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Worksheets

Pancreas and Fed, Fasting

Total questions: 60

Worksheet time: 30mins

Name
Class
Date
1.

Which cells of the pancreas are responsible for insulin secretion?

a)

Alpha (α) cells

b)

Beta (β) cells

c)

Delta (δ) cells

d)

Epsilon (ε) cells

2.

What effect does ghrelin have on pancreatic hormone secretion?

a)

Stimulates insulin, inhibits glucagon

b)

Inhibits both insulin and glucagon

c)

Stimulates glucagon, inhibits insulin

d)

Has no effect on pancreatic hormones

3.

Which of the following inhibits glucagon release?

a)

Ghrelin

b)

Somatostatin

c)

Cholecystokinin

d)

Cortisol

4.

What happens when dietary fat is consumed with carbohydrates?

a)

Rapid glucose absorption and sharp insulin peak

b)

Slower gastric emptying and blunted insulin response

c)

Increased glucagon release only

d)

Decreased insulin and GLP-1 secretion

5.

Which hormone is secreted by delta (δ) cells and acts as a universal inhibitor?

a)

Insulin

b)

Somatostatin

c)

Ghrelin

d)

Pancreatic polypeptide

6.

What stimulates pancreatic polypeptide (PP) release?

a)

Fasting and hyperglycemia

b)

Vagal input

c)

Insulin and leptin signaling

d)

Cortisol and stress

7.

Which mechanism triggers glucagon secretion during hypoglycemia?

a)

Activation of ATP-sensitive potassium channels

b)

Closure of ATP-sensitive potassium channels

c)

Inhibition of voltage-dependent calcium channels

d)

Decrease in intracellular calcium concentration

8.

Which statement best describes the relationship between insulin and glucagon?

a)

Both increase blood glucose levels

b)

They act synergistically to lower blood glucose

c)

They have opposing effects

d)

Insulin stimulates glucagon secretion under all conditions

9.

Which statement about ghrelin is TRUE?

a)

It increases insulin sensitivity

b)

It promotes fat breakdown and weight loss

c)

It stimulates growth hormone release and appetite

d)

It suppresses glucagon secretion

10.

What happens to ghrelin levels during fasting?

a)

They decrease

b)

They remain constant

c)

They increase to stimulate hunger

d)

They depend only on insulin levels

11.

Which hormone acts as an “off switch” for digestion?

a)

Ghrelin

b)

Somatostatin

c)

GLP-1

d)

Gastrin

12.

In the pancreas, which stimulus increases trypsinogen secretion?

a)

Fasting

b)

Insulin via cholecystokinin stimulation

c)

High ghrelin levels

d)

Glucagon during stress

13.

What is the role of amylin in pancreatic hormone regulation?

a)

Stimulates glucagon secretion

b)

Slows digestion and inhibits glucagon release

c)

Activates insulin secretion

d)

Stimulates CCK release

14.

Which of the following hormones directly suppresses ghrelin release?

a)

Cortisol

b)

Insulin

c)

Glucagon

d)

Gastrin

15.

Which of the following does NOT stimulate ghrelin?

a)

Glucagon

b)

Growth hormone

c)

Cortisol

d)

Vasopressin

e)

Leutinizing hormone

16.

Which of the following inhibits glucagon release?

a)

Cortisol

b)

Protein-rich meals

c)

Gastrin

d)

Cholecystokinin

e)

GLP-1

17.

Which of the following stimulates glucagon release?

a)

Insulin

b)

Somatostatin

c)

Amylin

d)

Hyperglycemia

e)

Parasympathetic stimulation

18.

Which of the following best summarizes somatostatin’s effects on β-cells?

a)

Promotes cAMP synthesis

b)

Enhances insulin granule exocytosis

c)

Reduces calcium influx

d)

Increases glucagon release from α-cells

19.

Which is the major somatostatin form in intestinal mucosa?

a)

S-14

b)

S-28

c)

S-56

d)

S-GHIH

20.

