What is NOT a lab that should be done to evaluate for Sjogren's Syndrome?

55 yo female pt presents with slow progression of burning, itching, photophobia, and foreign body sensation in the eyes. Pt also reports dysphagia, feeling of "cotton mouth," dental caries, loss of taste/smell. On PE you note parotid enlargement. What is NOT a possible treatment?

What is NOT something that can impact prognosis of Sjogren's Syndrome patients?

A syndrome of paroxysmal digital ischemia, most commonly caused by an exaggerated response of digital arterioles to cold or emotional stress. Intermittent arteriolar vasospasms. What is the diagnosis?

25 yo female pt with no PMH presents with changes to all fingers symmetrically on each hand, but excluding the thumbs. Pt denies pain, ulcers, and hx of peripheral vascular disease. Labs show negative ANA. What diagnsis do you suspect?

Pt with PMH of autoimmune disease presents with changes to fingers asymmetrically. Symptoms exclude the thumbs. Pt admits to pain and ulcers. What diagnsis do you suspect?

Pt presents with rubor, throbbing/pain, paraesthesia, and slight swelling in the fingers down to the distal palm. Initially it only impacted 1 or 2 fingertips affected. The symptoms improve with warmth. What is NOT included in the treatment?

Pt presents with rubor, throbbing/pain, paraesthesia, and slight swelling in the fingers down to the distal palm. Initially it only impacted 1 or 2 fingertips affected. The symptoms improve with warmth. What is NOT included in the treatment?

A chronic multisystem disease characterized by autoantibody production, collagen deposition, and vascular dysfunction. Fibrosis of skin and internal organs. Name meaning "hard skin." What is the diagnosis?

What is NOT a lab that should be done to evaluate for Scleroderma?

What is NOT an imaging test that can be done to evaluate for Scleroderma?

What is NOT part of CREST syndrome?

30 yo female pt presents with calcinosis, raynaud's phenomenon, esophageal dysmotility, sclerodactyly, telangiectasias, anticentromere antibody (CREST syndrome). PE reveals "salt and pepper" pigment change. What is NOT included in the treatment?

Idiopathic inflammatory myopathy. Autoimmune reaction to muscle tissue in genetically susceptible people. Consider underlying malignancy. Picornavirus-like structures have been found in muscle cells. Cellular damage and atrophy. What is the diagnosis?

50 yo female pt presents with gradual and progressive symmetrical proximal muscle weakness (legs prior to arms). Pt admits to pain, tenderness, dysphagia, muscle atrophy, and contractures. What is the diagnosis?

All of the following are diagnostic tests for Polymyositis, but what is the gold standard?

A multisystem autoimmune connective tissue disease. Complement-mediated vasculopathy. Celluar damage and atrophy. Variable degrees of inflammation. Proximal muscle weakness. What is the diagnosis?

50 yo female pt presents with proximal muscle weakness, fatigue, and rash. PE reveals visceral involvement. Erythematous rash that is slightly elevated and smooth/scaly, heliotrope rash, gottron papules, nail changes, desquamating dermatitis, secondary changes, rash on scalp (calcification in children). What is NOT a possible diagnostic test?

All of the following are diagnostic criteria for dermatomyositis, but what is the definitive test?

What is NOT a possible part of the treatment for polymyositis and dermatomyositis?

What is NOT a possible part of the treatment for polymyositis and dermatomyositis?

What can occur at any age and does not involve the skin?

What is similar to primary idiopathic polymyositis, but also involves the skin?

In what disease is pulmonary disease the leading cause of mortality?

What disease has "salt and pepper" pigment change and CREST syndrome?

What disease has keratoconjunctivitis sicca, xerostomia, and parotid enlargement?