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Worksheets96 physio
Total questions: 118
Worksheet time: 59mins
Through the nerve conductors, the nervous system receives
influenza A virus
streptococcal exotoxin
herpes viruses
pneumococci
adenovirus
The consequence of disinhibition syndrome can be
development of dystrophic changes in neurons and innervated structures
development of organ atrophy
development of deafferentation syndrome
development of denervation syndrome
formation of the GPUK (pathologically enhanced excitation generator)
The importance of the formation of the GPUV
promotes the formation of diffuse inhibition
promotes the formation of a pathological system
promotes the formation of the physiological system
increases the trophic effect of the neuron on the innervated structures
inhibits the development of neuropathological processes
Involuntary, slow, “worm-like” movements localized in the upper limbs, especially in the fingers, are designated by the term
convulsions
athetosis
ticks
chorea
tremor
The most common cause of hemorrhagic stroke is
arterial hypertension
stenotic atherosclerosis of cerebral vessels
thrombosis and embolism of cerebral vessels
angiospasm of cerebral vessels
increased hematocrit
The main causes of meningitis in newborns are
Neisseria meningitidis
pneumococci (Streptococcus pneumoniae)
Haemophilis influenzae
staphylococci, pseudomonas aeruginosa, mycobacterium tuberculosis
Ischerichia coli, beta-hemolytic streptococcus
The cause of meningitis in adults is mainly
Neisseria meningitidis
pneumococci (Streptococcus pneumoniae)
Haemophilis influenzae
staphylococci, pseudomonas aeruginosa, mycobacterium tuberculosis
Ischerichia coli, beta-hemolytic streptococcus
A neurological examination of a 30-year-old man revealed a decrease in the strength and amplitude of voluntary movements of the left arm and left leg. What term is used to describe this condition?
tetraplegia
paraparesis
hemiparesis
paraplegia
atony
A neurological examination of a 35-year-old woman revealed a complete lack of voluntary movements due to a disruption of the innervation of the corresponding muscles. What term is used to describe this condition?
paralysis
athetosis
paresis
chorea
akinesia
Central paralysis is described
preservation of voluntary movements
weakening of contractile reflexes
increased contractile reflexes
absence of pathological reflexes
decreased muscle tone
Peripheral paralysis occurs when
partial damage to the trunk of the peripheral nerve (sensory fibers)
lesion of neurons of the anterior horns of the spinal cord
lesion of neurons of the posterior horns of the spinal cord
lesion of the first neuron of the pyramidal tract
lesions of the basal ganglia of the extrapyramidal system
Central paralysis occurs when
partial damage to the trunk of the peripheral nerve (sensory fibers)
lesion of neurons of the anterior horns of the spinal cord
lesion of neurons of the posterior horns of the spinal cord
lesion of the first neuron of the pyramidal tract
in case of damage to the basal ganglia of the extrapyramidal system
Brown-Sequard syndrome occurs when
damage to the anterior horns of the spinal cord
complete transverse spinal cord injury
half lateral spinal cord injury
damage to the midbrain
damage to the pituitary gland
Causalgia develops when
amputations of limbs
complete transection of nerve trunks
incomplete transection of nerve trunks
decreased excitability of the cerebral cortex
increased excitability of the cerebral cortex
A 73-year-old woman presented with muscle weakness and paresthesia in her arms and legs. Neurological examination revealed weakness of the flexors and extensors of the lower extremities, increased knee reflexes, and decreased vibration sensitivity. The patient most likely has a vitamin deficiency.
RR
D
B12
C
A
In a 60-year-old man, in the early period after an acute cerebrovascular accident, the severity of neurological symptoms continued to increase, despite the restoration of blood flow in the damaged cerebral vessel. The cause of the worsening of neurological symptoms in the patient is
decrease in neuronal osmolarity
shrinkage of neurons
neuronal reperfusion
calcium release from neurons
inhibition of glutamate receptors
A 28-year-old man has clonic seizures, twitching of the neck, face and trunk muscles with a short period of apnea, and foamy saliva coming out of the mouth. After the seizure, which lasts 2 minutes, the muscles relax. This typical form of pathology is based on
decreased neuronal excitability
suppression of neuronal discharge generation
destruction of dopamine neurons in the area blue spot
increased neural activity with hypersynchronous discharges
predominance of cholinergic system activity in the striatum
A young man of 18 years old was admitted to the clinic with complaints of headache, vomiting that did not bring relief, and elevated body temperature. Examination revealed rigidity of the occipital muscles. The patient is in a characteristic pose: head thrown back, back arched, stomach drawn in, legs bent and brought to the stomach. In the pathogenesis of this disease, of great importance is
intracranial hypertension
hypohydration of neurons
narrowing of cerebrospinal fluid spaces
reduction in the formation of cerebrospinal fluid
decrease in the permeability of the blood-brain barrier
A 60-year-old man, 10 years after being diagnosed with Syphilis, was found to have decreased tendon reflexes, a feeling of numbness and crawling "ants", and a tingling sensation. What typical form of neurogenic sensitivity disorders is observed in the patient?
