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Quiz 10 (Pathologic calcification + cell aging )

Total questions: 10

Worksheet time: 5mins

Name
Class
Date
1.

Which of the following is true about dystrophic calcification?

a)

It occurs in normal tissues.

b)

It is always associated with hypercalcemia.

c)

It occurs in areas of necrosis or tissue damage.

d)

It is caused by excessive vitamin D intake.

2.

Metastatic calcification is commonly associated with which condition?

a)

Local tissue necrosis

b)

Chronic inflammation

c)

Hyperparathyroidism

d)

Ischemia

3.

What is the key microscopic feature of dystrophic calcification?

a)

Deposition of calcium in intact tissue

b)

Psammoma bodies in necrotic areas

c)

Calcium deposition in the walls of healthy arteries

d)

Amorphous, basophilic calcium deposits on necrotic cells

4.

Which of the following is NOT a cause of metastatic calcification?

a)

Hypercalcemia due to renal failure

b)

Vitamin D intoxication

c)

Elevated parathyroid hormone levels

d)

Chronic infection

5.

In metastatic calcification, where does calcium typically deposit?

a)

Necrotic tissues

b)

Normal blood vessels and lungs

c)

Healing scars

d)

Areas of chronic inflammation

6.

Which cellular mechanism is most closely associated with aging?

a)

Increased telomerase activity

b)

Accumulation of somatic DNA damage

c)

Enhanced mitochondrial biogenesis

d)

Increased synthesis of growth factors

7.

Telomere shortening during cell division is primarily mediated by which enzyme deficiency?

a)

DNA polymerase

b)

Telomerase

c)

Helicase

d)

RNA polymerase

8.

Which of the following proteins is associated with the regulation of cellular senescence?

a)

p53

b)

Myc

c)

Ras

d)

Akt

9.

What is the effect of caloric restriction on aging, according to studies mentioned in Robbins?

a)

Accelerates telomere shortening

b)

Decreases mitochondrial oxidative stress

c)

Reduces expression of DNA repair enzymes

d)

Increases metabolic rate

10.

Lipofuscin accumulation in aging cells is indicative of which process?

a)

Increased protein synthesis

b)

Failure of proteasomal degradation

c)

Free radical-induced lipid peroxidation

d)

Enhanced autophagy