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Tests of Cell-mediated Immunity: Intradermal Tests

Total questions: 65

Worksheet time: 33mins

Name
Class
Date
1.

Which immunoglobulin primarily mediates reactions detected by intradermal allergy tests?

a)

IgG antibodies circulating in plasma

b)

IgM antibodies during primary response

c)

IgE antibodies bound to mast cells

d)

IgA antibodies on mucosal surfaces

2.

What is the typical outcome indicating a positive intradermal reaction at an injection site?

a)

Blanching without any swelling

b)

A vesicle with purulent fluid

c)

A warm ecchymotic bruise

d)

A raised wheal or welt

3.

Which cells release histamine-containing granules during an immediate hypersensitivity skin reaction?

a)

Cytotoxic T lymphocytes

b)

Macrophages and dendritic cells

c)

Basophils and mast cells

d)

Neutrophils and eosinophils

4.

During intradermal testing, which substance serves as the negative control?

a)

Saline in sterile solution

b)

Histamine standard dose

c)

Epinephrine solution

d)

Allergen extract mix

5.

During intradermal testing, which substance serves as the positive control?

a)

Buffered saline diluent

b)

Patient serum sample

c)

Histamine reference dose

d)

Glycerinated allergen

6.

Which site is commonly chosen for placing intradermal test injections in small animals?

a)

Medial thigh region

b)

Ventral abdominal wall

c)

Lateral thorax area

d)

Dorsal cervical neck

7.

When should intradermal test sites typically be evaluated for reactivity after injection?

a)

At 90 and 120 minutes

b)

At 45 and 60 minutes

c)

At 15 and 30 minutes

d)

At 5 and 10 minutes

8.

Which preparation step is recommended for the skin before placing intradermal injections?

a)

Leave hair intact and dry

b)

Apply alcohol and iodine

c)

Shave hair but do not scrub

d)

Scrub with chlorhexidine soap

9.

Allergen selection for intradermal testing is best guided by which factors?

a)

Randomized allergen panels

b)

Geography and patient history

c)

Hospital formulary restrictions only

d)

Owner preference exclusively

10.

Which clinical manifestation is most consistent with type I hypersensitivity on skin testing?

a)

Lichenification with scaling crusts

b)

Petechiae and purpura patches

c)

Cyanosis and cold extremities

d)

Urticaria and erythematous wheals

11.

What measurement is commonly recorded to grade each intradermal reaction relative to controls?

a)

Change in local temperature

b)

Diameter of the wheal formed

c)

Duration until peak itching

d)

Volume of injected solution

12.

A patient shows no response to allergens and no response to saline, but a clear wheal to histamine. How should the test be interpreted?

a)

Test inconclusive due to artifacts

b)

Test positive to multiple allergens

c)

Test valid with negative allergens

d)

Test invalid due to anergic skin

13.

Which immunoglobulin is targeted by ELISA tests for allergen-specific antibodies in veterinary patients such as dogs, cats, and horses?

a)

IgE antibodies specific to allergens

b)

IgG antibodies after vaccination

c)

IgM antibodies during primary response

d)

IgA antibodies in mucosal secretions

14.

The tuberculin skin test primarily detects infection by which group of organisms in animals?

a)

Mycobacterium species causing tuberculosis

b)

Staphylococcus species causing dermatitis

c)

Brucella species causing abortion

d)

Leptospira species causing leptospirosis

15.

In the tuberculin skin test, a positive reaction is best described as which immunologic mechanism?

a)

Neutralizing antibody blockade of toxins

b)

Complement-mediated cytolysis of erythrocytes

c)

Delayed hypersensitivity to tuberculin antigens

d)

Immediate hypersensitivity to circulating IgE

16.

For primates, where is tuberculin commonly injected during the skin test to observe reactivity?

a)

Intradermally in the eyelid area

b)

Subcutaneously in the forearm

c)

Intramuscularly in the thigh

d)

Intradermally on the abdomen

17.

Which statement best contrasts ELISA for allergen-specific IgE with the tuberculin skin test?

a)

ELISA detects bacterial DNA; TST detects secreted exotoxins

b)

ELISA measures cell-mediated swelling; TST measures serum IgG

c)

ELISA requires intradermal injection; TST requires blood draw

d)

ELISA measures circulating antibodies; TST measures delayed skin reactivity

18.

A herd of cattle is screened for Mycobacterium infection. Which approach aligns with standard practice for this purpose?

a)

Measure serum IgE using allergen panels

b)

Collect nasal swabs for rapid antigen tests

c)

Give oral tuberculin and observe fever

d)

Administer intradermal tuberculin at the tail base

19.

