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WorksheetsGIT
Total questions: 80
Worksheet time: 3600secs
A patient with achlorhydria will have decreased release of which hormone?
Secretin
Gastrin
CCK
Motilin
Pancreatic enzyme secretion is most strongly stimulated by:
Gastrin
CCK
Somatostatin
VIP
Bile salts are synthesized in the liver from:
Triglycerides
Cholesterol
Amino acids
Glucose
A blockage of the common bile duct leads to elevated:
Amylase
Lipase
Bilirubin
Trypsin
Secretin primarily increases:
HCl secretion
Bicarbonate secretion
Pepsinogen release
Motility
The primary site of digestive enzyme action for fats is:
Stomach
Duodenum
Jejunum
Ileum
Gastric inhibitory peptide (GIP) is released in response to:
Fat & glucose in the small intestine
Acid in stomach
Distension of stomach
Amino acids in colon
Trypsinogen is activated by:
Enterokinase
Pepsin
HCl
Amylase
Failure of CCK secretion would most impair digestion of:
Carbohydrates
Proteins
Fats
Electrolytes
Secretin release is triggered by:
Fat in stomach
Acid in duodenum
Protein in jejunum
Distension of ileum
A patient with pancreatic insufficiency would have malabsorption of:
Vitamins A, D, E, K
B12 only
Iron only
Water-soluble vitamins
Bile salts facilitate fat absorption by forming:
Micelles
Chylomicrons
Proteosomes
Liposomes
Which hormone increases biliary secretion?
Motilin
Secretin
Ghrelin
Insulin
G cells in the stomach secrete:
CCK
Gastrin
Secretin
Somatostatin
Somatostatin inhibits secretion of:
Gastrin
Insulin
Glucagon
All of the above
The cephalic phase of gastric secretion is mediated by:
CCK
Vagus nerve
Secretin
Gastrin
Pepsinogen is secreted by:
Parietal cells
Chief cells
G cells
D cells
The primary stimulant for HCl secretion is:
Secretin
Gastrin
GIP
VIP
Failure of the sphincter of Oddi to relax would impair:
Pancreatic enzyme delivery
Bile into duodenum
Both A & B
Gastric emptying
A patient with Zollinger–Ellison syndrome has high:
Gastrin
Secretin
CCK
Motilin
Pancreatic secretion with high bicarbonate and low enzymes occurs in response to:
Secretin
CCK
Gastrin
Motilin
Enterokinase is located on the brush border of:
Ileum
Jejunum
Duodenum
Colon
The major end product of starch digestion is:
Glucose
Lactose
Maltose
Fructose
CCK release is MOST sensitive to:
Acid
Fatty acids
Proteins
Monosaccharides
A deficiency in bile salts leads to malabsorption of:
Carbohydrates
Proteins
Fats
Vitamins B & C
HCl secretion is inhibited by:
Gastrin
Histamine
Secretin
ACh
A drug that blocks histamine H2 receptors reduces:
Gastric emptying
Bile secretion
HCl secretion
Pancreatic enzyme secretion
The major pancreatic enzyme for fat digestion is:
Amylase
Lipase
Trypsin
Elastase
Motilin increases:
Pancreatic secretions
Gastric motility during fasting
Bile secretion
Insulin release
Digestion of triglycerides begins in the:
Mouth
Stomach
Duodenum
Jejunum
Which hormone increases insulin release after a meal?
CCK
GIP
Secretin
Gastrin
Bile is stored in the:
Pancreas
Gallbladder
Duodenum
Liver sinusoids
A pancreatic duct obstruction will cause elevated:
ALT
Lipase
Amylase
Bilirubin
Intrinsic factor is necessary for absorption of:
Iron
B12
Folate
Calcium
The migrating motor complex is stimulated by:
Motilin
Gastrin
CCK
Secretin
Gastric phase of secretion contributes what % of total gastric secretion?
