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principles of drug action p.2

Total questions: 46

Worksheet time: 27mins

Name
Class
Date
1.

Drug A and Drug B have the same efficacy at the M3 muscarinic receptor; however, Drug A is more potent than Drug B.

If Drug A is a full agonist, what can you deduce about Drug

B?

a)

a) Drug B is a competitive antagonist of Drug A

b)

b) Drug B is also a full agonist

c)

c) Drug B is a partial agonist

d)

d) Drug B is a physiological antagonist of Drug A

2.

Which of the following statements regarding agonists is incorrect?

a)

a) An agonist with high affinity will always trigger a strong response

b)

b) A partial agonist will have an intrinsic efficacy between 0 and 1

c)

c) An agonist acting on the central nervous system can produce an overall

inhibitory response to the central nervous system

d)

d) An agonist will bind to a receptor and activates its function

3.

Pharmacodynamics describes:

a)

the effects of the drug on the body

b)

the effects of the body on the drug

(adme)

c)

the study of genetic influences on responses to drugs, associated with adverse drug reactions (ADRs) within a gene population

d)

the study of drug effects at the population level

e)

the cost effectiveness of using individual or classes of drug

4.

Pharmacokinetics describes:

a)

the effects of the drug on the body

b)

the effects of the body on the drug

(adme)

c)

the study of genetic influences on responses to drugs, associated with adverse drug reactions (ADRs) within a gene population

d)

the study of drug effects at the population level

e)

the cost effectiveness of using individual or classes of drug

5.

Pharmacogenomics describes:

a)

the effects of the drug on the body

b)

the effects of the body on the drug

(adme)

c)

the study of genetic influences on responses to drugs, associated with adverse drug reactions (ADRs) within a gene population

d)

the study of drug effects at the population level

e)

the cost effectiveness of using individual or classes of drug

6.

example of pharmacogenomics:

a)

defects in the gene for the P450 enzyme CYP2D6 cause poor metabolism of the anti-depressant fluoxetine (Prozac) and has led to deaths

b)

withdrawal of the anti-arthritic drug rofecoxib (Vioxx) due to increased risk of heart attack/stroke

c)

use of herceptin for breast cancer

7.

Pharmacoepidemiology describes:

a)

the effects of the drug on the body

b)

the effects of the body on the drug

(adme)

c)

the study of genetic influences on responses to drugs, associated with adverse drug reactions (ADRs) within a gene population

d)

the study of drug effects at the population level

e)

the cost effectiveness of using individual or classes of drug

8.

example of pharmacoepidemiology

a)

defects in the gene for the P450 enzyme CYP2D6 cause poor metabolism of the anti-depressant fluoxetine (Prozac) and has led to deaths

b)

withdrawal of the anti-arthritic drug rofecoxib (Vioxx) due to increased risk of heart attack/stroke

c)

use of herceptin for breast cancer

9.

example of pharmacoeconomics

a)

defects in the gene for the P450 enzyme CYP2D6 cause poor metabolism of the anti-depressant fluoxetine (Prozac) and has led to deaths

b)

withdrawal of the anti-arthritic drug rofecoxib (Vioxx) due to increased risk of heart attack/stroke

c)

use of herceptin for breast cancer

10.

the definition of a drug

a)

An exogenous biologically active compound taken with the intent to produce a change in the body. This effect may be positive but can be negative.

b)

A chemical preparation which usually contains one or more drugs administered

with the purpose of producing a positive therapeutic effect. 

11.

the definition of medicine

a)

An exogenous biologically active compound taken with the intent to produce a change in the body. This effect may be positive but can be negative.

b)

A chemical preparation which usually contains one or more drugs administered

with the purpose of producing a positive therapeutic effect. 

12.

which of these is an example of a drug?

a)

Cannabidiol (CBD)

b)

Epidyolex

13.

which of these is an example of a medicine

a)

Cannabidiol (CBD)

b)

Epidyolex

14.

who proposed the magic bullet theory whereby a drug would have selectively toxicity e.g. therapy without toxicity.

a)

Paul Ehrlich

b)

John Newport Langley

c)

Brian McCain

d)

John Ronald

15.

described drug action (nicotinic acting at the neuromuscular junction) in terms of interaction with a “receptive substance”.

a)

Paul Ehrlich

b)

John Newport Langley

c)

Brian McCain

d)

John Ronald

16.

who tested receptor action by effects of nicotine and curare on skeletal muscle contraction

a)

Paul Ehrlich

b)

John Newport Langley

c)

Brian McCain

d)

John Ronald

17.

salbutomal works on which extra-cellular receptor to dilate the bronchioles (airways)

a)

b1-adrenoceptor

b)

b2-adrenoceptor

18.

to be able to set up the right test you have to ensure that you measure the

a)

access

b)

response

c)

duration

d)

side effects

e)

concentration

19.

in vitro assays describe

a)

•performed or taking place in a test tube, culture dish, or elsewhere outside a living organism.

b)

performed or taking place in a living organism.

20.

in vivo assays describe

a)

•performed or taking place in a test tube, culture dish, or elsewhere outside a living organism.

b)

performed or taking place in a living organism.

21.

examples of in vivo test

a)

Intervention (e.g. drug x)

b)

Genetic modification

(e.g. KO of  Gene Y)

c)

drug X on an isolated rodent heart preparation

d)

human stem cells

e)

drug X in a cellular model of depression

22.

why can isolating one process be challenging given the array of targets involved and give an example

a)

Several different targets to modulate.

All can impact on contractility.

b)

Model systems cannot replicate the same common pathways

c)

smooth muscle contractility.

d)

the effect of salbumatol on the bronchioles

23.

the measure contractility you can monitor intracellular (a)   levels via fluorescence.

24.

if a drug has a low therapeutic index it means

a)

only need a small increase in dose to produce toxic effects

b)

the safer the drug

c)

ratio of toxic to therapeutic dose.

25.

IC50 can also be used be referred as

a)

agonist potency

b)

antagonist potency

c)

efficacy

d)

EC50

26.

properties of a drug

a)

•Drugs must dissolve in H2O

b)

less than 500

27.

Electronegativity is a measure of an atoms ability to ....... electrons towards itself

a)

attract

b)

repel

28.

from the left to the right of the periodic table electronegativity

a)

increases

b)

decreases

29.

going down the periodic table electronegativity

a)

increases

b)

decreases

30.

what is the most electronegative element

a)

F

b)

O

c)

Cl

d)

Na

31.

Hydrogen bonding describes

a)

Interaction between electron-rich atom (Hydrogen Bond Acceptor, HBA)  and electron deficient hydrogen atom (Hydrogen Bond Donor, HBD).

b)

Ionic interaction between groups carrying opposite charges.

c)

an attractive interaction between atoms which results from induced dipoles, and a repulsive interaction, which results from overlap of the electron clouds of two atoms when they get too close

32.

Electrostatic interactions describes

a)

Interaction between electron-rich atom (Hydrogen Bond Acceptor, HBA)  and electron deficient hydrogen atom (Hydrogen Bond Donor, HBD).

b)

Ionic interaction between groups carrying opposite charges.

c)

an attractive interaction between atoms which results from induced dipoles, and a repulsive interaction, which results from overlap of the electron clouds of two atoms when they get too close

33.

Van der waals interactions describes

a)

Interaction between electron-rich atom (Hydrogen Bond Acceptor, HBA)  and electron deficient hydrogen atom (Hydrogen Bond Donor, HBD).

b)

Ionic interaction between groups carrying opposite charges.

c)

an attractive interaction between atoms which results from induced dipoles, and a repulsive interaction, which results from overlap of the electron clouds of two atoms when they get too close

34.

Isomer : Different compounds which have the same

a)

molecular formula

b)

empirical formula

c)

no. electrons

d)

enatomers

35.

WHY ARE ENANTIOMERS IMPORTANT?

4 lines
36.

select the reasons why adrenaline was not suitable for the treatment of asthma

a)

it has a short duration of action

b)

it has a long duration of action

c)

no selectivity for b2-receptors

d)

no selectivity for b1-receptors

e)

leading to side effects, particularly cardiovascular effects.

37.

adrenaline was replaced by which antiasthmatic agent.

a)

isoprenaline

b)

salbumatol

c)

noremphoxine

d)

fluticasone

38.

isoetharine was shown to be selective for b2-receptors ( because of of its bulky N-alkyl substituent) but why were the effects short lasting?

a)

a result of the drugs being taken up by tissues and methylated by the metabolic enzyme catechol-O-methyltransferase (COMT), to form an inactive ether.

b)

However it showed no selectivity between the different types of b-receptors

39.

what made salbutamol have an affinity for only the b-receptors?

a)

Increasing the size of the group on nitrogen

b)

forming an inactive ether.

40.

-COOH

a)

basic

b)

acidic

41.

-NH2

a)

acidic

b)

basic

42.

the definition of formation

a)

An exogenous biologically active compound taken with the intent to produce a change in the body. This effect may be positive but can be negative.

b)

A chemical preparation which usually contains one or more drugs administered

with the purpose of producing a positive therapeutic effect. 

c)

the process in which different chemical substances, including the active drug, are combined to produce a final medicine.

43.

LOW P VALUE IS

a)

A

b)

B

c)

C

44.

Dissolve in both

a)

A

b)

B

c)

C

45.

High LogP value

a)

A

b)

B

c)

C

46.

plasma concentration

a)

dosevolume of distribution \frac{dose}{volume\ of\ distribution\ }

b)

dose x kgvolume of distribtuion \frac{dose\ x\ kg}{volume\ of\ distribtuion\ }

c)

concentration of drugwater\frac{concentration\ of\ drug}{water}