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B cell activation stopp @ slide 16

Total questions: 52

Worksheet time: 31mins

Name
Class
Date
1.

Each B cell has Ig receptors of a single antigen specificity.

a)

True

b)

False

2.

Mature B cells circulate in the blood, lymph, and lymphoid organs until they encounter antigen.

a)

True

b)

False

3.

When a B cell is stimulated by an antigen, it will ____

a)

phagocytize the antigen and die

b)

create many clones bearing the same Ag receptor

4.

Selected B cell clones will become ____

a)

plasma cells

b)

memory B cells

5.

Which cell takes antigen to draining lymph nodes and activate antigen-specific naive Tcells?

a)

macrophages

b)

DCs

6.

T cells proliferate and differentiate into ___

a)

plasma cells

b)

effector T cells

7.

stay in the LN and interact with activated B cells

a)

TH1

b)

TFH

c)

cytotoxic T cells

8.

Which antibody is made first?

a)

high affinity IgM

b)

low affinity IgM

9.

Memory B cells are generated to provide long term protection

a)

True

b)

False

10.

The part of antigen that an antibody/BCR binds

a)

epitope

b)

TCR

11.

Usually an epitope is a/an

a)

protein

b)

carbohydrate

c)

lipid

d)

any of these

12.

Pathogens typically have one epitope that can be bound by antibody.

a)

True

b)

False

13.

antigen that contains more than 1 epitope or multiple copies of the same epitope

a)

multivalent antigen

b)

T cell

14.

B cells can bond to which of the following epitopes?

a)

protein

b)

carbohydrates

c)

lipids

15.

T cells can bond to which type of epitope?

a)

protein

b)

carbohydrate

c)

lipid

16.

Epitopes are composed of 2 groups:

a)

Linear epitopes

b)

Discontinuous epitopes

17.

Composed of several successive amino acid residues.

a)

Linear epitopes

b)

Discontinuous epitopes

18.

Formed from 2 or more parts of the protein antigen that are separated in the amino acid sequence but are brought together in the tertiary structure of the folded protein.

a)

Linear epitope

b)

discontinuous epitope

19.

Which one?

a)

linear epitope

b)

discontinuous epitope

20.

Which one?

a)

linear epitope

b)

discontinuous epitope

21.

What is required for B cell activation?

a)

cell-cell contact with MHC

b)

crossllinking of surface Ig

22.

B cell activation

a)

surface IgM on naive B cells binds to epitope

b)

multivalent antigens bind to multiple IgM causing cross linking

c)

cross-linking leads to signals to inside of B cell through Iga and IgB of BCR

d)

signaling leads to gene expression-> B cell activation

e)

additional signals required

1)
2)
3)
4)
5)
23.

After cross linking causes clustering, signals are sent inside of B cell through ___ and ___ of the BCR

a)

Iga

b)

IgB

c)

IgM

d)

IgG

24.

B cell co-receptor is composed of ...

a)

CR2

b)

CD19

c)

CD81

25.

Part of the b cell co-receptor that can bind complement components fixed on pathogen.

a)

CR2

b)

CD19

c)

CD81

26.

acts as the signaling chain in interactions with Iga and IgB of BCR

a)

CR1

b)

CD19

c)

CD81

27.

Brings CD19 to the surface and organizes the coreceptor on the membrane

a)

CR2

b)

CD19

c)

CD81

28.

The B cell co receptor increases signal by 1000 to 10,000 fold and incrases B cells sensitivity to antigen.

a)

True

b)

False

29.

B cell response that requires help from T cells (CD4+ helper T cells)

a)

T-dependent

b)

T-independent

30.

Which T cells help during B cell T-dependent responses?

a)

CD4 + helper T cells

b)

CD8 + cytoxic T cells

31.

B cell respose that makes TI-1 and TI-2 antigens and do NOT require T cell help.

a)

T dependent

b)

T independent

32.

What two subclasses of B cells mediate response to T independent antigen?

a)

B1-B cells

b)

Marginal zone B cells

33.

B cell that produces T independent antigen that produces IgM antibodies that bind to broad spectrum of Ag with low affinity.

a)

B1-B

b)

Marginal zone B cells

34.

Specialized to respond to blood born Ag entering interacting with the immune cells in the spleen.

a)

B1-B cells

b)

Marginal zone B cells

35.

B1-B cells produce....

a)

high affinity IgM

b)

low affinity IgM

36.

Rank the following in order from Best to Worst with number 1 being the best and number 3 being the worst.

a)

TD antigens

b)

Type II TI antigens

c)

Type I TI antigens

1)
2)
3)
37.

Why is type II T independent antigens better than Type I TI antigens?

a)

there is no polyclonal activation in Type II TI

b)

not this

38.

TI-1 antigens are typically ___

a)

bacterial cell wall components

b)

polymeric protein antigens

c)

capsule bacterial polysaccharides

d)

Polymeric flagellin

39.

TI-1 antigens bind to innate immunity PRR on all B cells

a)

True

b)

False

40.

Which one results in polyclonal antibody response?

a)

TI type I

b)

TI type II

41.

TI type II antigens:

a)

bacterial cell wall componenets

b)

polymeric protein antigens

c)

capsular bacterial polysaccharides

d)

polymeric flagellin

42.

Bind to and cross link many IgM BCRs

a)

TI-1

b)

TI-2

43.

Typically have bound complement fragments

a)

TI-1

b)

TI-2

44.

Can crosslink BCR and CR2 of the B cell co receptor

a)

TI-1

b)

TI-2

45.

Interaction with T cells can push TI-2 activated cells to produce isotypes of antibody other than ____

a)

IgG

b)

IgM

46.

Polyclonal antibody response

a)

TI-1

b)

TI-2

47.

Following B cell development, naive B cells migrate to the ____

a)

lymphoid follicles

b)

blood

48.

In the lymphoid follicles,

a)

B cell can interact with antigen and become activated

b)

if not activated, can recirculate through the blood and lymph and back

49.

If the B cell does not come in contact with antigen in the lymphoid follicle....

a)

it will be activated

b)

it will recirculate

50.

Naive B cells migrate to the

a)

lyphoid follicles

b)

medullary cord

51.

What are the 3 pathways of TD activation of B cells?

a)

become an antibody producing plasma cell

b)

become low affinity memory cells

c)

enter germinal center to participate in the germinal center reaction

52.

In the germinal center, B cells can undergo...

a)

clonal expansion of B and TFH cells

b)

somatic hypermutation

c)

Isotype switching