10%
30%
50%
70%
Enterogastric reflex decreases:
Gastric emptying
Pancreatic secretion
Bile secretion
Intestinal motility
VIP causes:
Increased HCl
Increased pancreatic secretion
Increased bile salt synthesis
Smooth muscle relaxation
A lack of ileal bile salt absorption leads to:
Constipation
Steatorrhea
High B12 absorption
Hypoglycemia
Gastric acid secretion at basal state is mostly stimulated by:
Histamine
CCK
Secretin
GIP
The hormone that inhibits gallbladder contraction is:
Somatostatin
CCK
Gastrin
Motilin
Pancreatic acinar cells secrete:
Bicarbonate
Enzymes
Hormones
Bile salts
The stimulus for bicarbonate secretion in the pancreas is:
Gastric acid
Fat in stomach
Glucose
Amino acids
Trypsin inhibitor in pancreatic juice prevents:
Acid damage
Premature activation
Bicarbonate loss
CCK release
Gallbladder emptying occurs mainly in response to:
Secretin
CCK
Gastrin
GIP
Which enzyme digests proteins in the small intestine?
Amylase
Lipase
Peptidases
Lactase
The pancreas secretes enzymes in a(n) _____ form:
Active
Proenzyme
Hormonal
Glycolytic
The “ileal brake” mechanism slows motility when:
Fat present in ileum
Acid in duodenum
Carbohydrates in jejunum
Sodium in colon
Pepsin works best at pH:
1–3
6–7
8–9
4–5
Gastric mucosal cells that secrete pepsinogen are:
Parietal cells
Chief cells
G cells
Enterochromaffin cells
Secretin release mainly occurs from the:
Duodenum
Stomach
Colon
Esophagus
CCK also stimulates:
Pancreatic enzyme secretion
Gastric HCl
Large intestine absorption
Hepatic glucose output
Hepatocytes produce:
Gastrin
Bile
Insulin
Amylase
The main stimulus for gastric motility is:
Food presence
Secretin
Passive diffusion
Glucagon
A patient with CFTR mutation (cystic fibrosis) often has:
Excess bile
Thick pancreatic secretions
Excess bicarbonate
Low gastrin
The hormone that increases pancreatic bicarbonate secretion is:
Secretin
CCK
Gastrin
Motilin
The main product of carbohydrate digestion absorbed is:
Sucrose
Glucose
Maltose
Starch
Enterohepatic circulation recycles:
Bile salts
Pepsinogen
Trypsinogen
Amylase
The major ion in pancreatic juice is:
Cl−
HCO3−
Na+
Ca2+
A decrease in bile acids reduces:
Water absorption
Fat absorption
Protein digestion
Carbohydrate digestion
Pancreatic duct cells secrete:
Enzymes
Bicarbonate
Gastrin
Pepsin
Gastric secretion of HCl is directly stimulated by:
Secretin
Histamine
Motilin
CCK
The major buffering component of pancreatic juice is:
NaCl
HCO3−
Amylase
Trypsin
A patient with ileal resection will have deficiency of:
Fat-soluble vitamins
Glucose
Amino acids
Iron
Brunner’s glands secrete:
Mucus & bicarbonate
Gastrin
Bile
Pancreatic enzymes
The vagovagal reflex enhances:
Gastric secretion
Pancreatic enzymes
Bile release
All of the above
A patient with decreased CCK would have:
Poor fat digestion
High pepsinogen
High HCl
Low bicarbonate
Gastric acid promotes absorption of:
Iron
B12
Fatty acids
Calcium
Trypsinogen → trypsin conversion occurs in the:
Small intestine
Stomach
Pancreas
Liver
Secretin’s main role is to:
Neutralize acid in duodenum
Increase HCl
Increase motilin
Inhibit bile
A patient with gastric bypass may have deficiency of:
B12
Glucose
Iron
Both A & C
A hormone released by small intestine that inhibits gastric emptying is:
GIP
CCK
Motilin
Gastrin
The major stimulus for pepsinogen is:
Vagus nerve
CCK
Secretin
GIP
Bile pigments are derived from:
Cholesterol
Bilirubin
Amino acids
Fatty acids
Pancreatic secretions have high bicarbonate because they:
Neutralize chyme
Digest proteins
Activate gastrin
Inhibit motility
The hormone that increases during a fatty meal is:
CCK
Secretin
Gastrin
Motilin
Bile acids are conjugated with:
Glycine & taurine
Glucose & fructose
Alanine & valine
Histamine & histidine
A patient with steatorrhea likely has:
Fat malabsorption
Iron deficiency
Lactose intolerance
Acid reflux
Pancreatic enzyme secretion is greatest during:
Intestinal phase
Cephalic phase
Gastric phase
Fasting
Secretin inhibits:
Gastric HCl
Pancreatic bicarbonate
CCK release
Bile production
