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Egan Ch. 15

Total questions: 53

Worksheet time: 2hrs 46mins

Name
Class
Date
1.

What is the effect when the deflation reflex is stimulated?

a)

A strong inspiratory effort

b)

Expiration is initiated

c)

Cuts off all inspiratory signals

d)

Stimulates the termination of expiration

2.

Which components are located in the medulla oblongata? (Check all that apply)

a)

Apneustic center

b)

Dorsal respiratory group neurons (DRGs)

c)

Pneumotaxic center

d)

Ventral respiratory group neurons (VRGs)

e)

Peripheral chemoreceptors

3.

What centers are located in the pons of the brainstem?

1. Apneustic center

2. Dorsal respiratory neurons

3. Pneumotaxic center

a)

1

b)

2, 3

c)

1 , 2

d)

1, 3

e)

all the above

4.

In the face of chronically elevated levels of CO2, what happens to the response mediated by the central chemoreceptors?

a)

It is accentuated or increased.

b)

It is muted or decreased.

c)

There is no change in the response.

d)

There is no way to predict the body’s response.

5.

Which of the following is indirectly responsible for minute-to-minute control of breathing?

a)

CO2 levels

b)

HCO3- levels

c)

Lactate levels

d)

Oxygen (O2) levels

6.

The pneumotaxic center controls which of the following?

a)

Response to changes in blood pH and PCO2

b)

Rhythm of the full breathing cycle

c)

When inspiration switches off (the inspiratory time)

d)

When inspiration switches on (the expiratory time)

7.

When given high concentrations of O2, a patient with chronic hypercapnia may develop a more serious respiratory acidosis. Which of the following might be contributing to the patient’s increased PCO2?

1. Worsening ventilation-perfusion (VV/QQ) balance

2. Desensitization of the carotid bodies

3. Removal of the hypoxic stimulus

a)

1, 2

b)

1

c)

1, 3

d)

all the above

8.

In what manner would O2 therapy induce worsening mismatch and thus a further elevation in CO2 in a chronically hypercapnic patient?

a)

By improving blood flow to poorly ventilated alveoli

b)

By decreasing blood flow to poorly ventilated alveoli

c)

Causing bronchoconstriction, which worsens gas flow to low V/Q areas

d)

Causing bronchodilation, which improves gas flow to poorly ventilated alveoli

9.

What receptors are associated with causing coughing, sneezing, and tachypnea when stimulated?

a)

Irritant receptors

b)

J receptors

c)

Muscle spindles

d)

Peripheral proprioceptors

10.

Why does splashing cold water on the skin stimulate ventilation?

a)

It decreases medullary inspiratory activity causing hyperpnea.

b)

Hering-Breuer inflation reflex.

c)

It increases medullary inspiratory activity causing hyperpnea.

d)

J receptor.

11.

Which inhibitory neuron(s) switch off the inspiratory ramp signal?

a)

Peripheral chemoreceptors

b)

Apneustic center

c)

Central chemoreceptors

d)

Pneumotaxic center

12.

Sensory input to the dorsal respiratory neurons from the lungs, airways, and peripheral chemoreceptors is provided via which nerves?

1. Glossopharyngeal

2. Phrenic

3. Vagus

4. Trigeminal

a)

1, 2, 3

b)

2, 3

c)

1, 3

d)

all the above

13.

A healthy 33-year-old woman relocates to an area approximately 8000 ft above sea level. On her first day, she begins to hyperventilate, but in 24 hr she shows signs of recovery. What is the probable cause of her condition?

a)

Hypoxemia-mediated hyperventilation

b)

Hypercapnia

c)

Hyperoxia-mediated hyperventilation

d)

Increased H+

14.

Where are the peripheral chemoreceptors located?

1. In the arch of the aorta

2. In the bifurcations of carotid arteries

3. On the ventrolateral surfaces of the medulla

a)

1, 2

b)

1, 3

c)

2, 3

d)

all the above

15.

While observing a patient’s breathing, you note that the depth and rate first increase, then decrease, followed by a period of apnea. Which of the following terms would you use in charting this observation?

a)

Apneustic breathing

b)

Biot’s breathing

c)

Cheyne-Stokes breathing

d)

Paradoxical breathing

e)

Mr. Aisner snoring while sitting in a chair in the RT dept.

16.

Which of the following will occur during even strenuous exercise in a normal healthy individual?

a)

Blood gases remain stable.

b)

The arterial PCO2 rises.

c)

The arterial pH falls.

d)

The arterial PO2 falls.

17.

Which of the following can cause laryngospasm and bradycardia through a vagovagal reflex?

1. Bronchoscopy

2. Endotracheal intubation

3. Tracheal suctioning

a)

1, 2

b)

2, 3

c)

1, 3

d)

all the above

18.