A veterinary team observes a firm swelling 72 hours after intradermal tuberculin injection in a macaque eyelid. What is the most defensible conclusion?

a)

The injection was placed too deeply into muscle

b)

An acute IgE-mediated allergy to tuberculin occurred

c)

Cell-mediated sensitization to mycobacterial antigens is present

d)

The animal has protective neutralizing antibody levels

20.

A dog with seasonal pruritus is tested with an ELISA panel. What result would most directly support selecting allergen-specific immunotherapy?

a)

Detection of allergen-specific IgE to environmental proteins

b)

Presence of high titer IgM against vaccine antigens

c)

Positive delayed-type reaction to tuberculin antigen

d)

Low total IgA concentration in mucosal secretions

21.

What primary immune mechanism is identified by intradermal testing when evaluating allergic reactions?

a)

IgA-mediated mucosal immunity

b)

Cell-mediated cytotoxic responses

c)

IgM-mediated complement activation

d)

IgE-mediated hypersensitivity responses

22.

Which material is required to perform intradermal testing accurately?

a)

Live attenuated microorganisms

b)

Autologous patient plasma only

c)

Standardized extracts of antigens

d)

Neutral saline without proteins

23.

When assessing an intradermal test site, which parameters should be recorded to evaluate the lesion adequately?

a)

Injection depth and needle gauge

b)

Skin temperature and hydration

c)

Patient age and past exposures

d)

Erythema presence and lesion size

24.

Basophils and mast cells contribute to the local reaction during intradermal testing primarily through which mediator?

a)

Complement C9 forming membrane pores

b)

Prostaglandin I2 causing vasoconstriction

c)

Interleukin-2 driving clonal expansion

d)

Histamine release causing inflammation

25.

A clinic wants to screen for prior exposure to Mycobacterium using intradermal methods. Which statement best justifies this choice?

a)

Antigen extracts are unnecessary for mycobacterial tests

b)

Intradermal tests bypass any inflammatory pathways

c)

Mycobacteria produce immediate IgM antibodies only

d)

Mycobacterial antigens can be detected intradermally

26.

Which statement best describes the purpose of a Coombs' test?

a)

Measures complement levels during acute infection

b)

Detects autoantibodies targeting patient tissues

c)

Identifies bacterial antigens in patient serum

d)

Quantifies total immunoglobulin concentration

27.

What does a direct Coombs' reaction primarily detect on red blood cells?

a)

Bacterial endotoxin attached to RBC surfaces

b)

Antibodies bound to patient RBC membranes

c)

Free antibodies circulating in patient plasma

d)

Complement proteins attached to platelets

28.

A positive direct Coombs' test most strongly supports which diagnosis?

a)

Iron‑deficiency anemia without hemolysis

b)

Immune‑mediated hemolytic disease

c)

Hereditary spherocytosis only

d)

Acute bacterial sepsis causing fever

29.

What does the indirect Coombs' test detect most specifically?

a)

Complement fragments bound to leukocytes

b)

Antigens on donor lymphocytes in plasma

c)

Unbound antibodies circulating in the serum

d)

Hemoglobin released from lysed erythrocytes

30.

In an indirect Coombs' procedure, patient serum is typically incubated with which material to screen for antibodies?

a)

Latex beads coated with immunoglobulin

b)

Reagent red blood cells from another source

c)

Patient’s own washed red blood cells

d)

Purified bacterial surface antigens

31.

Which finding most likely yields a positive indirect Coombs' result?

a)

Serum antibodies against self tissues present

b)

No antibodies detected in patient plasma

c)

Hemolysis due to mechanical valve trauma only

d)

Only complement activation on platelets present

32.

Which statement best defines an antibody titer?

a)

Greatest dilution at which the sample remains positive

b)

Average antibody concentration measured in milligrams

c)

Volume of serum required to neutralize all antigen

d)

Lowest dilution that first yields any visible agglutination

33.

When are antibody titers particularly useful in practice?

a)

Distinguishing active infection from prior exposure

b)

Detecting acute toxin ingestion in the liver

c)

Screening for congenital enzyme deficiencies

d)

Confirming normal electrolyte balance after therapy

34.

Which interpretation aligns best with a high antibody titer in a symptomatic patient?

a)

Usually means only previous exposure long ago

b)

Often indicates an active ongoing infection

c)

Always guarantees complete protective immunity

d)

Proves current antigen test is unreliable

35.

