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Derm/Msk Exam 2 - Jones PPT

Total questions: 74

Worksheet time: 44mins

Name
Class
Date
1.

What is a cellular event that leads to whole muscle force generation being the summation of the force generated by the individual cells

a)

muscle contraction

b)

muscle relaxation

2.

What are the anatomical types of muscles

a)

skeletal

b)

cardiac

c)

visceral

d)

striated

e)

smooth

3.

What are the histological types of muscles

a)

voluntary

b)

involuntary

c)

visceral

d)

striated

e)

smooth

4.

What are the control types of muscles

a)

voluntary

b)

involuntary

c)

visceral

d)

striated

e)

smooth

5.

What are some characteristics of skeletal muscles

a)

large forceful movements

b)

rapid and slow fatigue

c)

source of heat

d)

pumping action to push blood through the circulation

e)

control movement of fluid through visceral organs and vasculature

6.

What describes cardiac muscles

a)

large forceful movements

b)

rapid and slow fatigue

c)

long and short contraction times

d)

pumping action to push blood through the circulation

e)

control movement of fluid through visceral organs and vasculature

7.

What are characteristics of smooth (visceral) muscle

a)

large forceful movements

b)

rapid and slow fatigue

c)

long and short contraction times

d)

pumping action to push blood through the circulation

e)

control movement of fluid through visceral organs and vasculature

8.

What characteristics makes up the morphology of skeletal muscles

a)

sarcolemma

b)

sarcoplasm

c)

multi-nucleated

d)

sarcomere

9.

What are sarcomeres composed of

a)

actin

b)

myosin

c)

myofibrils

d)

transverse tubules

10.

What are characteristics of the sarcomere

a)

repeating cellular structure within the muscle myofibril

b)

surrounded by mitochondria and sarcoplasmic reticulum

c)

titan anchors myosin to α-actin Z disc

d)

control movement of fluid through visceral organs and vasculature

11.

What happens to the I-band during contraction

a)

decreases in size

b)

increases in size

c)

stays the same

12.

What happens to the A-band during contraction

a)

decreases in size

b)

increases in size

c)

stays the same

13.

What are the thin filaments

a)

actin

b)

myosin

c)

tropomyosin

d)

troponin

14.

What are the thick filaments

a)

actin

b)

myosin

c)

tropomyosin

d)

troponin

15.

What describes actin

a)

double helical filament made of g-actin monomers

b)

lies between actin grooves

c)

3 subunit protein attached to tropomyosin

16.

What describes tropomyosin

a)

double helical filament made of g-actin monomers

b)

lies between actin grooves

c)

3 subunit protein attached to tropomyosin

17.

What describes troponin

a)

double helical filament made of g-actin monomers

b)

lies between actin grooves

c)

3 subunit protein attached to tropomyosin

18.

Where are myosin binding sites

a)

actin

b)

tropomyosin

c)

troponin

19.

What covers myosin binding sites

a)

actin

b)

tropomyosin

c)

troponin

20.

What does the tail of myosin contain

a)

light and heavy (S2) meromyosin

b)

α helical dimer

c)

globular region (S1), 2 light chains

d)

ATP enzymatic activity

e)

actin binding sites

21.

What does the head of myosin contain

a)

light and heavy (S2) meromyosin

b)

α helical dimer

c)

globular region (S1), 2 light chains

d)

ATP enzymatic activity

e)

actin binding sites

22.

What is a neuromuscular junction

a)

where a neuron synapses onto a muscle cell

b)

where a muscle cell synapses onto a neuron

23.

What is directly involved in somatic neural signaling

a)

muscle fibers

b)

α-motor neuron

c)

motor endplate

d)

vertebrae

24.

What is the first step in neuromuscular junction

a)

motor neuron action potential (depolarization)

b)

Ca2+ entry voltage-gated channels

c)

Acetylcholine gets released into the synaptic cleft

d)

Na+ enters muscle cell via nicotinic receptors

e)

Local current b/w depolarized end plate and adjacent muscle plasma membrane

25.

In the neuromuscular action potential, what happens after depolarization

a)

muscle fiber action potential initiated

b)

Ca2+ entry voltage-gated channels

c)

Acetylcholine gets released into the synaptic cleft

d)

Na+ enters muscle cell via nicotinic receptors

e)

Local current b/w depolarized end plate and adjacent muscle plasma membrane

26.

In the neuromuscular action potential, what happens after Ca2+ enters voltage-gated channels

a)

muscle fiber action potential initiated through voltage-gated Na+ channels

b)

Propagated action potential in muscle plasma membrane

c)

Acetylcholine gets released into the synaptic cleft

d)

Na+ enters muscle cell via nicotinic receptors

e)

Local current b/w depolarized end plate and adjacent muscle plasma membrane

27.