Which respond more rapidly to high levels of CO2?

a)

Aortic chemoreceptors

b)

Carotid chemoreceptors

c)

Central chemoreceptors

d)

Ventral respiratory centers

19.

Which breathing pattern can result from a failure to switch off the brainstem inspiratory neurons?

a)

Apnea

b)

Apneustic breathing

c)

Biot's breathing

d)

Cheyne-Stokes breathing

e)

Mr. Aisner back in the RT Dept

20.

Biot’s respiration is most frequently observed in patients with which of the following?

a)

Congestive heart failure

b)

Increased intracranial pressure

c)

Metabolic acidosis

d)

Peripheral nerve disorders

21.

Which of the following centers responds more strongly to high levels of CO2?

a)

Aortic chemoreceptors

b)

Carotid chemoreceptors

c)

Central chemoreceptors

d)

Ventral respiratory centers

22.

Adjustment of respiratory muscle contractions to accommodate varying loads is regulated by which of the following?

a)

Medullary respiratory centers

b)

Muscle spindle fiber feedback

c)

Pontine apneustic center

d)

Pontine pneumotaxic center

23.

A patient in the emergency room is displaying prolong inspiratory gasps interrupted by occasional expirations, what serious injury should be suspected on this patient?

1. Pneumotaxic center has been severed.

2. Vagus nerve has been severed.

3. Glossopharyngeal nerve has been severed.

a)

1, 3

b)

2, 3

c)

1, 2

d)

all the above

24.

Failure to switch off the brainstem inspiratory neurons can result in which of the following?

a)

Apnea

b)

Apneustic breathing

c)

Biot’s breathing

d)

Cheyne-Stokes breathing

25.

A patient is presenting with coughing, sneezing, and reflex bronchoconstriction. Which receptors are causing the client’s presentation?

a)

J receptors

b)

Central chemoreceptors

c)

Hering-Breuer reflex

d)

Irritant receptors

26.

You observe a patient’s breathing pattern as very irregular, with periods of breathing interspersed with long periods of apnea. Which of the following terms would you use in charting this observation?

a)

Apneustic breathing

b)

Biot’s respiration

c)

Cheyne-Stokes breathing

d)

Kussmaul’s breathing

27.

What does apneustic breathing indicate?

a)

Damage to the cerebrum

b)

Damage to the pons

c)

Spinal cord transaction

d)

Vagal nerve damage

28.

The medulla oblongata contains which of the following areas?

1. Apneustic center

2. Dorsal respiratory group neurons (DRGs)

3. Pneumotaxic center

4. Ventral respiratory group neurons (VRGs)

a)

1, 2, 4

b)

2, 4

c)

2, 3, 4

d)

all the above

29.

What stimulates the Hering-Breuer inflation reflex?

a)

The DRG when it is time to end inspiratory efforts.

b)

The stretch of receptors at high lung volumes.

c)

The VRG when it is time for inspiration.

d)

Very low lung volumes stimulate inspiration.

30.

What has the primary responsibility for sensing and responding to changes in blood levels of CO2?

a)

Apneustic centers

b)

Central chemoreceptors

c)

Peripheral chemoreceptors

d)

Pneumotaxic center

31.

What receptors cause a rapid shallow breathing pattern when stimulated by pulmonary disease?

a)

Irritant receptors

b)

J receptors

c)

Muscle spindles

d)

Peripheral proprioceptors

32.

To which anatomic structures do the ventral respiratory centers send motor signals during inspiration?

1. Diaphragm

2. Larynx

3. Pharynx

4. Sternocleidomastoids

a)

1, 2, 4

b)

2, 3, 4

c)

1, 2, 3

d)

all the above

33.

How do central chemoreceptors respond to chronically elevated levels of carbon dioxide (CO2)?

a)

No change in the response

b)

Increased response

c)

No way to predict the response

d)

Muted or decreased response

34.

Both anemia and carbon monoxide (CO) poisoning can cause severe hypoxia, yet neither condition results in a major stimulation of breathing. Why is this so?

a)

The peripheral chemoreceptors do not respond to low O2 content.

b)

Anemia and CO poisoning depresses the peripheral chemoreceptors.

c)

Anemia and CO poisoning depresses the central chemoreceptors.

d)

Anemia and CO cause stagnant hypoxia, not hypoxemia.

35.

What reflex is associated with the sensory stimulation of the pulmonary stretch receptors that stimulate a deeper breath upon inspiration?

a)

Carotid

b)

Head’s paradoxical

c)

Hering-Breuer

d)

J receptor

36.

