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Mycology II

Total questions: 32

Worksheet time: 32mins

Name
Class
Date
1.

Yeast colonies are generally measured in:

a)

mm

b)

cm

c)

nm

d)

um

e)

m

2.

When we use the term glabrous to describe a fungal colony, which property of a colony Is it referring to?

a)

surface pigmentation

b)

topography

c)

radius

d)

reverse pigmentation

e)

texture

3.

How many classes of antifungal drugs are currently approved?

a)

6

b)

4

c)

3

d)

2

e)

5

4.

Which of the following is NOT an approved class of antifungal drug?

a)

glycolipid inhibitors

b)

triazoles

c)

allylamines

d)

echinocandins

e)

none of the above

5.

Which of the following classes of drugs targets the cell wall?

a)

5'flycytosine

b)

trizazoles

c)

echinocandins

d)

polyenes

e)

allyamines

6.

The antifungal drug nystatin binds to which component of the fungus?

a)

ribosomal RNA machinery

b)

chitin

c)

mannoproteins

d)

beta-glucans

e)

ergosterol

7.

Which of the following antifungal drugs should NOT be used as a monotherapy and why?

a)

amphotericin B (AMB), as it requires IV administration

b)

nystatin, as it is very poorly bioavailable

c)

caspofungin, as resistance to it develops very quickly

d)

fluconazole, as its a first-generation azole

e)

5'flucytosine, as resistance to it develops very quickly

8.

The ability of the yeast Cryptocococcus neoformans to grow in the presence of caspofungin is best described as:

a)

intrinsic antifungal resistance

b)

acquired antifungal resistance

c)

long term antifungal resistance

d)

transient antifungal resistance

e)

antifungal resistance due to low bioavailability

9.

A mechanism observed to underlie resistance in fungi to several classes of antifungal drug is:

a)

inhibition of nucleic acid synthesis

b)

modifications to efflux pumps

c)

upregulation of cellular stress response

d)

thickening of cell wall

e)

none of the above

10.

Mutations in the genes that underlie ergosterol biosynthesis in the fungal cell underlie resistance to which class of antifungal drugs?

a)

polyenes

b)

allylamines

c)

echinocandins

d)

flucytosine

e)

none of the above

11.

T or F. Fungal colonial morphology tells us whether the fungus is dimorphic

(a)  

12.

What needs to be described when looking at colonial morphology?

a)

colony diameter

b)

surface topography

c)

surface texture

d)

surface pigment

e)

reverse pigment

13.

What is an example of a dimorphic fungi?

a)

Sporothirx spp.

b)

Sporomyocin spp.

c)

Spornate spp.

d)

Sporotin spp.

14.

T or F. Sporothrix schenckii lives in soil or plant matter & spores come into contact with skin through small cuts and scrapes. Also known as gardeners disease

(a)  

15.

T or F. The central feature of antifungal agents is the requirement for eukaryotic machinery to be differently blocked or destroyed in fungus BUT do limited or no damage to host cellular functions

(a)  

16.

What are the targets of an antifungal?

a)

plasma membrane (azoles, polyenes and allylamines)

b)

cell wall (echinocandins)

c)

nucleic acid synthesis (flucytosine)

d)

nucleus (ribocytine)

17.

Select all that are correct about polyenes

a)

bind to ergosterol to form a pore complex

b)

disrupts plasma membrane leading to leakage of cytoplasmic contents

c)

accumulation of ROS results in DNA, protein, mitochondrial and membrane damage

d)

example drug is Amphotericin B

e)

toxicity is not an issue (can bind to cholesterol and injure host cells)

18.

Select all that are correct about azoles

a)

inhibit ergosterol biosynthesis via cytochrome P450 enzyme 14-a demethylase

b)

catalyses conversion of lanosterol to ergosterol

c)

affects integrity of plasma membrane

d)

uncommon class of antifungals

e)

example drugs are imidazoles and triazoles

19.

Select all that are correct about allylamines

a)

inhibit early stages of ergosterol biosynthesis

b)

targets the enzyme squalene epoxidase

c)

inhibits integrity of plama membrane

d)

highly effective against dermatophytes

e)

example drugs are terbinafine and butenafine

20.

Select all that are correct about echinocandins

a)

target synthesis of B-1,3 glucans by blocking enzyme glucan synthase

b)

causes decrease of glucose monomers linking B-1,3 and B-16 glucans

c)

weakens cell wall and leads to fungal cell lysis

d)

the first class of antifungals to be developed

e)

example drugs are caspofungin, micafungin, anidulafungin

21.

Select all that are correct about flucytosine

a)

synthetic analogue of cytosine converted to 5-fluorouracil

b)

5-fluorouracil encourages DNA and RNA synthesis

c)

rapid development of resistance occurs during monotherapy

d)

example drug is flucytosine

22.

T or F. Superficial infections are caused by dermatophytes and yeasts

(a)  

23.

T or F. Invasive infections are a risk to healthy individuals

(a)  

24.

Fluconazole and nystatin can be used to treat superficial infections with yeasts like .....

a)

Candida spp.

b)

Aspergillus

c)

Cryptococcus

d)

E. coli

25.

Terbinafine is successful in treating ....

a)

dermatophytes

b)

yeasts

c)

arachnids

d)

mosquito bites

26.

Amphotericin B can be used to treat _______ but toxic to kidneys and requires IV

a)

Aspergillus

b)

Candida spp.

c)

Cryptococcus

d)

Streptococcus spp.

27.

T or F. Antifungal tolerance is the ability to grow at drug concentrations above MICs that stop growth or kill most species

(a)  

28.

Select all that are correct

a)

antifungal resistance can be classified as either intrinsic or acquired

b)

intrinsic is not associated with drug exposure

c)

acquired is associated with antifungal use

d)

acquired isolates are not able to mutate

e)

example of intrinsic is Cryptococcus neoformans

29.

What is the correct sequence of mechanism for azoles

1. upregulation of stress response pathways

2. elevation of efflux pump activity that decreases drug concentrations in fungal cell

3. target site modification

a)

2, 3, 1

b)

1, 2, 3

c)

3, 1, 2

d)

2, 1, 3

30.

What is the correct sequence of mechanism for echinocandins

1. point mutation in Fks1, Fks2, Fks3

2. genes encoding glucan synthase

3. upregulation of mechanisms protecting cell from stress

a)

1, 2, 3

b)

3, 2, 1

c)

2, 3, 1

d)

3, 1, 2

31.

What is the correct sequence of mechanism for polyenes

1. thicker cell walls

2. upregulation of mechanisms protecting cells from stress

3. defects in ERG3 gene lead to accumulation of other sterols in fungal membrane

a)

3, 1, 2

b)

1, 2, 3

c)

3, 2, 1

d)

2, 3, 1

32.

What is the correct sequence of mechanism for flucytosine

1. mutations in cytosine deaminase

2. mutations in USX1 gene in Cryptococcus spp.

3. mutations in uracil phosphoribosyl

a)

3, 1, 2

b)

2, 3, 1

c)

1, 2, 3

d)

3, 2, 1