In the neuromuscular action potential, what happens after Acetyl-CoA gets released into the synaptic cleft

a)

muscle fiber action potential initiated through voltage-gated Na+ channels

b)

Propagated action potential in muscle plasma membrane

c)

Acetyl-CoA degradation

d)

Na+ enters muscle cell via nicotinic receptors

e)

Local current b/w depolarized end plate and adjacent muscle plasma membrane

28.

In the neuromuscular action potential, what happens after sodium enters the muscle cells

a)

muscle fiber action potential initiated through voltage-gated Na+ channels

b)

Propagated action potential in muscle plasma membrane

c)

Acetyl-CoA degradation

d)

Acetylcholinesterase

e)

Local current b/w depolarized end plate and adjacent muscle plasma membrane

29.

In the neuromuscular action potential, what happens after the local current b/w depolarized end plate and adjacent muscle plasma membrane occurs

a)

muscle fiber action potential initiated through voltage-gated Na+ channels

b)

Propagated action potential in muscle plasma membrane

c)

Acetyl-CoA degradation

d)

Acetylcholinesterase

e)

motor neuron action potential (depolarization)

30.

In the neuromuscular action potential, what happens after a muscle fiber action potential is initiated through voltage-gated Na+ channels

a)

Ca2+ entry voltage-gated channels

b)

Propagated action potential in muscle plasma membrane

c)

Acetyl-CoA degradation

d)

Acetylcholinesterase

e)

motor neuron action potential (depolarization)

31.

In the neuromuscular action potential, what happens after a propagated action potential in muscle plasma membrane occurs

a)

Ca2+ entry voltage-gated channels

b)

motor neuron action potential (depolarization)

c)

Acetyl-CoA degradation

d)

Acetylcholinesterase

32.

In a neuromuscular action potential, what does the muscle cell use in acetylcholine degradation

a)

acetylcholinesterase

b)

acetyl-CoA

c)

nicotinic receptors

d)

acetic acid

e)

choline

33.

What are nicotinic acetylcholine receptors

a)

ionotropic receptors

b)

Na+/K+ channels

c)

Ca2+ entry voltage-gated channels

d)

cholinergic synaptic vesicle

34.

What is the role of Na+/K+ channels

a)

driving force in a muscle at rest favors a greater Na+ entry

b)

driving force in a muscle at rest favors a lower Na+ entry

35.

What are some characteristics of muscle action potential

a)

end plate potentials are local (graded) potentials with quantal release

b)

local potentials sum to threshold level depolarization

c)

action potential ion channel permeability changes similar to neural action potential

d)

slightly longer duration than neuronal action potential

36.

After a muscle action potential occurs, the action potential then propagates into the T-tubule structure to signal

a)

Ca2+ to release

b)

Na+ to release

c)

K+ to release

d)

Acetyl-CoA to realease

37.

How does the action potential generate a contraction in excitation-contraction coupling

a)

action potential propagates into T-tubule membrane

b)

DHP (voltage sensor) and the ryanodine receptor is triggered

c)

Ca2+ releases on the sarcoplasmic reticulum

d)

DHP (voltage sensor) and the ryanodine receptor is shut down

e)

Na+ releases on the sarcoplasmic reticulum

38.

What does a skeletal muscle action potential lead to in the sarcoplasm

a)

increased Ca2+

b)

decreased Ca2+

c)

increased Na+

d)

decreased Na+

39.

In the contractile mechanism, what regulates contractions in skeletal muscles

a)

neurons

b)

Ca2+

c)

tropomyosin

40.

In the contractile mechanism, what is cross-bridge cycling

a)

shortening of the sarcomere

b)

lengthening of the sarcomere

41.

What is the first step in cross-bridge cycling

a)

hydrolysis of ATP

b)

binding myosin head to actin (ADP and Pi attached)

c)

ADP and Pi released causes myosin head to "ratchet"

d)

new ATP binds to myosin head and causes detachment from the actin

42.

What occurs in cross-bridge cycling after the hydrolysis of ATP

a)

new ATP binds to myosin head and causes detachment from the actin

b)

binding myosin head to actin (ADP and Pi attached)

c)

ADP and Pi released causes myosin head to "ratchet"

43.

What occurs in cross-bridge cycling after the myosin head is bound to actin (ADP and Pi attached)

a)

new ATP binds to myosin head and causes detachment from the actin

b)

ADP and Pi released causes myosin head to "ratchet"

c)

hydrolysis of ATP

44.

What occurs in cross-bridge cycling after the ADP and Pi released causes myosin head to "ratchet"

a)

new ATP binds to myosin head and causes detachment from the actin

b)

binding myosin head to actin (ADP and Pi attached)

c)

hydrolysis of ATP

45.

