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electrolytes practice quiz

Total questions: 15

Worksheet time: 8mins

Name
Class
Date
1.

Which statement best describes sodium’s role in excitable tissues at rest and during signaling?

a)

Sets action potential peak and drives repolarization

b)

Blocks resting membrane potential and prevents depolarization

c)

Sets resting membrane potential and drives depolarization

d)

Raises resting membrane potential and drives hyperpolarization

2.

Which factor pair primarily regulates how much filtered sodium is reabsorbed by the kidneys?

a)

GFR and dietary potassium

b)

Aldosterone and urine pH

c)

Blood pressure and osmolarity

d)

Plasma glucose and bicarbonate

3.

In the nephron, which segment reabsorbs the largest fraction of filtered sodium under normal conditions?

a)

Proximal tubule

b)

Distal convoluted tubule

c)

Thin descending limb

d)

Collecting duct

4.

According to the aldosterone mechanism shown, what is the primary cellular effect in principal cells that increases sodium reabsorption?

a)

Upregulating ENaC channels and Na⁺/K⁺ pumps

b)

Blocking aquaporins in collecting ducts

c)

Reducing transcription of ion transport proteins

d)

Inhibiting Na⁺/K⁺/2Cl⁻ cotransporters

5.

Based on the RAAS diagram, which stimulus most directly increases aldosterone secretion from the adrenal zona glomerulosa?

a)

Elevated atrial natriuretic peptide

b)

Reduced angiotensin II signaling

c)

Increased blood volume and pressure

d)

Elevated plasma potassium concentration

6.

When mean arterial pressure increases, what renal response most directly promotes pressure natriuresis?

a)

Increased Na+ reabsorption distally

b)

Increased aldosterone secretion

c)

Increased renin and angiotensin II

d)

Increased Na+ and water excretion

7.

A patient with excess plasma volume has atrial stretch. Which change is most consistent with ANP action on the kidney?

a)

Efferent dilation and reduced filtrate

b)

Increased aldosterone and Na+ reabsorption

c)

Increased GFR and Na+ excretion

d)

Decreased GFR and water retention

8.

Which finding best distinguishes hypernatremia from hyponatremia in terms of likely symptoms?

a)

Bradycardia and dry mucous membranes

b)

Pulmonary edema with pink froth

c)

Confusion from cerebral edema

d)

Hypertension and peripheral edema

9.

Which nephron segment is the primary site of action for loop diuretics, and what transporter do they inhibit there?

a)

Distal convoluted tubule; ENaC channel

b)

Thick ascending limb; Na+-K+-2Cl− cotransporter

c)

Proximal tubule; Na+/glucose symporter

d)

Collecting duct; aquaporin-2 water channel

e)

Descending limb; passive Na+ channel

10.

A loop diuretic reduces the medullary osmotic gradient. What is the most direct consequence for water handling in the collecting duct?

a)

Decreased water reabsorption because gradient collapses

b)

Unchanged water handling across all segments

c)

Enhanced ADH-independent insertion of aquaporins

d)

Compensatory increase in proximal water secretion

e)

Increased water reabsorption due to stronger gradient

11.

Which factor most directly promotes potassium movement from extracellular fluid into cells after a meal?

a)

Hyperosmolality drawing water out

b)

Cell lysis releasing cytosolic ions

c)

Acidosis exchanging H⁺ for K⁺

d)

Insulin activation of Na⁺/K⁺-ATPase

12.

In the cortical collecting duct principal cell, which mechanism increases potassium secretion into the tubular lumen?

a)

Reduced distal sodium delivery rate

b)

Inhibition of Na⁺/K⁺-ATPase pump

c)

Aldosterone upregulating ENaC and ROMK

d)

Activation of α-intercalated H⁺ pump

13.

Which nephron segment primarily adjusts potassium excretion in response to dietary intake through aldosterone-sensitive secretion?

a)

Proximal tubule reabsorptive segment

b)

Thick ascending limb transporter region

c)

Distal tubule and collecting duct

d)

Inner medullary loop thin segment

14.

Which renal effect of parathyroid hormone (PTH) most directly increases blood calcium levels?

a)

Upregulation of Ca2+ channels in distal tubule

b)

Inhibition of aquaporins in collecting duct

c)

Activation of NKCC2 in thick ascending limb

d)

Stimulation of urea transport in inner medulla

15.

A patient on a thiazide develops mild hypercalcemia. Which mechanism best explains this effect in the distal convoluted tubule?

a)

Direct inhibition of ATP-driven H+ secretion by intercalated cells

b)

Increased NKCC2 activity creating lumen-negative gradient

c)

Reduced CaSR activation with decreased cAMP formation

d)

Enhanced TRPV5-mediated Ca2+ entry with Na+/Ca2+ exchange