Which clinical decision is most appropriately guided by measuring antibody titers?

a)

Choosing the correct antibiotic susceptibility

b)

Assessing the need for revaccination boosters

c)

Confirming blood type for transfusion

d)

Estimating hematocrit for anemia staging

36.

Which immunoglobulin class primarily mediates Type I hypersensitivity reactions?

a)

IgE antibodies bound to mast cells

b)

IgG antibodies activating complement

c)

IgM antibodies on B lymphocytes

d)

IgA antibodies in mucosal secretions

37.

A patient develops bronchoconstriction minutes after peanut exposure. Which mediator release best explains this response?

a)

Mast cell mediators causing smooth muscle contraction

b)

Neutrophil enzymes causing tissue necrosis

c)

Cytotoxic T cells causing apoptosis of epithelium

d)

NK cell perforin causing airway cell lysis

38.

Which clinical syndrome is most consistent with a Type I hypersensitivity mechanism?

a)

Contact dermatitis with T cell activation

b)

Hemolytic anemia due to anti-RBC antibodies

c)

Serum sickness with immune complex vasculitis

d)

Allergic rhinitis with increased mucus secretion

39.

Type II hypersensitivity is best characterized by which mechanism?

a)

Delayed T cell–mediated cytokine injury

b)

IgE-mediated degranulation of mast cells

c)

Immune complexes depositing in vessel walls

d)

Antibodies directed against self cell surface antigens

40.

Immune-mediated hemolytic anemia (IMHA) exemplifies Type II hypersensitivity because it involves:

a)

Eosinophil cytotoxicity to parasites

b)

IgE crosslinking on mast cell surfaces

c)

Host antibodies targeting red blood cells

d)

Circulating immune complexes in glomeruli

41.

Which antibody classes most commonly mediate Type II hypersensitivity?

a)

IgG and IgM binding to cell antigens

b)

IgE and IgA in mucosal tissues

c)

IgD and IgA on naïve B cells

d)

IgM and IgE in anaphylaxis

42.

A transfusion reaction primarily represents which hypersensitivity type and mechanism?

a)

Type IV with delayed T cell response

b)

Type II with antibodies against donor cells

c)

Type I with allergen-specific IgE binding

d)

Type III with immune complex deposition

43.

Immune-mediated thrombocytopenia (IMT) occurs when:

a)

Immune complexes inflame vessel walls

b)

Autoantibodies lead to platelet destruction

c)

Cytotoxic T cells lyse thrombocytes

d)

Mast cells constrict airway smooth muscle

44.

Type III hypersensitivity reactions are defined by which process?

a)

Deposition of antigen–antibody complexes in tissues

b)

CD8+ T cell–mediated cytolysis of target cells

c)

IgE-mediated release of histamine from mast cells

d)

Direct antibody binding to cell surface proteins

45.

Glomerulonephritis in Type III hypersensitivity is caused by:

a)

NK cell attack on podocyte membranes

b)

Autoantibodies lysing renal tubular cells

c)

IgE-driven edema of Bowman’s capsule

d)

Immune complex deposition in renal glomeruli

46.

Systemic lupus erythematosus frequently features which immunopathologic mechanism?

a)

CD8+ T cells destroying epidermal cells

b)

Anti-platelet IgG causing thrombocytopenia

c)

IgE-mediated urticaria after allergen exposure

d)

Circulating immune complexes causing vasculitis

47.

Which immediate physiologic change is most typical minutes after mast cell degranulation?

a)

Increased vascular permeability leading to edema

b)

Progressive destruction of erythrocyte membranes

c)

Delayed activation of macrophage cytokines

d)

Complement fixation on basement membranes

48.

Differentiate Type I and Type II hypersensitivity by their primary targets:

a)

Type I targets self cells via IgG; Type II targets tissues

b)

Type I targets T cells; Type II targets parasites

c)

Type I targets allergens via IgE; Type II targets cells

d)

Type I targets immune complexes; Type II targets allergens

49.

Which scenario best illustrates strategic management of anaphylaxis at first recognition?

a)

Administer intramuscular epinephrine immediately

b)

Order complement levels for confirmation

c)

Begin high-volume IV antibiotics promptly

d)

Schedule outpatient skin testing later

50.

A dog presents with hemoglobinuria after receiving mismatched blood. What mechanism most likely explains the hemolysis?

a)

CD4+ T cells recruit macrophages to RBCs

b)

IgM or IgG binds donor RBCs activating complement

c)

IgE crosslinks on basophils releasing histamine

d)

Immune complexes deposit in renal vessels

51.

