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Worksheets

DR. CORSO LEARNING OBJECTIVES

Total questions: 42

Worksheet time: 2hrs 4mins

Name
Class
Date
1.

How are triglycerides digested?

a)

Triglycerides are reacted with the enzyme ATPase (which comes from the intestines) to create free fatty acids.

b)

Bile salts emulsify TGs in the small intestine.

c)

Triglycerides are reacted with the enzyme lipase (which comes from the pancreas) to create free fatty acids.

d)

Bile salts emulsify TGs in the pancreas.

2.

How does Orlistat work?

a)

it is used to decrease dietary triglyceride absorption.

b)

it is used to increase dietary triglyceride absorption.

3.

What is Orlistat used for?

a)

Orlistat is a lipase agonist

b)

Orlistat is a lipase inhibitor

4.

What do bile salts/acids look like and how do they work? Select all that apply

a)

Steroid type molecule composed of Taurocholic acid (bile acid)

b)

Bile acts as a detergent inducing micelles to form thus dissolving the fat in an aqueous environment

c)

Biles main job is to emulsify fats b/c fats are not H2O soluble

d)

Contain: Cholic acid, taurocholic acid, and glycoholate acid

e)

Bile is made in the liver, stored in the gallbladder and released into the small intestine

5.

What does a triglyceride look like, and which bonds are broken during digestion? Select all that apply

a)

Glycerol esterified to 3 fatty acids

b)

Amide bonds are broken during digestion

c)

Ester bonds are broken during digestion

d)

Glycerol esterified to 2 fatty acids

6.

List out the steps of triglyceride digestion (The H2O soluble lipases acts primarily on the emulsified fats

Converts the triacylglycerols to diacylglycerols, monoacylglycerols, free fatty acids and glycerol.

The majority of the products are 2-monoacylglycerols and free fatty acids)

Step 1: Bile salts emulsify dietary fats in the small intestine, forming mixed micelles

Step 2: Intestinal lipases degrade triacylglycerides

Step 3: Fatty Acids and ether breakdown products are taken up by the intestinal mucosa and converted to triacylglycerides

Step 4: Triacylglycerols are incorporated with cholesterol and apolipoproteins into chylomicrons

Step 5: Chylomicrons move through the lymphatic system and bloodstream to tissues

Step 6: Lipoproteins lipase, activated by apoC-2 in the capillary, releases fatty acids and glycerol

Step 7: Fatty acids enter cells

Step 8: Fatty acids are oxidized as fuel or resterfied for storage

a)

1, 2, 3, 4, 5, 6, 7, 8

b)

8, 2, 3, 4, 5, 1, 7, 6

c)

1, 2, 3, 7, 5, 6, 4, 8

d)

2, 1, 3, 4, 5, 6, 7, 8

7.

How are fats absorbed by the intestine?

a)

FFA + 4-Monoacylglycerol

Fats are broken down by lipase into free FAs. FAs + 4-monoacylglycerol

Lipase is from the pancreas

Bile salts emulsify dietary fats in the intestine, forming mixed micelles (The bile salts comes and forms FA + 4-MG micelles)

b)

FFA + 2-Monoacylglycerol

Fats are broken down by lipase into free FAs. FAs + 2-monoacylglycerol

Lipase is from the pancreas

Bile salts emulsify dietary fats in the intestine, forming mixed micelles (The bile salts comes and forms FA + 2-MG micelles)

8.

Which of the following statements are TRUE about a triglyceride, a diglyceride and cholesterol? Select all that apply

a)

Triglycerides: Glycerol esterified to 3 fatty acids

b)

Diglycerides: 2 Fatty Acids esterified to a glycerol molecule with a polar head group

c)

Cholesterol: Steroid molecule with a polar head group and a alkyl side chain

d)

Triglycerides: Glycerol esterified to 1 fatty acid

9.

How do fats move from the intestine to tissues that absorb fat?

a)

Chylomicrons

b)

Triglycerides

c)

Cholesterol

d)

Insulin

10.

Which of the following pack TGs, cholesterols and apolipoproteins and then move through the lymphatic system and bloodstream into tissues?

a)

Chylomicrons

b)

Insulin

11.

What is LPL and how is it used to absorb fats into tissues? Select all that apply

a)

LPL gene encodes lipoprotein lipase, is synthesized in muscle, heart, and adipose tissue, then secreted & attached to endothelium of the adjacent blood capillaries.

b)

LPL is an enzyme that recognizes chylomicron TG (muscle + fat cell)

c)

This enzyme hydrolyzes the TGs carried in chylomicrons & VLDL to FAs, which can be taken up by cells.

d)

None of them are correct

12.

How does insulin affect lipid metabolism?

a)

Insulin increases triglycerides and increases cholesterol

b)

Insulin decreases triglycerides and decreases cholesterol

13.

What is the relationship between the citric acid cycle and fat synthesis during the fed state?

a)

ATP is decreased during the fed state and the CAC increases or which decreases fat synthesis

b)

ATP is increased during the fed state and the CAC stops or decreases which increases fat synthesis

14.

Where does fatty acid synthesis take place in the cell?  How is this different from elongation and desaturation? Select all that apply

a)

FA Synthesis: Liver cytoplasm

b)

FA Synthesis: pancreas

c)

Elongation & desaturation: Smooth ER of the liver

d)

Elongation & desaturation: occurs mostly in the kidneys

15.