What effect does fat alone have on glucagon and GLP-1?

a)

Decreases both hormones

b)

Increases glucagon and GLP-1 secretion

c)

Increases insulin and decreases glucagon

d)

No effect

21.

What is the effect of ghrelin on pancreatic hormone release?

a)

Stimulates insulin, inhibits glucagon

b)

Inhibits insulin, stimulates glucagon

c)

Stimulates both insulin and glucagon

d)

No effect on pancreatic hormones

22.

Which hormone is co-secreted with insulin and slows digestion?

a)

Amylin

b)

CCK

c)

GLP-1

d)

Ghrelin

23.

What stimulates somatostatin secretion?

a)

Glucagon and β-cell activity

b)

Ghrelin and insulin

c)

Cortisol and GLP-1

d)

Low blood glucose

24.

What receptor mediates somatostatin’s inhibitory effects on insulin?

a)

SSTR1

b)

SSTR2

c)

β2 receptor

d)

α1 receptor

25.

Which of the following decreases insulin sensitivity in tissues?

a)

Somatostatin

b)

GLP-1

c)

Ghrelin

d)

Gastrin

26.

Which pancreatic hormone promotes hepatic glucose production?

a)

Glucagon

b)

Insulin

c)

Somatostatin

d)

Amylin

27.

Which pancreatic hormone increases during stress or fasting?

a)

Glucagon

b)

Somatostatin

c)

Amylin

d)

GLP-1

28.

What is ghrelin’s effect on glucose levels?

a)

Decreases hepatic glucose production

b)

Increases glycogenolysis and gluconeogenesis

c)

Decreases glucagon secretion

d)

Increases insulin receptor sensitivity

29.

Which of the following correctly matches the hormone to its secreting cell type?

a)

Ghrelin – β-cell

b)

Glucagon – α-cell

c)

Somatostatin – ε-cell

d)

Insulin – δ-cell

30.

Which hormone acts as a paracrine inhibitor of α-cell function?

a)

Insulin

b)

Ghrelin

c)

GLP-1

d)

Gastrin

31.

Which hormones act as insulin counter-regulatory agents?

a)

Aldosterone and vasopressin

b)

Epinephrine, cortisol, and glucagon

c)

Insulin and amylin

d)

Calcitonin and parathyroid hormone

32.

What happens if glucose and lipid levels are not maintained?

a)

Increased osmotic balance

b)

Hypo-osmotic state

c)

Hyperosmotic and glycosylation damage

d)

Reduced oxygen consumption

33.

Which of the following is a long-term complication of poorly controlled diabetes?

a)

Dehydration only

b)

Glycosylation of hemoglobin and vascular damage

c)

Hypoglycemia

d)

Hypotension

34.

What is the C-peptide?

a)

A non-functional byproduct of glucagon

b)

A connecting peptide cleaved to activate insulin

c)

A peptide that inhibits insulin

d)

A cofactor for glucose uptake

35.

How do sulfonylureas stimulate insulin release?

a)

Open K⁺ channels

b)

Block K⁺ channels

c)

Inhibit Ca²⁺ channels

d)

Inhibit glycolysis

36.

What characterizes Type 1 Diabetes?

a)

Insulin resistance

b)

Overproduction of insulin

c)

Autoimmune destruction of β-cells

d)

Excess glucagon synthesis

37.

How does insulin affect carbohydrate metabolism?

a)

Stimulates gluconeogenesis

b)

Inhibits glycolysis

c)

Promotes glucose uptake and storage

d)

Increases glucagon release

38.

How does glucagon affect glycolysis and gluconeogenesis?

a)

Increases glycolysis and decreases gluconeogenesis

b)

Decreases glycolysis and increases gluconeogenesis

c)

Increases both

d)

Decreases both

39.

How does insulin affect fat and protein metabolism?

a)

Stimulates lipolysis and proteolysis

b)

Promotes lipid synthesis and protein synthesis

c)

Inhibits glycogen formation

d)

Decreases amino acid uptake

40.