Which mechanism best defines Type IV hypersensitivity?

a)

T cell-mediated response against self antigens

b)

IgE-mediated mast cell degranulation reactions

c)

Immune complex deposition causing complement activation

d)

Autoantibody binding to cell surface receptors

52.

Which clinical example is most directly linked to Type IV hypersensitivity mechanisms?

a)

Contact dermatitis from nickel exposure

b)

Anaphylaxis after peanut ingestion

c)

Myasthenia gravis with anti-AChR antibodies

d)

Hemolysis from ABO incompatibility

53.

A disease primarily involving cytotoxic T lymphocyte destruction of pancreatic beta cells exemplifies which category?

a)

Type II hypersensitivity antibody disease

b)

Type I hypersensitivity allergic disease

c)

Type IV hypersensitivity autoimmune disease

d)

Type III hypersensitivity immune complex disease

54.

Which pair correctly matches disease with predominant immune pathway?

a)

Type 1 diabetes—immune complex deposition

b)

Contact urticaria—CTL-mediated cytolysis

c)

Rheumatoid arthritis—IgE dependent degranulation

d)

Tuberculosis—delayed-type T cell response

55.

In contact hypersensitivity, what is the proximal effector causing tissue injury?

a)

Eosinophil major basic protein release

b)

Circulating IgM against haptens

c)

Complement membrane attack complex

d)

Activated T lymphocytes and macrophages

56.

Which statement best characterizes lymphoma?

a)

Tumor arising from uncontrolled lymphocyte proliferation

b)

Autoimmune inflammation driven by self-reactive antibodies

c)

Fibrotic replacement of lymphoid organs by collagen

d)

Infection-induced hyperplasia that resolves spontaneously

57.

Why is lymphoma categorized as an immune disorder?

a)

It is caused exclusively by pathogenic bacteria

b)

It involves dysregulated cells of the immune system

c)

It always results from chronic allergic reactions

d)

It only occurs with congenital complement defects

58.

Which observation most supports a role for immune surveillance in preventing cancer establishment?

a)

Complement classical pathway lyses gram-negative bacteria

b)

Cytotoxic T cells recognize and eliminate transformed cells

c)

IgE antibodies neutralize polysaccharide toxins in serum

d)

Neutrophils phagocytose encapsulated bacteria efficiently

59.

A patient with chronic delayed-type skin reactions and autoimmunity is most likely experiencing pathology mediated predominantly by which cells?

a)

Hepatic Kupffer cells producing cytokines

b)

B cells secreting neutralizing IgA

c)

Th1 cells and cytotoxic T lymphocytes

d)

Basophils and mast cells with IgE

60.

Which scenario distinguishes Type IV hypersensitivity from antibody-mediated hypersensitivities?

a)

Immediate wheal-and-flare occurs within minutes

b)

Pathology depends on immune complex deposition

c)

Disease improves rapidly after plasmapheresis

d)

Onset peaks after sensitization with delayed response

61.

Which description best matches Type I hypersensitivity?

a)

Immune complex deposition within target tissues

b)

Immediate reaction driven by IgE and atopy

c)

Delayed T cell–mediated cytotoxic inflammation

d)

Antibody binding to cell surfaces causing lysis

62.

A patient develops hemolysis after a mismatched transfusion. Which hypersensitivity type most directly explains this event?

a)

Type I immediate hypersensitivity with atopy

b)

Type IV T cell–mediated delayed reaction

c)

Type II antibody-mediated cytotoxicity

d)

Type III immune complex deposition

63.

Immune complex deposition leading to tissue inflammation is characteristic of which hypersensitivity type?

a)

Type III with circulating immune complexes

b)

Type I mediated by IgE and mast cells

c)

Type IV mediated by sensitized T cells

d)

Type II with antibodies to cell antigens

64.

Which mechanism primarily underlies Type IV hypersensitivity?

a)

IgE cross-linking causing mast cell degranulation

b)

Deposition of antigen–antibody complexes in tissues

c)

Complement-mediated lysis of targeted erythrocytes

d)

Activation of sensitized T cells causing inflammation

65.

Lymphoma is best defined as which process?

a)

Autoantibody attack on erythrocyte membranes

b)

Immediate hypersensitivity with systemic anaphylaxis

c)

Uncontrolled proliferation of lymphocytes

d)

Immune complex deposition in renal glomeruli