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Worksheetspatho renal
Total questions: 74
Worksheet time: 41mins
dysuria
<400mL of urine
painful urination
<100mL of urine
accumulation of nitrogenous wastes
functional part of the kidney
nephron
loop of henle
distal tubule
glomerulus
#1 cause for renal disease
obesity
HTN
diabetes
anemia
kidneys regulate production of
erythropoietin
vit C
renin
vit D
first step in renin
rise in BP
ACE rxn
drop in BP
angiotensin II
2nd step in renin
angiotensin I
drop in BP
angiotensin II
ACE rxn
3rd step in renin
angiotensin I
aldosterone
ACE rxn
angiotensin II
4th step in renin
aldosterone
angiotensin I
ACE rxn
angiotensin II
5th step in renin
ACE rxn
aldosterone secreted
angiotensin I
drop in BP
6th step in renin
drop in BP
angiotensin II
rise in BP
aldosterone secreted
inflmn of kidney medulla, parenchyma, renal pelvis, affecting tubules
acute pyelonephritis
chronic pyelonephritis
cystitis
responds well to 2wks of abx
acute pyelonephritis
chronic pyelonephritis
cystitis
structural abnormalities. impairs fxn of kidneys
acute pyelonephritis
chronic pyelonephritis
cystitis
inflmn of bladder
pyelonephritis
cystitis
BPH
bacteria contamination by vaginal secretions, prostatic hypertrophy, close proximity of urethra to anus
pyelonephritis
cystitis
BPH
frequency, urgency, burning w urination, hematuria, cloudy urine, foul odor
BPH
pyelonephritis
cystitis
AKI
urinalysis, urine culture
cystitis
pyelonephritis
BPH
AKI
vesicoureteral reflux, kidney stones, neurogenic bladder
pyelonephritis
cystitis
BPH
AKI
fever, chills, flank pain, WBC in medulla, renal inflmn, purulent urine
BPH
cystitis
pyelonephritis
AKI
urinalysis, urine culture, CBC
AKI
cystitis
BPH
pyelonephritis
dx for UTI
clean catch urine
bladder scan
culture & sensitivity
MRI
BPH
malignant
non cancerous
enlarged prostate
prostatitis
BPH
cystitis
BPH
not infxn related
infxn related
treated but not cured
curable
pain w urination
inflmn of prostate
cystitis
BPH
prostatitis
pyelonephritis
prostatitis
caused by infxn
not infxn related
curable
pain w ejaculation & constant pain
treated but not curable
most common kidney stone: (a) oxylate
stage 1 of renal calculi
aggregation
growth
nucleation
stage 2 renal calculi formation
growth
aggregation
nucleation
stage 3 renal calculi
aggregation
growth
nucleation
stone forming, nucleation & crystallization
unsaturated urine
supersaturated urine
crystals cant form, crystals dissolve
unsaturated urine
supersaturated urine
most important renal calculi inhibitor (a)
major cause of kidney stone formation (a)
R/F for kidney stones
high temp environment
high sodium diet
excess animal protein
vit C
vit D
most affected by kidney stones
females
males
impaired kidney fxn, develops rapidly over few hrs or days, high mortality rate
BPH
AKI
pyelonephritis
CKD
sudden reduction in blood flow, ischemia of nephrons, sharp decrease in urine output
postrenal AKI
intrarenal AKI
prerenal AKI
renal parenchymal injury, direct damage to kidneys, ATN, interstitial nephritis, glomerulonephritis
prerenal AKI
intrarenal AKI
postrenal AKI
dx kidney fxn test (a)
by product of muscle metabolism, measures how well kidneys are working
creatinine
BUN
GFR
by product of protein metabolism
GFR
BUN
creatinine
damaged glomeruli, >3.5g of protein leak in urine
AKI
pyelonephritis
cystitis
nephrotic syndrome
weight gain, edema, foamy urine, loss of appetite
nephrotic syndrome
BPH
cystitis
pyelonephritis
post streptococcal glomerulonephritis
group A beta hemolytic streptocci
acute onset
develop 1-2 wks after throat/skin infxn
progressive, leading to chronic kidney disease
obstruction in urinary tract from tubule to urethral meatus, trauma, edema
post renal failure
intra renal failure
pre renal failure
abd. distention w complete obstruction, suprapubic tenderness w palpation
intra renal s/sx
pre renal s/sx
post renal s/sx
BUN
2.5-4.5
8-20
0.6-1.2
18-60
creatinine
0.6-1.2
8-20
18-60
9-11
G1
60-89
>90
45-59
15-29
G2
60-89
>90
<15
15-29
G3a
<15
15-29
60-89
45-59
G3b
<15
60-89
15-29
30-44
G4
>90
<15
15-29
45-59
G5
<15
15-29
30-44
60-89
stage 1&2 CKD
blood/protein in urine
fam hx of polycystic kidney disease
urination changes
kidney pain felt in back
evidence of kidney damage in MRT, CT or xray
stage 3&4 CKD
fluid retention, SOB
kidney pain felt in back
sleep problems
bad breath
headaches
stage 5 CKD
headaches
difficulty concentrating
muscle cramps
changes in skin color
little/no urine
urine specific gravity
1.025-1.040
1.00-1.015
1.010-1.025
low potassium foods
green beans, cabbage
apples, celery
almond milk, popcorn
avocados, broccoli
tomatoes, oranges, almonds
high potassium foods
popcorn, rice
avocados, potatoes
bananas, milk
oranges, tomatoes, peas
hyperkalemia
excessive urine output
diminished urine output
cardiac arrest, weak pulse
leg cramps, abnormal heart rhythm
hypokalemia
excessive urine output
diminished urine output
leg cramps, constipation
cardiac arrest, nausea
care of av fistula
no bp, iv, lab draws on same arm
thrill should be palpated
bruit should be auscultated
no bruit or thrill should be palpated or ausculatated
nephrotoxins (harmful to kidneys)
IV radiocontrast
abx
NSAIDS
salicylates
what substance is likely to appear when glomerulus is inflamed
urea
albumin
sodium
creatinine
What is the cause of most cases of pyelonephritis
Dialysis or other invasive procedure
Abnormal immune response, causing inflammation
An ascending infection by E. coli
Severe pH imbalance of urine
Which disease is manifested by dysuria and pyuria
Cystitis
Glomerulonephritis
Urolithiasis
Pyelonephritis may be distinguished from cystitis by the presence in pyelonephritis of
microbes, leukocytes, and pus in the urine
painful micturition
urgency and frequency
urinary casts and flank pain
What are the significant signs of nephrotic syndrome
Hyperlipidemia and lipiduria
Pyuria and leucopenia
Hypertension and heart failure
Which factor contributes to severe anemia in individuals with chronic renal failure
Compensatory increase in bone marrow activity
Increased erythropoietin secretion
Limited protein intake
Inability to absorb vitamin B12 and iron
Excess urea and other nitrogen wastes in the blood is referred to as
dysuria
azotemia
bacteremia
hematuria
what type of edema is in nephrotic syndrome
interstitial
pulmonary
cerebral
CKD progression
ELIMINATION of nitrogenous waste DIMINISHES
Metabolic ACIDOSIS increases
POTASSIUM levels INCREASE
body not efficiently producing RBCs
sodium & water RETAINED