What is the normal range for blood glucose?

a)

40–60 mg/dL

b)

60–80 mg/dL

c)

80–100 mg/dL

d)

100–140 mg/dL

41.

How does glucagon affect fat and protein metabolism?

a)

Promotes fat and protein storage

b)

Stimulates lipolysis and gluconeogenesis

c)

Increases protein synthesis

d)

Inhibits β-oxidation

42.

What are the products of starch digestion?

a)

Maltose and maltotriose

b)

Sucrose and lactose

c)

Fructose and sucrose

d)

Glucose only

43.

Which glucose transporter is responsible for fructose absorption?

a)

GLUT2

b)

GLUT4

c)

GLUT5

d)

SGLT1

44.

Which drug inhibits SGLT1/SGLT2 to reduce glucose reabsorption?

a)

Miglitol

b)

Metformin

c)

Sotagliflozin

d)

Simvastatin

45.

Which drug promotes GLUT4 translocation by inhibiting gluconeogenesis?

a)

Metformin

b)

Simvastatin

c)

Thiazolidinedione

d)

Sulfonylurea

46.

Which event triggers insulin release from pancreatic β-cells?

a)

Decrease in intracellular ATP

b)

Increase in intracellular ATP

c)

Decrease in intracellular calcium

d)

Increase in glucagon secretion

47.

Which step of insulin biosynthesis occurs in the Golgi apparatus?

a)

Cleavage of signal peptide

b)

Packaging of proinsulin and Zn²⁺ into storage vesicles

c)

Synthesis of preproinsulin on rough ER

d)

Phosphorylation of insulin receptor

48.

How does the drug class Thiazolidinediones (TZDs) affect glucose metabolism?

a)

Inhibits gluconeogenesis directly

b)

Activates PPAR-γ to increase GLUT4 expression

c)

Blocks SGLT1 in intestines

d)

Reduces insulin release from β-cells

49.

During the fed state, which of the following occurs?

a)

Lipolysis increases

b)

Glucagon dominates

c)

Insulin promotes glucose uptake and glycogen synthesis

d)

Protein breakdown increases

50.

Which of the following increases glucagon release?

a)

High glucose

b)

Epinephrine

c)

Insulin

d)

Low amino acid concentration

51.

Which glucose transporter is insulin-dependent?

a)

GLUT1

b)

GLUT2

c)

GLUT4

d)

SGLT1

52.

Which transporter is responsible for glucose sensing in β-cells?

a)

GLUT1

b)

GLUT2

c)

GLUT3

d)

GLUT4

53.

Which drug inhibits maltase, sucrase, and isomaltase?

a)

Metformin

b)

Acarbose

c)

Sotagliflozin

d)

Pioglitazone

54.

Which of the following describes the role of GLUT5?

a)

Transports glucose into muscle

b)

Transports fructose across intestinal cells

c)

Transports sucrose into the liver

d)

Reabsorbs glucose in the kidney

55.

In Type 2 Diabetes, which tissue is most resistant to insulin?

a)

Adipose tissue and skeletal muscle

b)

Pancreas

c)

Intestine

d)

Brain

56.

Which of the following drugs blocks renal glucose reabsorption?

a)

Sulfonylureas

b)

Thiazolidinediones

c)

SGLT2 inhibitors

d)

α-glucosidase inhibitors

57.

How does insulin glargine differ from human insulin?

a)

Has additional Arg residues at B31 and B32

b)

Has shorter half-life

c)

Has reversed amino acids at B28 and B29

d)

Is an oral insulin analog

58.

What modification makes insulin aspart faster-acting?

a)

B30 deletion

b)

Proline at B28 replaced by aspartate

c)

Lysine acetylation

d)

Myristic acid attachment

59.

What are the initial products formed when starch is digested by salivary amylase?

a)

Maltose, maltotriose, and limit dextrins

b)

Disaccharides

c)

Glucose and fructose

d)

Sucralose and galactose

60.

What are the energy sources for the brain during prolonged fasting?

a)

Ketone bodies

b)

Fatty acids

c)

Lactic acid

d)

Glycerol