What happens when the skeletal muscle relaxes

a)

Ca2+ levels drop through Ca2+ ATPase putting calcium back into the sarcoplasmic reticulum

b)

no more action potential

c)

ACh is removed by AChE

d)

active sites are covered by tropomyosin due to Ca2+ being removed

e)

ACh releases signals to cause a relaxation action potential

46.

What role does ATP have when it comes to muscle energy

a)

energize the head

b)

allow for head detachment

c)

mediate muscle relaxation

47.

Where do muscle cells get ATP from

a)

creatine phosphate

b)

oxidative phosphorylation

c)

glycolysis

d)

lactic acid

e)

blood

48.

What is A

a)

Single Twitch

b)

Unfused Tetanus

c)

Fused Tetanus

49.

What is B

a)

Single Twitch

b)

Unfused Tetanus

c)

Fused Tetanus

50.

What is C

a)

Single Twitch

b)

Unfused Tetanus

c)

Fused Tetanus

51.

What are isotonic muscle contractions

a)

no change in tension

b)

no change in length

c)

muscle changes length

d)

tension increases

52.

What are isometric muscle contractions

a)

no change in tension

b)

no change in length

c)

muscle changes length

d)

tension increases

53.

In an isotonic concentric contraction, the muscle

a)

shortens

b)

tension remains constant

c)

lengthens

54.

In an isotonic eccentric contraction, the muscle

a)

shortens

b)

tension remains constant

c)

lengthens

55.

What does the length-tension relationship of sarcomere (changing sarcomere length alters force generated) determine

a)

isometric contractions

b)

isotonic contractions

56.

What types of tension occurs in isometric contraction

a)

passive

b)

active

c)

slow

d)

fast

57.

Which tension is generated by muscle stretch

a)

passive tension

b)

resting length

c)

active tension

d)

total tension

58.

Which tension is the maximum contractile force

a)

passive tension

b)

resting length

c)

active tension

d)

total tension

59.

Which tension is the force generated by contractile force

a)

passive tension

b)

resting length

c)

active tension

d)

total tension

60.

Which tension is the sum of passive and active tension

a)

passive tension

b)

resting length

c)

active tension

d)

total tension

61.

What is A

a)

Resting Length

b)

Total Tension

c)

Active Tension

d)

Passive Tension

62.

What is B

a)

Resting Length

b)

Total Tension

c)

Active Tension

d)

Passive Tension

63.

What is C

a)

Resting Length

b)

Total Tension

c)

Active Tension

d)

Passive Tension

64.

What is D

a)

Resting Length

b)

Total Tension

c)

Active Tension

d)

Passive Tension

65.

In muscle loading, what is preload

a)

load placed on a muscle before it contracts

b)

load to muscle encounters after it starts to shorten

66.

In muscle loading, what is afterload

a)

load placed on a muscle before it contracts

b)

load to muscle encounters after it starts to shorten

67.

In muscle mechanics, what is velocity describing

a)

increasing the load on a muscle slows contraction

b)

there is a shift from isotonic contraction to isometric contraction as load increases in weight

c)

increasing the load on a muscle speeds up contraction

d)

there is a shift from isometric contraction to isotonic contraction as load increases in weight

68.

In this muscle velocity graph, what is A

a)

initial isometric phase absent; fastest isotonic shortening

b)

initial isometric phase is small; fast isotonic shortening

c)

initial isometric phase is large; slow isotonic shortening

d)

completely isometric; no shortening

69.

In this muscle velocity graph, what is B

a)

initial isometric phase absent; fastest isotonic shortening

b)

initial isometric phase is small; fast isotonic shortening

c)

initial isometric phase is large; slow isotonic shortening

d)

completely isometric; no shortening

70.

In this muscle velocity graph, what is C

a)

initial isometric phase absent; fastest isotonic shortening

b)

initial isometric phase is small; fast isotonic shortening

c)

initial isometric phase is large; slow isotonic shortening

d)

completely isometric; no shortening

71.

In this muscle velocity graph, what is D

a)

initial isometric phase absent; fastest isotonic shortening

b)

initial isometric phase is small; fast isotonic shortening

c)

initial isometric phase is large; slow isotonic shortening

d)

completely isometric; no shortening

72.

What is a whole muscle made out of

a)

many fibers

b)

motor units

c)

slow-oxidative fiber

d)

fast-oxidative-glycolytic fiber

e)

fast-glycolytic fiber

73.

What types of fibers do most muscles contain

a)

all types

b)

slow-oxidative fiber

c)

fast-oxidative-glycolytic fiber

d)

fast-glycolytic fiber

74.

Why do most muscles contain all types of muscle fibers

a)

muscles carry out different functions at different times

b)

muscles only use one type of fiber, the others are not used