athetosis
myasthenia
paresthesia
allodynia
hyperpathy
A 40-year-old man has been diagnosed with damage to the motor neurons of the lumbar spine. What will a neurological examination of the lower extremities reveal?
increased spinal reflexes
appearance of pathological reflexes
muscle hypertrophy
muscle hypotonia
muscle hypertonicity
A 40-year-old man has been diagnosed with damage to the motor neurons of the cervical spine. What will a neurological examination of the lower extremities reveal?
preservation of voluntary movements
weakening of tendon reflexes
increased tendon reflexes
absence of pathological reflexes
decreased muscle tone
A 17-year-old boy developed pain in the big toe of his right foot after surgical amputation of the same foot. What is the mechanism of pain?
ectopic impulses from neuromas in the severed nerve of the stump causing central sensitization
decreased nociceptor density in the stump reducing afferent input
ischemia of the amputated limb tissues
inflammation of the toe joint
activation of vestibular pathways
The reduction in pain sensitivity during massage is due to
decreased sensitivity of nociceptors
blockade of nerve conductors
decreased excitability of neurons in the reticular formation
suppression of excitability of thalamic neurons
activation of inhibitory neurons of the gelatinous substance of the spinal cord
A 25-year-old woman developed double vision and respiratory muscle dysfunction. Botulism was diagnosed. What is the most likely mechanism for the muscle dysfunction?
inhibition of glycine secretion into the synaptic cleft
inhibition of acetylcholine secretion into the synaptic cleft
inhibition of norepinephrine secretion into the synaptic cleft
blocking of glycine-sensitive receptors on the postsynaptic membrane
blocking of acetylcholine-sensitive receptors on the postsynaptic membrane
An 18-year-old young man was admitted to the clinic complaining of headache, vomiting that did not bring relief, and elevated body temperature. Examination revealed rigidity of the occipital muscles. The patient is in a characteristic pose: head thrown back, back arched, stomach pulled in, legs bent and brought to the stomach. The pathogenesis of headache in this disease is due to
irritation of the endings of the trigeminal nerve that innervate the membranes of the brain
irritation of the vagus nerve receptors located at the bottom of the fourth ventricle
irritation of the vomiting center in the reticular formation of the medulla oblongata
irritation of the posterior roots and cells of the spinal ganglia
reflex tonic contraction of muscles
A young man of 18 years old was admitted to the clinic with complaints of headache, vomiting that did not bring relief, and elevated body temperature. Examination revealed rigidity of the occipital muscles. The patient is in a characteristic pose: head thrown back, back arched, stomach drawn in, legs bent and brought to the stomach. The pathogenesis of the characteristic posture in this disease is due to
irritation of the endings of the trigeminal nerve
increased tone of parasympathetic and sympathetic fibers
irritation of the vagus nerve receptors located at the bottom of the fourth ventricle
irritation of the posterior roots and cells of the spinal ganglia
reflex tonic contraction of muscles, rigidity of the occipital muscles
Cerebellar ataxia, memory disorders for current events, nystagmus, dysarthria, dysphagia, hiccups, dizziness are characteristic of cerebrovascular accidents due to damage to
vertebral artery
anterior cerebral artery
middle cerebral artery
internal carotid artery
pial arteries
Paresis or spastic paralysis of the limbs (proximal arm and distal leg), loss of sensitivity on the side opposite the lesion, most likely observed with damage to
vertebral artery
anterior cerebral artery
middle cerebral artery
posterior cerebral artery
pial arteries
A 64-year-old man was diagnosed with ischemic stroke, revealed: decreased sensitivity and paresis of the muscles of the left leg, positive Babinsky reflex on the left. Which artery embolism caused these changes?
left vertebral artery
left anterior cerebral artery
left middle cerebral artery
right posterior cerebral artery
right anterior cerebral artery
A 50-year-old man with a history of atrial fibrillation suddenly felt weakness in the muscles of the left half of his body and lost his sight. The symptoms persisted for several weeks. Most likely the patient
brain tumor
hematoma
stroke due to cerebral embolism
stroke due to rupture of blood vessels in the brain
transient ischemic attack
During the examination of a 65-year-old homeless man, the following was revealed: multiple cuts on the feet and a bilateral decrease in pain and temperature sensitivity of the stocking and glove type, a pronounced smell of alcohol from the mouth. Most likely, the man
polyneuritis
syringomyelia
amyotrophic lateral sclerosis
stroke
parkinsonism
During examination of a 35-year-old woman, the following was revealed: paresis of the right arm, reflexes on the right arm are reduced. Reflexes of the right leg are increased and the Babinski reflex is determined. Reflexes of the left half of the body are normal. What brain structures are damaged?
peripheral nerves
anterior roots of the spinal cord
spinal cord
brain stem
alpha motor neurons
An 18-year-old boy was diagnosed with pharyngitis. A few days later, he developed a severe headache and a fever of 38.8°C. Revealed: stiff neck, positive Kernig and Brudzinski reflexes. Cerebrospinal fluid analysis: high protein, low glucose, high neutrophil count. What causes this disease?
