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WorksheetsHematologic Emergencies Practice Questions (Set 3, Part 1)
Total questions: 50
Worksheet time: 25mins
The most common cause of acute blood loss anemia in trauma patients is:
Hemoglobinopathy
Bone marrow suppression
Internal or external hemorrhage
IMHA
A hallmark sign of regenerative anemia is:
Increased reticulocytes
Decreased reticulocytes
Absence of polychromasia
Decreased bilirubin
Which is NOT part of the intrinsic coagulation pathway?
Factor IX
Factor XI
Factor VIII
Factor VII
Patients with thrombocytopenia may show:
Only internal hemorrhage
Hematuria only
Surface bleeding such as petechiae
No clinical signs
Immune-mediated thrombocytopenia (IMT) leads to:
Dehydration
Excess WBC destruction
Excess platelet destruction
Excess RBC destruction
Secondary hemostasis defects typically cause:
Petechiae
Large cavity bleeds
Nail bed hemorrhage only
No visible bleeding
A platelet transfusion is most indicated when:
Fibrinogen is high
PT is prolonged only
Platelet count < 20,000 and active bleeding
PCV is low
The amplification phase of coagulation involves:
RBC agglutination
Platelet activation and factor release
Leukocyte adhesion
Vasoconstriction
Carboxyhemoglobinemia results in:
A low MCV anemia suggests:
Iron deficiency
IMHA
Hemorrhage
Bone marrow neoplasia
A high MCHC is commonly associated with:
Lipemia
Laboratory error or hemolysis
Iron deficiency
Dehydration
A normal activated clotting time (ACT) rule-out helps exclude:
Hypoglycemia
Intrinsic pathway defects
Extrinsic pathway defects
Platelet disorders
Vitamin K therapy is required for:
Thrombocytosis
DIC treatment
Rodenticide toxicity
IMHA
Prolonged PT and aPTT together indicate:
Platelet dysfunction
Global coagulation defect
Hypercoagulability
Normal hemostasis
A nonregenerative anemia lasting longer than 5 days indicates dysfunction in:
Bone marrow
Stomach
Pancreas
Kidneys only
PLR requires intact:
Retina, optic nerve, midbrain
Eyelids only
Cornea only
Lens
Lack of menace but intact PLR suggests:
Optic nerve dysfunction
Cortical blindness
Retinal detachment
Glaucoma
A superficial ulcer best appears under fluorescein as:
Blue streaks
Dull red stain
Bright green stain
No staining
Deep stromal ulcers often require:
Aggressive medical therapy or grafting
Artificial tears only
E-collar alone
No treatment
A common presenting complaint in dogs with acute glaucoma:
Squinting, red eye, fixed dilated pupil
Mild blepharospasm only
Cloudy discharge only
Excessive tearing only
A patient with acute lens luxation may show:
Deep anterior chamber
Retinal bleeding
Aphakic crescent
Proptosis
A hallmark sign of retrobulbar abscess is:
Severe pain when opening mouth
Blepharitis only
Blindness
No pain on opening mouth
Treatment for melting ulcers includes:
Mydriatics only
No medication
Anticollagenases such as serum or EDTA
Steroids
Aqueous flare is seen in:
SARD
Uveitis
Keratoconjunctivitis sicca
Cataracts
Tonometry is contraindicated in:
Glaucoma
Corneal ulcer with risk of perforation
SARD
Uveitis
Pupil dilation that does not respond to light suggests:
SARD
Lens opacity
Uveitis
Acute glaucoma
Retinal detachment may produce:
Painful eye
No menace response
Normal vision
Miotic pupil
A dog with sudden blindness and normal fundus should undergo:
Nasolacrimal flush
Tear testing
PT/PTT
ERG testing
Birds often develop ocular disease secondary to:
Excessive grooming
Trauma or infection
Flea infestation
Hypertension only
The most common location for a foreign body in the eye:
Retina
Under third eyelid
Lens
Optic nerve
The first step in neurologic assessment is:
Mentation evaluation
Gait analysis
Spinal reflexes
PLR
Proprioceptive deficits indicate:
Kidney disease
Neurologic dysfunction in spinal cord or brain
Hyperthyroidism
Hypoglycemia
A generalized seizure involves:
One limb only
Entire body
Only facial twitching
Only vocalization
A focal seizure may involve:
Staring or fly biting
Whole body paddling
Collapse
Immediate coma
A dog in status epilepticus is at risk for:
Slow heart rate only
No complications
Hyperthermia & brain damage
Hypothermia
Vestibular disease hallmark:
Tetany
Blindness
Head tilt
Fever
Central vestibular disease differs from peripheral by:
Vertical nystagmus possible
Normal proprioception
Ear infection always present
Head tilt absent
Dogs with strokes typically:
Decline for weeks
Begin improving within 72 hours
Require immediate surgery
Become comatose
Schiff-Sherrington posture indicates:
Rabies
Brain tumor
Severe spinal cord injury
Metabolic disease
Myasthenia gravis most commonly results in:
Aggression
Painful limbs
Exercise intolerance and collapse
Blindness
Tick paralysis leads to:
Hyperactivity
Ascending paralysis
Seizures
Blindness
Botulism causes:
Fever
Ascending paralysis
Descending paralysis
Only GI signs
Head trauma secondary injury includes:
Hypoxia, inflammation, edema
,
Skull fracture
A dog with severe traumatic brain injury may show:
Red eyes only
Normal mentation
Altered consciousness
Hyperreflexia only
Rabies diagnosis requires:
Blood test
Tear test
Post-mortem brain testing
Urine analysis
Lack of deep pain in spinal injury:
Is a good prognostic sign
Suggests poor prognosis
Suggests mild injury
Indicates vestibular disease
A dog with megaesophagus shows:
Seizures
Diarrhea
Vomiting
Regurgitation
A normal PLR but no menace suggests:
Optic nerve rupture
Cortical blindness
Lens luxation
Glaucoma
Cervical pain with fever suggests:
Stroke
Meningitis
Botulism
Vestibular disease
A patient with wide-based stance likely has:
Vestibular disease
Cerebellar dysfunction
IMHA
Thrombocytopenia