Why does it take approximately 24 hr for a full ventilatory response to develop to acute hypoxemia?

a)

Initial cerebrospinal fluid alkalemia blunts the hypoxic ventilatory stimulus.

b)

Peripheral chemoreceptors are slow to respond to decreased blood O2 levels.

c)

Renal compensation for respiratory alkalosis increases chemoreceptor sensitivity.

d)

A full ventilatory response is not possible until after the muscles become fatigued.

37.

What is the response of a patient with chronic hypercapnia to a sudden acute rise in carbon dioxide?

a)

In almost all of these patients, there will be no response.

b)

The patient’s drive to breathe will be increased.

c)

This will further depress his or her respiratory centers.

d)

This will induce apnea and sudden death.

38.

Coexisting arterial hypoxemia, acidemia, and high PaCO2 (i.e., asphyxia) will have what effect on the peripheral chemoreceptors?

a)

There is a decreased drive to breathe.

b)

There is a maximally increased drive to breathe.

c)

There is insufficient information to make a determination.

d)

There is virtually no response.

39.

Which of the following can cause Cheyne-Stokes breathing?

1. Brain injuries

2. Congestive heart failure

3. Metabolic acidosis

a)

2

b)

1, 2

c)

2, 3

d)

all the above

40.

Which of the following causes hypoxic stimulation of the carotid bodies?

1. Large decrease in arterial PO2

2. Large decrease in O2 content

3. CO poisoning

a)

1

b)

1, 2

c)

1, 3

d)

all the above

41.

Stimulation of the irritant receptors in the lung can result in which of the following?

1. Bronchoconstriction

2. Coughing

3. Narrowing of the glottis

a)

1, 2

b)

1, 3

c)

2, 3

d)

all the above

42.

What group of nerve cells senses and responds to changes in the chemical composition of its fluid environment?

a)

Chemoreceptors

b)

Gamma-efferent system

c)

Muscle spindle fibers

d)

Proprioceptors

43.

How might oxygen therapy worsen ventilation/perfusion (V/Q) mismatch and increase CO2 in a chronically hypercapnic patient?

a)

Causing bronchodilation, which improves gas flow to poorly ventilated alveoli

b)

By improving blood flow to poorly ventilated alveoli

c)

By decreasing blood flow to poorly ventilated alveoli

d)

Causing bronchoconstriction, which worsens gas flow to low V/Q areas

44.

Stimulation to increase ventilation does not occur until the PaO2 falls below what level?

a)

90 mm Hg

b)

80 mm Hg

c)

70 mm Hg

d)

60 mm Hg

45.

Which of the following control the inhibitory neurons that switch off the inspiratory ramp signal?

1. Apneustic center

2. Pneumotaxic center

3. Pulmonary stretch receptors

a)

1, 3

b)

1, 2

c)

2, 3

d)

all the above

46.

Where are the inspiratory and expiratory centers in the brain found?

a)

Broca’s area

b)

Neurons in the cerebellum

c)

Pons

d)

No such centers exist

47.

While observing a patient's breathing, the RCP notes that the depth and rate increases, then decreases, followed by a period of apnea. Which term does the RCP document in the client’s medical record?

a)

Paradoxical breathing

b)

Apneustic breathing

c)

Biot's breathing

d)

Cheyne-Stokes breathing

48.

What happens in chronic hypercapnia?

1. The central chemoreceptive response to CO2 is decreased.

2. The cerebrospinal fluid pH is restored to normal.

3. Responsiveness to increased CO2 is decreased.

a)

1, 2

b)

1, 3

c)

2, 3

d)

all the above

49.

In patients with closed-head injuries, what may happen in the presence of hypercapnia?

a)

High CO2 increases the risk of psychotic events.

b)

High CO2 levels cause cerebral vasodilation and improved oxygenation.

c)

Severe cerebral vasoconstriction results in anoxia and stroke.

d)

Vasodilation causes increased intracranial pressure and possibly stops blood flow.

50.

Which receptors are primarily responsible for the initial increase in ventilation that occurs at the beginning of exercise?

a)

Carotid chemoreceptors

b)

Irritant receptors

c)

Proprioceptors

d)

J receptors

51.

In the face of hyperoxia, what is the response of the peripheral chemoreceptors to hypercapnia?

a)

There is insufficient information.

b)

There is virtually no response.

c)

There is an increased drive to breathe.

d)

There is a decreased drive to breathe.

52.

How should O2 therapy be administered to chronically hypercapnic patients?

a)

Give as much O2 as possible (60% to 100%).

b)

Withhold O2 until the patient is intubated.

c)

Give as much O2 as required to maintain adequate oxygenation.

d)

Avoid giving any supplemental O2.

53.

What receptors are known to cause an increase in ventilation when the patient’s limbs are moved or cold water is splashed on the patient’s face?

a)

Irritant receptors

b)

J receptors

c)

Muscle spindles

d)

Peripheral proprioceptors