Neisseria meningitidis
Cryptococcus neoformans
Mycobacterium tuberculosis
Toxoplasma gondii
Poliovirus
A 26-year-old female student presented with a history of increasing weakness and headaches over several weeks. Neurological examination revealed no significant changes. Cerebrospinal fluid analysis: colorless fluid with a slight increase in protein, normal glucose content, lymphocytes present, no neutrophils. Antibodies to which pathogens would a serological examination most likely reveal?
Toxoplasma gondii
Listeria monocytogenes
Cryptococcus neoformans
Neisseria meningitidis
Echovirus
A 15-year-old boy was admitted to the clinic with complaints of headache, fever, muscle weakness. Muscle hypotonia and hyporeflexia were detected. The diagnosis was poliomyelitis. What is the basis for the damaging effects of the polio virus?
demyelination of axons of motor neurons of the anterior horns of the spinal cord
demyelination of axons of cortical motor neurons
myocyte damage
damage to neuromuscular synapses
destruction of motor neurons of the anterior horns of the spinal cord
After a stroke, a 67-year-old man developed episodes of severe, difficult to bear polytopic pain, combined with vegetative and motor disorders. Most likely, the pathogenesis of these pains is due to
suppression of brainstem excitability
formation of the GPUV in the thalamus
increased conductivity of nerve trunks
decreased excitability of the cerebral cortex
formation of the GPUC in the spinal cord
The correct statement is
hemorrhagic stroke is most often the result of arterial hypertension
the cause of cerebral embolism is deep vein thrombosis of the lower extremities
the most common route of infection to the central nervous system is through nerve conductors
causalgia is a decrease in pain sensitivity
pain mediator – GABA
A 50-year-old patient consulted a doctor complaining of limited voluntary movement in his left arm and leg. A year ago, he suffered a cerebral hemorrhage. During examination, the left arm was sharply bent and brought to the body, the left leg was sharply straightened. Muscle tone and periosteal reflexes of the indicated extremities were increased. There were pathological tendon reflexes. Are these disorders based on damage?
corticospinal pyramidal tract, disinhibition of α-motor neurons of the spinal cord
corticospinal pyramidal tract, disinhibition of spinal cord gamma motor neurons
corticospinal pyramidal tract, destruction of alpha-motor neurons of the spinal cord
corticospinal pyramidal tract, destruction of gamma motor neurons of the spinal cord
precentral gyrus
A 50-year-old patient consulted a doctor complaining that after an injury to the back of the right thigh, active movements in this limb became sharply limited, and atrophy of the calf muscles appeared. During the examination, along with muscle atrophy, a lack of reflex from the Achilles tendon was noted. Name the form of nervous system dysfunction
peripheral paresis of the right lower limb
peripheral paralysis of the right lower limb
central paresis of the right lower limb
central paralysis of the right lower limb
amyotrophic lateral sclerosis
After a cerebrovascular accident, the patient developed spastic contraction of the muscles of the right arm and right leg. Muscle tone in these limbs is increased. Voluntary movements of these limbs are impossible, tendon and periosteal reflexes are increased. Muscle atrophy is not observed. Determine the form of akinesia in this patient
right-sided hemiplegia, central paralysis
right-sided hemiparesis
right-sided hemiplegia, peripheral paralysis
Parkinson's disease
parkinsonism
The reason for the heart being overloaded with blood volume is
coarctation of the aorta
arterial hypertension
arterial hypotension
valvular stenosis
heart valve insufficiency
The reason for the overload of the heart by resistance is
aortic valve insufficiency
hypervolemia
arterial hypertension
physical activity
overhydration
The cause of absolute coronary insufficiency is
atherothrombosis of coronary arteries
paroxysmal tachycardia
hypercatecholaminemia
metabolic disorders in the myocardium
activation of b-adrenergic receptors of coronary vessels
The cause of relative coronary insufficiency is
stenotic coronary sclerosis
paroxysmal tachycardia
coronary artery spasm
metabolic disorders in the myocardium
coronary artery thromboembolism
Prevents the restoration of microcirculation after myocardial reperfusion
formation of active oxygen species
negative charge of endothelial cells
increased collateral blood flow
accumulation of adenosine at the site of ischemia
In pathogenesis secondary arterial hypertension is important
persistent increase in excitability of sympathetic nerve centers
chronic excitation of emotional centers
reduction of the inhibitory effect of the cerebral cortex on the vasomotor center
hereditary defect of membrane ion pumps of vascular myocytes
excessive production of aldosterone
A genetic defect in the cell membranes of vascular myocytes leads to hypertension due to
increase in calcium content in the cytoplasm of vascular smooth muscle cells
increase in the electrical potential of the cell membrane
increasing the rate of reuptake of mediators by nerve endings
suppression of ATPase activity of myosin
reducing the time of action of mediators on the vascular wall
In the pathogenesis of renovascular renal hypertension is important
activation of the renin-angiotensin-aldosterone system
reduction of renal parenchyma
reduction in the production of depressant substances in the kidneys
increased secretion of renal kinins
decreased sodium reabsorption in the kidneys
In the pathogenesis of renoparenchymatous arterial hypertension is important
activation of the renin-angiotensin-aldosterone system
decreased secretion of erythropoietin
increased reabsorption of water in the kidneys
decrease in secretion of renal kinins, prostaglandins
increased sodium reabsorption in the kidneys
Pathogenetic treatment of arterial hypertension includes the appointment of
painkillers
cardioprotectors
ACE inhibitors
antibacterial
bronchodilators
The clinical sign of right ventricular failure is
hemoptysis
pulmonary hypertension
expiratory dyspnea
ascites
pulmonary edema
Right ventricular failure is characterized by
pulmonary edema
hepatomegaly
hemoptysis
cardiac asthma
hypertension of the pulmonary circulation
A manifestation of left ventricular heart failure is
venous congestion of blood in the systemic circulation
swelling of the jugular veins
pulmonary edema
ascites
hepatomegaly
The emergency stage of cardiac hyperfunction is characterized by
cardiomyocyte hypertrophy
proliferation of connective tissue
myogenic dilation
hyperfunction of non-hypertrophic myocardium
normalization of oxygen consumption, energy production, protein biosynthesis per unit of myocardial mass
The cause of the myocardial form of heart failure is
streptococcal infection
hypertension
hypervolemia
stenosis of the heart valves
tricuspid valve insufficiency
The cause of myocardial insufficiency of non-coronary origin is
coronary thrombosis
hypercatecholaminemia
coronary atherosclerosis
tricuspid valve insufficiency
primary metabolic disorder in the heart muscle
The compensation stage of heart failure is characterized by
tonogenic dilatation
shortness of breath at rest
decrease in stroke volume
increase in residual blood volume in the heart cavities
myogenic dilatation
The method of pathogenetic therapy for ventricular fibrillation is
defibrillation of the heart
prescription of cardiac glycosides
use of antihypertensive drugs
use of sedatives
prescription of central analeptics
The leading link in the pathogenesis of decreased blood pressure in cardiogenic shock
decreased contractility of the myocardium
decreased tone of resistive vessels
pathological deposition of blood
hypoxia and metabolic acidosis
pathological vasoconstriction
The cause of right ventricular failure is
arterial hypertension of the large circle of blood circulation
non-closure of the interventricular septum
infarction of the anterior wall of the left ventricle of the heart
mitral valve insufficiency
myocarditis
Congenital heart defect accompanied by severe cyanosis
narrowing of the pulmonary artery orifice
low ventricular septal defect
patent ductus arteriosus
tetralogy of Fallot
aortic coarctation
Leads to right ventricular failure
arterial hypertension of the large circle of blood circulation
pneumosclerosis
infarction of the anterior wall of the left ventricle of the heart
mitral valve insufficiency
myocarditis
Specific markers for myocardial infarction are
myoglobin
troponins TnT and TnI
ALT, AST
LDH-1/LDH-2 ratio
alkaline phosphatase
In the pathogenesis of an increase in blood deoxyhemoglobin in heart failure, it is important
acceleration of blood flow
increased oxygenation of blood in the lungs
increased oxygen utilization by tissues
increase in oxygen capacity of blood
decreased oxygen utilization by tissues
In the pathogenesis of sinus bradycardia is important
slowing of spontaneous diastolic depolarization of the sinus node
increased sympathoadrenal effects on the heart
the appearance of fault currents
In the pathogenesis of sinus (respiratory) arrhythmia is important.
Formation of an ectopic impulse focus
Fluctuations in vagal tone
Impaired conduction of excitation from the atria to the ventricles
Re-entry mechanism
Decrease in the threshold potential of pacemaker cells
The overload form of heart failure occurs when.
