms