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L4 Patho I Hypersensitivity

Total questions: 51

Worksheet time: 26mins

Name
Class
Date
1.

What is an allergy?

a)

A response directed against beneficial foreign tissues.

b)

A misdirected response against the host’s own cells.

c)

An exaggerated response against an environmental antigen.

d)

An altered immunologic response to an antigen that results in disease or damage to the host.

2.

What is hypersensitivity?

a)

A response directed against beneficial foreign tissues.

b)

An altered immunologic response to an antigen that results in disease or damage to the host.

c)

A misdirected response against the host’s own cells.

d)

An exaggerated response against an environmental antigen.

3.

What is autoimmunity?

a)

A response directed against beneficial foreign tissues.

b)

An altered immunologic response to an antigen that results in disease or damage to the host.

c)

An exaggerated response against an environmental antigen.

d)

A misdirected response against the host’s own cells.

4.

What is alloimmunity?

a)

An altered immunologic response to an antigen that results in disease or damage to the host.

b)

An exaggerated response against an environmental antigen.

c)

A response directed against beneficial foreign tissues.

d)

A misdirected response against the host’s own cells.

5.

Which of the following is an inappropriate immune response?

a)

Allergy

b)

Hypersensitivity

c)

Autoimmunity

d)

All of the above

6.

Which type of hypersensitivity is IgE-mediated and immediate?

a)

Type I

b)

Type II

c)

Type III

d)

Type IV

7.

Which immunoglobulins are involved in tissue-specific hypersensitivity?

a)

IgE

b)

IgG, IgM

c)

IgA

d)

IgD

8.

What type of hypersensitivity involves immune complexes and is immediate?

a)

Type I

b)

Type II

c)

Type III

d)

Type IV

9.

Which type of hypersensitivity involves mast cells and is immediate?

a)

Type I

b)

Type II

c)

Type III

d)

Type IV

10.

What type of hypersensitivity is associated with systemic lupus erythematosus?

a)

Type I

b)

Type II

c)

Type III

d)

Type IV

11.

Which type of hypersensitivity is delayed and involves lymphocytes and macrophages?

a)

Type I

b)

Type II

c)

Type III

d)

Type IV

12.

Which type of hypersensitivity involves neutrophils and is immediate?

a)

Type I

b)

Type II

c)

Type III

d)

Type IV

13.

Which type of hypersensitivity is associated with autoimmune hemolytic anemia?

a)

Type I

b)

Type II

c)

Type III

d)

Type IV

14.

Which type of hypersensitivity does not involve complement activation?

a)

Type I

b)

Type II

c)

Type III

d)

Type IV

15.

What is the primary difference between immediate and delayed hypersensitivity reactions?

a)

Immediate reactions occur within minutes to hours, while delayed reactions take several hours to days.

b)

Immediate reactions involve T cells, while delayed reactions involve B cells.

c)

Immediate reactions require multiple exposures, while delayed reactions occur after a single exposure.

d)

Immediate reactions are always more severe than delayed reactions.

16.

Which type of hypersensitivity reaction is more likely to require multiple exposures over years?

a)

Immediate hypersensitivity

b)

Delayed hypersensitivity

c)

Both immediate and delayed hypersensitivity

d)

Neither immediate nor delayed hypersensitivity

17.

What is required for both immediate and delayed hypersensitivity reactions to occur?

a)

Multiple exposures to the antigen

b)

Primary and secondary immune responses

c)

Only a primary immune response

d)

Only a secondary immune response

18.

What is the most rapid and immediate type of hypersensitivity reaction?

a)

Type I

b)

Type II

c)

Type III

d)

Type IV

19.

Which of the following is a common antigen that can trigger anaphylaxis?

a)

Bee stings

b)

Peanuts

c)

Fish

d)

All of the above

20.

Which clinical manifestation is NOT typically associated with severe cases of anaphylaxis?

a)

Contraction of bronchial smooth muscle

b)

Laryngeal edema

c)

Vascular collapse

d)

Increased blood pressure

21.

What is the primary immunoglobulin involved in Type I hypersensitivity reactions?

a)

IgA

b)

IgG

c)

IgE

d)

IgM

22.

Which cells are primarily responsible for the release of histamine in Type I hypersensitivity reactions?

a)

B cells

b)

T cells

c)

Mast cells

d)

Neutrophils

23.

Which of the following is a common clinical manifestation of a Type I hypersensitivity reaction in the respiratory tract?

a)

Urticaria

b)

Rhinitis

c)

Diarrhea

d)

Abdominal pain

24.

Which cells present antigens to Th2 cells during the sensitization phase of a Type I hypersensitivity reaction?

a)

B cells

b)

Dendritic cells

c)

Mast cells

d)

Neutrophils

25.

What is the role of histamine binding to H1 receptors in Type I hypersensitivity reactions?

a)

Increased gastric acid secretion

b)

Decreased histamine release from mast cells

c)

Contraction of bronchial smooth muscles

d)

Attraction of eosinophils

26.

Why are tissues of the GI tract, respiratory tract, and skin most affected by Type I hypersensitivity reactions?

a)

They contain large numbers of mast cells

b)

They have high blood flow

c)

They are exposed to environmental antigens

d)

They have a high density of nerve endings

27.

Which mechanism of Type II hypersensitivity involves the destruction of cells by antibodies and the complement system?

a)

Phagocytosis

b)

Cell malfunction

c)

Complement

d)

NK cells

28.

Which cells are primarily involved in the tissue damage caused by neutrophil granules in Type II hypersensitivity reactions?

a)

Macrophages

b)

Neutrophils

c)

T cells

d)

NK cells

29.

Graves disease is an example of which mechanism of Type II hypersensitivity?

a)

Complement

b)

Phagocytosis

c)

Neutrophil granules

d)

Cell malfunction

30.

Which cells are primarily involved in the phagocytosis mechanism of Type II hypersensitivity?

a)

Macrophages

b)

Neutrophils

c)

T cells

d)

NK cells

31.

Which mechanism of Type II hypersensitivity involves the release of toxic substances by NK cells?

a)

Complement

b)

Phagocytosis

c)

Neutrophil granules

d)

Antibody-dependent cell-mediated cytotoxicity (ADCC)

32.

Which of the following mechanisms of Type II hypersensitivity does NOT activate the complement system?

a)

Complement

b)

Phagocytosis

c)

Neutrophil granules

d)

Cell malfunction

33.

What is a key characteristic of Type III hypersensitivity reactions?

a)

They involve IgE antibodies.

b)

They are organ-specific.

c)

They involve immune complexes formed in the circulation.

d)

They do not activate the complement system.

34.

Which of the following is an example of a Type III hypersensitivity reaction?

a)

Graves disease

b)

Serum sickness

c)

Allergic rhinitis

d)

Contact dermatitis

35.

What differentiates Type II from Type III hypersensitivity reactions?

a)

Type II reactions involve soluble antigens, while Type III reactions involve cell-bound antigens.

b)

Type II reactions are not organ-specific, while Type III reactions are organ-specific.

c)

Type II reactions involve cell-bound antigens, while Type III reactions involve soluble antigens.

d)

Type II reactions do not activate the complement system, while Type III reactions do.

36.

What is a key difference between Type II and Type III hypersensitivity reactions?

a)

Type II reactions involve soluble antigens, while Type III reactions involve cell-bound antigens.

b)

Type II reactions are organ-specific, while Type III reactions are not organ-specific.

c)

Type II reactions do not activate the complement system, while Type III reactions do.

d)

Type II reactions involve immune complexes formed in the circulation, while Type III reactions do not.

37.

What is the primary cause of tissue damage in Type III hypersensitivity reactions?

a)

Direct cytotoxicity by T cells

b)

Immune complex deposition and subsequent complement activation

c)

IgE-mediated mast cell degranulation

d)

Antibody-dependent cell-mediated cytotoxicity (ADCC)

38.

What is the primary mediator of Type IV hypersensitivity reactions?

a)

IgE

b)

IgG

c)

T lymphocytes

d)

Complement system

39.

Which of the following is an example of a Type IV hypersensitivity reaction?

a)

Acute graft rejection

b)

Asthma

c)

Systemic lupus erythematosus

d)

Hemolytic anemia

40.

What primarily mediates Type IV hypersensitivity reactions?

a)

IgE

b)

IgG

c)

T lymphocytes

d)

Complement system

41.

Which of the following is an example of a Type IV hypersensitivity reaction?

a)

Serum sickness

b)

Contact dermatitis

c)

Graves disease

d)

Allergic rhinitis

42.

Why does the first exposure to an antigen not result in clinical manifestations in Type IV hypersensitivity reactions?

a)

The immune system is not sensitized yet and is producing memory T cells.

b)

The antigen is immediately neutralized by antibodies.

c)

The antigen is too weak to cause a reaction.

d)

The antigen is rapidly cleared by the liver.

43.

What is a key characteristic of Type I hypersensitivity skin reactions?

a)

Lesions are widely distributed.

b)

Lesions are localized to the site of contact.

c)

Lesions are only found on the face.

d)

Lesions are only found on the hands.

44.

Which type of hypersensitivity reaction is associated with contact dermatitis?

a)

Type I

b)

Type II

c)

Type III

d)

Type IV

45.

How can the distribution of lesions help distinguish between Type I and Type IV hypersensitivity reactions?

a)

Type I reactions have localized lesions, while Type IV reactions have widely distributed lesions.

b)

Type I reactions have widely distributed lesions, while Type IV reactions have localized lesions.

c)

Both Type I and Type IV reactions have widely distributed lesions.

d)

Both Type I and Type IV reactions have localized lesions.

46.

How can hemolytic anemia result from hypersensitivity reactions?

a)

Through the destruction of erythrocytes by antibodies against antigens bound to their plasma membranes.

b)

By the overproduction of erythrocytes in response to allergens.

c)

Due to the inhibition of erythrocyte production in the bone marrow.

d)

By the direct attack of erythrocytes by T cells.

47.

Which of the following allergens can induce Type II hypersensitivity reactions?

a)

Bee venom

b)

Penicillin metabolites

c)

Pollen

d)

Dust mites

48.

What clinical manifestations can result from the destruction of cells by antibodies in Type II hypersensitivity reactions?

a)

Asthma and eczema

b)

Anemia and thrombocytopenia

c)

Rashes and hives

d)

Fever and chills

49.

What is a common local skin reaction to vaccines?

a)

Redness at the injection site

b)

Fever

c)

Headache

d)

Nausea

50.

Which of the following is a delayed local skin reaction to vaccines?

a)

Immediate swelling

b)

Delayed rash

c)

Immediate pain

d)

Immediate redness

51.

What type of local skin reaction can occur within hours after vaccination?

a)

Bruising

b)

Itching

c)

Redness and swelling

d)

Blistering