What is the difference between fatty acid synthesis and triglyceride synthesis? Select all that apply

a)

FA (has C10; 12; 14) is synthesized first.

b)

FA (has C16; 18; 20) is synthesized first.

c)

TG synthesis composed of Glycerol + 3 FA (needs to have FAs before can synthesize TG)

d)

TG synthesis composed of Glycerol + 2 FA (needs to have FAs before can synthesize TG)

16.

How does insulin affect cholesterol synthesis?

a)

Insulin causes cholesterol synthesis to increase

b)

Insulin causes cholesterol synthesis to decrease

c)

by activating phosphatase which then activates acetyl CoA carboxylase

d)

by NOT activating phosphatase which then activates acetyl CoA carboxylase

17.

What is the rate controlling step of cholesterol synthesis?

a)

Angiotensin-converting enzyme inhibitor

b)

HMG-CoA reductase

18.

What class of drugs inhibit the rate controlling step of cholesterol synthesis?

a)

Statins

b)

Beta Blockers

c)

ACE Inhibitors

d)

Fibrates

19.

Carry TGs from the intestine to the liver, skeletal muscles and adipose tissues

a)

Chylomicrons

b)

VLDL

20.

Produced in the liver. It carries newly synthesized TGs from the liver to adipose tissue

a)

VLDL

b)

Chilomycrons

21.

Product of VLDL after the removal of TGs

a)

HDL

b)

LDL

22.

Collects cholesterol from the bodies tissues and excess cholesterol in the blood back to the liver for degradation and removal from the body

a)

LDL

b)

HDL

23.

Responsible for reverse cholesterol from the tissues and transport to the liver.

a)

LDL

b)

HDL

24.

Carry cholesterol from the liver to body cells

a)

LDL

b)

HDL

25.

Can buildup in the artery walls and cause them to narrow

a)

LDL

b)

HDL

26.

High _________ levels are associated with atherosclerosis

a)

HDL

b)

VLDL

27.

Which of the following are TRUE about "good cholesterol"? Select all that apply

a)

Does not cause damage

b)

HDL

c)

Facilitates the storage of LDL and promotes excretions

d)

Helps reduce cholesterol from the blood and removes it from the body

e)

Has the highest protein content

28.

Which of the following are TRUE about "bad cholesterol"? Select all that apply

a)

Has the highest cholesterol

b)

Stores cholesterol in the bloodstream

which is bad for us

c)

LDL

d)

Has the lowest cholesterol

29.

What is familial hypercholesterolemia?

a)

LDL receptor defect

b)

HDL receptor defect

30.

What is the effect of statins on other isoprenoids? Select all that apply

a)

Causes isoprenoids to decrease

b)

Causes isoprenoids to increase

c)

decrease in CoQ10

d)

increase in CoQ10

31.

Explain the hormonal control for the release & transport of lipids from the adipose tissue to other tissue such as muscle?

a)

In fasting state: Decreased insulin & increased epinephrine (breakdown of Fat)

b)

In fed state: Decreased insulin & increased epinephrine (breakdown of Fat)

32.

Explain the hormonal control for the release & transport of lipids from the adipose tissue to other tissue such as muscle? Select the best correct order.

Step 1: Hormone (Glucagon), Receptor, and Adenylyl cyclase (AC) bind together and use ATP to release cAMP

Step 2: cAMP activate protein kinase

Step 3: Activate Triacylglycerol lipase to release glycerol and FAs

Step 4: FAs are packed by serum albumin flowing through the bloodstream to Myocyte (Muscle)

Step 5: Beta oxidation occurs, CAC cycle

Step 6: Breaks down FA for energy (ATP) and release CO2

a)

1, 2, 3, 4, 5, 6

b)

2, 1, 4, 3, 5, 6

33.

What is beta-oxidation?

a)

The burning of fatty acids to provide energy to the body in the form of ATP

b)

The burning of chylomicrons to provide energy to the body in the form of insulin

34.

Breaks down a long chain acyl-CoA molecule to acetyl-CoA molecule

a)

beta-oxidation

b)

fed state

35.

How are unsaturated fats catabolized? Select all that apply

Step 1: The body makes sure the double bonds are in the correct position.

Step 2: Enzyme: Enoyl-CoA isomerase will move the double bond to the correct position.

Step 3: 𝛽-oxidation removes 2 Carbons at a time

Does this until Acetyl-CoA is remaining).

a)

3, 1, 2

b)

1, 2, 3

c)

4 extra enzymes

d)

2 extra enzymes

36.

What are Ketone Bodies?

a)

4 carbon body that is water soluble and acidic

b)

2 carbon body that is water soluble and acidic

37.

What is Ketoacidosis? Select all that apply

a)

Increased ketone body production causes a drop in blood pH

b)

More common in Atherosclerosis because body can’t produce enough insulin

c)

More common in T1DM because body can’t produce enough insulin

d)

decreased ketone body production causes an increase in blood pH

38.

It is a molecule 4 rings of a steroid molecule

a)

Triglyceride

b)

Cholesterol

39.

When MEVALONATE is reduced it makes the enzyme ENZYME HMG-COA REDUCTASE .

a)

True

b)

False

40.

Binds to LDL receptor

a)

ApoB-100

b)

ApoC-II

41.

Activates lipoprotein lipase

a)

ApoC-II

b)

ApoB-100

42.

WITHOUT CARNITINE WE CANNOT MOVE FAT TO THE MITOCHONDRIA

a)

True

b)

False