Myocardial infarction
Myocarditis
Heart valve insufficiency
Atherosclerosis of the coronary arteries
Coronary artery thromboembolism
The consequence of the mechanism of re-entry of the excitation wave is.
Atrial fibrillation
Sinus bradycardia
Sinus tachycardia
Atrioventricular block
Sinus arrhythmia
Characterize complete transverse heart block.
Tachycardia attacks
Asynchronous contractions of the atria and ventricles
Alternating periods of increased and decreased heart rate
"Interruptions" in the heart
Nomotopic heart rhythms
Manifestation of left ventricular failure.
Venous congestion of blood in the systemic circulation
Cardiac asthma
Swelling of the jugular veins
Ascites
Splenomegaly
After the drug was prescribed, the patient's blood pressure increased and the total peripheral resistance to blood flow decreased. The drug caused.
Vasoconstriction and decrease in cardiac output
Vasodilation and increase in cardiac output
Vasoconstriction and increase in cardiac output
Vasodilation and decrease in cardiac output
Vasoconstriction and cardiac output did not change
Stress → increased corticosteroids → increased synthesis of angiotensinogen and angiotensin-converting enzyme → ? → vasospasm → increased total peripheral vascular resistance → arterial hypertension. The missing link in the pathogenesis of arterial hypertension.
Increased water reabsorption in the kidneys
Increased sensitivity of vascular myocytes to catecholamines
Increased formation of angiotensin II
Decrease in total peripheral vascular resistance
Disorder of aldosterone secretion
A common feature of chronic renal failure, pheochromocytoma, Conn's syndrome, coarctation of the aorta and acromegaly is.
Arterial hypotension
Systemic vasculitis
Venous thrombosis
Arterial hypertension
Obliterating endarteritis
Patient B., 56 years old, complains of dyspnea at rest, swelling of the legs, and attacks of suffocation at night. Objectively: the liver is significantly enlarged. Free fluid is detected in the abdominal cavity. The left border of the heart is displaced by 2.5 cm from the midclavicular line to the left, and the right border is displaced by 2.5 cm to the right from the right edge of the sternum. What form of heart failure does the patient have?
Myocarditis
Angina pectoris
Myocardial infarction
Acute heart failure
Chronic heart failure
Patient K., 66 years old, complains of shortness of breath at rest, swelling of the legs. Objectively: The liver is significantly enlarged. Free fluid is detected in the abdominal cavity. What form of heart failure by localization does the patient have?
Overload
Myocardial
Left ventricular
Right ventricular
Chronic
Patient I., 52 years old, was admitted to the therapeutic department complaining of severe pain in the heart area that did not subside after taking nitroglycerin. Blood tests revealed neutrophilic leukocytosis, increased AST and CPK-MB activity, increased troponins T and I. ECG: sinus rhythm, deepened Q wave and ST segment elevation in lead I with a mirror image in lead III. What form of heart failure does the patient have?
Myocarditis
Angina pectoris
Myocardial infarction
Idiopathic cardiomyopathy
Chronic heart failure
A female student, attending a surgical operation for the first time, suddenly experienced a feeling of "faintness", which was accompanied by tinnitus, dizziness, nausea and led to loss of consciousness. Objectively: the skin and mucous membranes are very pale, the extremities are cold to the touch. BP 70/30 mm Hg. Breathing is rare. Spraying her face with cold water and inhaling ammonia vapors quickly brought the patient to consciousness. What type of circulatory failure does the patient have?
Acute cardiac
Chronic heart disease
Cardiovascular
Acute vascular
Chronic vascular
Patient K., 34 years old, was admitted to the clinic with a fracture of the right femur. The following day, sharp pains in the chest appeared, the skin became cyanotic, the respiratory rate was 36, the pulse was 120, blood pressure was 85/60 mm Hg, and the heart rate was within normal limits. There was a sharp swelling of the jugular veins. The liver was enlarged. What type of heart failure does the patient have based on location?
Overload
Myocardial
Left ventricular
Right ventricular
Chronic
A 38-year-old woman was admitted to the hospital with a fracture of the right femur. The following day, sharp chest pains appeared, the skin became cyanotic, the respiratory rate was 36, the pulse was 120, blood pressure was 85/60 mm Hg, and the heart rate was within normal limits. There was a sharp swelling of the jugular veins. The liver was enlarged. What type of heart failure does the patient have?
Right ventricular
Myocardial
Left ventricular
Sharp
Chronic
Patient A., 63 years old, complains of shortness of breath with minor physical exertion, attacks of suffocation at night, accompanied by a cough with a small amount of liquid transparent sputum. Objectively: skin with a cyanotic tint. Pronounced edema in the legs. Blood pressure 100/70 mm Hg. What type of heart failure does the patient have?
Right ventricular
Myocardial
Left ventricular
Sharp
Chronic
A 46-year-old woman, irritable, a low-level manager, often has conflicts with the staff. Over the past 2–3 years, after stress and conflicts, she has begun to notice an increase in blood pressure. She was brought to the emergency department of the hospital with complaints of headache, anxiety, tremors throughout the body, stabbing pain in the heart area, palpitations, and interruptions. Blood pressure 170/105 mm Hg. After the introduction of a tranquilizer and a β-blocker, blood pressure returned to normal and her health improved. What form of arterial hypertension does the patient have?
Endocrine
Renal
Essential
Neurogenic reflex
Hemodynamic
Patient N., 26 years old, has a constant increase in blood pressure to 170 – 180/90 – 110 mmHg, detected by chance 7–8 years ago; there are changes in the general urine analysis (low specific gravity, proteinuria, erythrocyturia). At the age of 12, after suffering from tonsillitis, swelling of the face and shins was noted, and changes in the urine were detected. Which form of arterial hypertension is the patient’s?
Secondary vasorenal
Secondary renoprivate
Endocrine
Essential
Neurogenic reflex
Patient P., 32 years old, suffers from suddenly beginning and suddenly ending attacks of severe headache with a sharp increase in blood pressure (up to 280 – 300 mmHg), sweating, anxiety, fear, rapid heartbeat, nausea, vomiting. During an attack, the face is red, tremor, hands and feet are cold, heart rate is 100 – 140 per minute. Additional research revealed a tumor of the right adrenal gland. Which form of arterial hypertension is the patient’s?
Secondary vasorenal
Secondary renoprivate
Endocrine
Essential
Neurogenic reflex
A 32-year-old woman suffers from sudden onset and sudden end of severe headache attacks with a sharp increase in blood pressure (up to 280 – 300 mmHg), sweating, anxiety, fear, rapid heartbeat, nausea, vomiting. During an attack, the face is red, tremor, hands and feet are cold, heart rate is 100 – 140 per minute. Additional research revealed a tumor of the right adrenal gland. What hormones are the cause and what mechanism forms the arterial hypertension?
Aldosterone causing sodium and water retention with increased plasma volume
Glucocorticoids causing increased vascular sensitivity to catecholamines
Catecholamines (adrenaline and noradrenaline) causing widespread vasoconstriction and increased cardiac output
Renin causing activation of the renin–angiotensin system with increased angiotensin II
Patient M., 63 years old, suffering from bronchial asthma, had been taking glucocorticoids for several years. During these years, his body weight gradually increased, diabetes mellitus and arterial hypertension developed. What form of arterial hypertension does the patient have?
endocrine
renal
essential
primary
hemodynamic
Patient D., 46 years old, after subtotal resection of the thyroid gland performed for a hormonally active tumor, was prescribed thyroid hormone replacement therapy. Six months later, at an endocrinology appointment, blood pressure was recorded as 140/70 mm Hg. What causes the development of arterial hypertension?
excessive dose of the drug
insufficient dose of the drug
hyperthyroidism
hypofunction of the thyroid gland
inflammation of the thyroid gland
Patient D., 35 years old, who has been observed for 15 years for glomerulonephritis, has had an increase in blood pressure up to 170/120 mm Hg over all these years. What form of arterial hypertension does the patient have?
endocrine
renal
essential
neurogenic reflex
hemodynamic
Patient S., 47 years old, trolleybus driver, complains of headaches, dizziness, nausea. Over the past year, increased blood pressure of 160/95 mm Hg has been observed repeatedly. What form of arterial hypertension does the patient have?
endocrine
renal
essential
neurogenic reflex
hemodynamic
Patient G., 30 years old, complains of significant weight gain. On examination, attention is drawn to a moon‑shaped face, fat deposits mainly on the abdomen and neck, and swelling in the legs. The examination revealed: blood pressure 160/100 mm Hg, hyperglycemia, hypokalemia, increased glucocorticoids in the blood and urine, and an adrenal tumor on ultrasound. What form of arterial hypertension does the patient have?
endocrine
renal
essential
neurogenic reflex
hemodynamic
Patient E., 45 years old, who has been observed for 15 years for glomerulonephritis, has had an increase in blood pressure up to 180/130 mm Hg over all these years. What form of arterial hypertension does the patient have?
endocrine
renal renopriv
essential
neurogenic reflex
renal renovascular
Patient S., 54 years old, suffering from alcoholic liver cirrhosis, underwent paracentesis with evacuation of about five liters of ascitic fluid. Immediately after the intervention, the patient felt sharp weakness, nausea, and darkening of the eyes. Systolic blood pressure was about 60 mm Hg, diastolic was not determined. What pathological process developed in the patient?
traumatic shock
collapse
coma
pain shock
precoma
Patient K., 38 years old, complained of shortness of breath during a routine medical examination during significant physical exertion. He suffers from a congenital heart defect. Objectively: the borders of the heart are expanded to the left and downwards, the cardiac impulse is well expressed. During auscultation, a systolic murmur is heard on the sternum, spreading throughout the chest. Palpation reveals the symptom of "cat purring". The second sound on the aorta is weakened. Blood pressure 110/85 mm Hg, pulse 60 min⁻¹. No significant changes were found in other organs. What type of circulatory failure does the patient have?
acute cardiac
chronic heart disease
cardiovascular
acute vascular
chronic vascular
Patient K., aged 38, complained of shortness of breath during a routine medical examination during significant physical exertion. He suffers from a congenital heart defect. Objectively: the borders of the heart are expanded to the left and downwards, the cardiac impulse is well expressed. During auscultation, a systolic murmur is heard on the sternum, spreading throughout the chest. Palpation reveals the symptom of "cat purring". The second sound on the aorta is weakened. Blood pressure 110/85 mm Hg, pulse 60 min⁻¹. No significant changes were found in other organs. What stage of pathological hyperfunction and myocardial hypertrophy does the patient have according to F.Z. Meerson?
emergency with increased energy production
activation of the genetic apparatus of cardiomyocytes
hyperfunction of nothypertrophied cardiomyocytes
completed hypertrophy and relatively stable hyperfunction
progressive cardiosclerosis and myocardial function depletion
Patient A., 20 years old, has had high blood pressure since childhood, currently 180–200/110–120 mm Hg. There is no influence of psychoemotional factors and practically no fluctuations in blood pressure. There is no headache. It is not possible to normalize blood pressure with antihypertensive drugs. A systolic murmur is heard to the left and right of the navel. General urine analysis is normal. What form of arterial hypertension does the patient have?
endocrine
renal renopriv
essential
neurogenic reflex
renal renovascular
Patient M., 46 years old, developed severe chest pain during intense physical work in the garden, which was relieved by nitroglycerin. In the evening, the pain resumed and was not relieved by nitroglycerin. Shortness of breath and cough with abundant liquid sputum developed. The patient was hospitalized. Objectively: the skin and visible mucous membranes are pale with a cyanotic tint. Respiration 42 min⁻¹, pulse 120 min⁻¹. On auscultation, moist rales of various sizes are heard over the entire surface of the right and left lungs. Cardiac output per minute is 2.8 l, blood pressure 90/60 mm Hg. The oxyhemoglobin content in arterial blood is 81%, and in venous blood 45%. What type of heart failure does the patient have according to pathogenesis?
overload resistance
coronary myocardial
non‑coronary myocardial
overload by volume
pericardial dysfunction
Patient M., 46 years old, developed severe chest pain during intense physical work in the garden, which was relieved by nitroglycerin. In the evening, the pain resumed and was not relieved by nitroglycerin. Shortness of breath and cough with abundant liquid sputum developed. The patient was hospitalized. Objectively: the skin and visible mucous membranes are pale with a cyanotic tint. Respiration 42 min⁻¹, pulse 120 min⁻¹. On auscultation, moist rales of various sizes are heard over the entire surface of the right and left lungs. Cardiac output per minute is 2.8 l, blood pressure 90/60 mm Hg. The oxyhemoglobin content in arterial blood is 81%, and in venous blood 45%. What type of heart failure does the patient have based on localization?
right ventricular
myocardial
left ventricular
sharp
chronic
Patient M., 46 years old, developed severe chest pain during intense physical work in the garden, which was relieved by nitroglycerin. In the evening, the pain resumed and was not relieved by nitroglycerin. Shortness of breath and cough with abundant liquid sputum developed. The patient was hospitalized. Objectively: the skin and visible mucous membranes are pale with a cyanotic tint. Respiration 42 min⁻¹, pulse 120 min⁻¹. On auscultation, moist rales of various sizes are heard over the entire surface of the right and left lungs. Cardiac output per minute is 2.8 l, blood pressure 90/60 mm Hg. The oxyhemoglobin content in arterial blood is 81%, and in venous blood 45%. The content of erythrocytes in peripheral blood is 5.0 × 10¹²/l, leukocytes 19.0 × 10⁹/l. What type of heart failure does the patient have?
right ventricular
myocardial
left ventricular
sharp
chronic
Patient R., 42 years old, has persistent headaches, general weakness, muscle weakness, a crawling sensation, polyuria, and nocturia. Relative urine density is 1001–1002. Blood pressure is 230/120 mm Hg. The blood has an elevated sodium level and a reduced potassium level. Ultrasound examination revealed a tumor of the right adrenal gland. What form of arterial hypertension does the patient have?
endocrine
renal renopriv
essential
neurogenic reflex
renal renovascular
Patient R., 42 years old, has persistent headaches, general weakness, muscle weakness, a crawling sensation, polyuria, and nocturia. The relative density of urine is 1001–1002. Blood pressure is 230/120 mm Hg. The blood has an elevated sodium content and a reduced potassium content. An ultrasound examination revealed a tumor of the right adrenal gland. What hormone does the tumor produce?
aldosterone
adrenalin
thyroxine
cortisol
norepinephrine
Patient T., 54 years old, suffering from arterial hypertension, came to the clinic complaining of periodic dyspnea with difficult and unsatisfied inhalation, especially pronounced during physical exertion. Several days ago, he had an attack of severe inspiratory dyspnea ("suffocation") with fear of death at night; the doctor diagnosed "cardiac asthma". During examination: blood pressure 155/120 mm Hg, X‑ray shows left ventricular dilation. What type of heart failure does the patient have according to pathogenesis?
overload (afterload)
coronary myocardial
non‑coronary myocardial
Restrictive type hypoventilation of the lungs arises at
Edema of the larynx
Hypersecretion of mucus in the bronchi
Bronchiolospasm
Strangulation
Pleurisy
Restrictive respiratory failure is developing as a result of
Edema of the respiratory paths
Blockages of the respiratory paths
Spasm of smooth muscles of the bronchi
Removals easy
Compression of the respiratory paths
Restrictive ventilation disorders may result from
Bronchus tumor
Pulmonary emphysema
Bronchospasm
Hydrothorax
Laryngospasm
For obstruction of the top respiratory paths, it is characteristic to have
Stenotic breathing
Infrequent superficial breathing
Periodic breathing
Difficulty with exhalation
Apneustic breathing
Terminal type breathing refers to
Biot’s breathing
Cheyne–Stokes breathing
Tachypnea
Bradypnea
Gasping breathing
Insufficiency of external respiration with impaired ventilation of the lungs is accompanied by
Increase in the partial pressure of oxygen (pO2) and carbon dioxide (pCO2) in arterial blood
Decrease in pO2 and pCO2 in arterial blood
Decrease in pO2 and pCO2 in venous blood
Increased pO2 and normal pCO2 in the blood
Decrease in pO2 and increase in pCO2 in arterial blood
Obstructive hypoventilation of the lungs occurs when
Impaired respiratory muscle function
Pulmonary atelectasis
Bronchial asthma
Pneumonia
Barbiturate poisoning
The intrapulmonary cause of restrictive hypoventilation of the lungs is
Changes in the pleura and mediastinum
Chest deformity
Ossification of costal cartilages
Ascites
Surfactant deficiency
Surfactant deficiency in newborns is caused by dysfunction of
Pulmonary capillary endothelial cells
Bronchial mucosa cells
Epithelial cells of the respiratory bronchioles
Type I alveolocytes
Alveolar cells type II
A valve mechanism of bronchial obstruction can occur when
Pneumonia
Surfactant deficiency
Lung lobe resections
Pulmonary emphysema
Pulmonary edema
Lower airway obstruction is accompanied by
Reduction of the Tiffeneau index
Difficulty in the inhalation phase
Stenotic breathing
Increase in the Tiffeneau index
Decrease in OOL
Precapillary pulmonary hypertension can develop with
Left ventricular failure
Impaired blood flow from the pulmonary vessels to the left atrium
Thromboembolism of pulmonary arterioles
Compression of the pulmonary veins
Stenosis of the mitral valve orifice
Postcapillary pulmonary hypertension can develop with
Compression of pulmonary arterioles
Obstruction of pulmonary arterioles
Spasm of pulmonary arterioles
Mitral valve defects
Acute decrease in partial pressure of oxygen in inhaled air
Alpha‑1 antitrypsin deficiency is characteristic of
Pulmonary emphysema
Pleural empyema
Pneumonia
Tuberculosis
Exudative pleurisy
Violation of respiratory regulation associated with a lack of excitatory afferentation occurs when
Inhalation of irritating substances (ammonia)
Inhaling hot or cold air
Diaphragmatic paralysis
Drug addiction
Neurotic conditions
Violation of the diffusion properties of alveolar‑capillary membranes is observed in
Tracheitis
Bronchial asthma
Laryngeal edema
Bronchitis
Silicosis
Periodic breathing in which the amplitude and frequency of breathing increases in a wave‑like manner and then decreases is
Biot’s
Cheyne–Stokes
Apneustic
Kussmaul
Gasping
The leading pathogenetic factor of cardiogenic pulmonary edema is
Increased permeability of pulmonary capillaries
Decreased lymph flow
Increased blood oncotic pressure
Decreased aldosterone
Increased hydrostatic pressure in the pulmonary capillaries
